Monocytes from patients with indeterminate and cardiac forms of Chagas' disease display distinct phenotypic and functional characteristics associated with morbidity.
about
A sensitive flow cytometric methodology for studying the binding of L. chagasi to canine peritoneal macrophages.Current understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas diseaseOxidant generation by single infected monocytes after short-term fluorescence labeling of a protozoan parasiteCoinfection with different Trypanosoma cruzi strains interferes with the host immune response to infection.Regulatory T cells phenotype in different clinical forms of Chagas' diseaseTumor necrosis factor is a therapeutic target for immunological unbalance and cardiac abnormalities in chronic experimental Chagas' heart disease.Immunoregulatory networks in human Chagas disease.Trypanosoma cruzi-induced activation of functionally distinct αβ and γδ CD4- CD8- T cells in individuals with polar forms of Chagas' disease.Different infective forms trigger distinct immune response in experimental Chagas diseaseTcVac3 induced control of Trypanosoma cruzi infection and chronic myocarditis in mice.Monocytes play an IL-12-dependent crucial role in driving cord blood NK cells to produce IFN-g in response to Trypanosoma cruziPolymorphic sites at the immunoregulatory CTLA-4 gene are associated with chronic chagas disease and its clinical manifestations.IL-10 inhibits the NF-κB and ERK/MAPK-mediated production of pro-inflammatory mediators by up-regulation of SOCS-3 in Trypanosoma cruzi-infected cardiomyocytes.Plasma cytokine expression is associated with cardiac morbidity in chagas disease.Killer Cell Immunoglobulin-like Receptors and Their HLA Ligands are Related with the Immunopathology of Chagas Disease.Differential Activation of Human Monocytes and Lymphocytes by Distinct Strains of Trypanosoma cruzi.Protective human leucocyte antigen haplotype, HLA-DRB1*01-B*14, against chronic Chagas disease in Bolivia.Changes in Proteome Profile of Peripheral Blood Mononuclear Cells in Chronic Chagas DiseaseDifferential Expression of Matrix Metalloproteinases 2, 9 and Cytokines by Neutrophils and Monocytes in the Clinical Forms of Chagas Disease.Vesicles from different Trypanosoma cruzi strains trigger differential innate and chronic immune responses.Methodological approach to the ex vivo expansion and detection of T. cruzi-specific T cells from chronic Chagas disease patients.Delivery of antigenic candidates by a DNA/MVA heterologous approach elicits effector CD8(+)T cell mediated immunity against Trypanosoma cruzi.High interleukin 17 expression is correlated with better cardiac function in human Chagas disease.Current concepts in immunoregulation and pathology of human Chagas disease.Acute heart inflammation: ultrastructural and functional aspects of macrophages elicited by Trypanosoma cruzi infectionChagas' disease: an update on immune mechanisms and therapeutic strategiesInnate immunity and regulatory T-cells in human Chagas disease: what must be understood?Serum-mediated activation of macrophages reflects TcVac2 vaccine efficacy against Chagas disease.Lipid bodies: inflammatory organelles implicated in host-Trypanosoma cruzi interplay during innate immune responses.Participation of TLR2 and TLR4 in Cytokines Production by Patients with Symptomatic and Asymptomatic Chronic Chagas Disease.Synergic and antagonistic relationship between MMP-2 and MMP-9 with fibrosis and inflammation in Chagas' cardiomyopathy.Pathogenesis of Chagas disease: time to move on.Cellular and genetic mechanisms involved in the generation of protective and pathogenic immune responses in human Chagas disease.Immunoregulatory actions of melatonin and zinc during chronic Trypanosoma cruzi infection.Trypanosoma cruzi SSP4 Amastigote Protein Induces Expression of Immunoregulatory and Immunosuppressive Molecules in Peripheral Blood Mononuclear Cells.Trypanosoma cruzi infection induces differential modulation of costimulatory molecules and cytokines by monocytes and T cells from patients with indeterminate and cardiac Chagas' disease.Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven InflammationAre increased frequency of macrophage-like and natural killer (NK) cells, together with high levels of NKT and CD4+CD25high T cells balancing activated CD8+ T cells, the key to control Chagas' disease morbidity?Distinct Trypanosoma cruzi isolates induce activation and apoptosis of human neutrophils.Specific activation of CD4- CD8- double-negative T cells by Trypanosoma cruzi-derived glycolipids induces a proinflammatory profile associated with cardiomyopathy in Chagas patients.
P2860
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P2860
Monocytes from patients with indeterminate and cardiac forms of Chagas' disease display distinct phenotypic and functional characteristics associated with morbidity.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Monocytes from patients with i ...... ics associated with morbidity.
@en
type
label
Monocytes from patients with i ...... ics associated with morbidity.
@en
prefLabel
Monocytes from patients with i ...... ics associated with morbidity.
@en
P2093
P2860
P1476
Monocytes from patients with i ...... ics associated with morbidity.
@en
P2093
Andréa C L Chaves
Cristiane A S Menezes
Manoel O C Rocha
Paulo E A Souza
Walderez O Dutra
P2860
P304
P356
10.1128/IAI.72.9.5283-5291.2004
P407
P5008
P577
2004-09-01T00:00:00Z