Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death. - Wikidata - awgldk - TriplyDB

Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

http://www.wikidata.org/entity/Q40125595

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lncRNAdb: a reference database for long noncoding RNAs The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases Partners in Crime: The Role of CMV in Immune Dysregulation and Clinical Outcome During HIV Infection Decoding human cytomegalovirus Viral control of mitochondrial apoptosis Human cytomegalovirus UL29/28 protein interacts with components of the NuRD complex which promote accumulation of immediate-early RNA Genomic and functional characteristics of human cytomegalovirus revealed by next-generation sequencing Discrete clusters of virus-encoded micrornas are associated with complementary strands of the genome and the 7.2-kilobase stable intron in murine cytomegalovirus BclAF1 restriction factor is neutralized by proteasomal degradation and microRNA repression during human cytomegalovirus infection Antisense transcription in the human cytomegalovirus transcriptome.HtrA2/Omi terminates cytomegalovirus infection and is controlled by the viral mitochondrial inhibitor of apoptosis (vMIA)GRIM-19 and p16(INK4a) synergistically regulate cell cycle progression and E2F1-responsive gene expression.Cytomegalovirus latency and reactivation: recent insights into an age old problem.Viral mitochondria-localized inhibitor of apoptosis (UL37 exon 1 protein) does not protect human neural precursor cells from human cytomegalovirus-induced cell death Human Cytomegalovirus Latency: Approaching the Gordian Knot.Regulation of the subcellular distribution of key cellular RNA-processing factors during permissive human cytomegalovirus infection On the classification of long non-coding RNAs.Current knowledge of MicroRNAs and noncoding RNAs in virus-infected cells.Identification of a structural motif in the tumor-suppressive protein GRIM-19 required for its antitumor activity Cytokine-induced tumor suppressors: a GRIM story Quantitative proteomic analysis of A549 cells infected with human respiratory syncytial virus Down-regulation of GRIM-19 expression is associated with hyperactivation of STAT3-induced gene expression and tumor growth in human cervical cancers.GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses.Noncoding RNPs of viral origin A novel neuroprotective therapy for Parkinson's disease using a viral noncoding RNA that protects mitochondrial complex I activity.The human cytomegalovirus protein UL37 exon 1 associates with internal lipid rafts.Cis and trans acting factors involved in human cytomegalovirus experimental and natural latent infection of CD14 (+) monocytes and CD34 (+) cells.Mitochondria in innate immune responses.Whole transcriptome sequencing enables discovery and analysis of viruses in archived primary central nervous system lymphomas Viral noncoding RNAs: more surprises.Quantitative proteomic analyses of human cytomegalovirus-induced restructuring of endoplasmic reticulum-mitochondrial contacts at late times of infection.The role of long non-coding RNAs in genome formatting and expression Kaposi's sarcoma-associated herpesvirus noncoding polyadenylated nuclear RNA interacts with virus- and host cell-encoded proteins and suppresses expression of genes involved in immune modulation Viral product trafficking to mitochondria, mechanisms and roles in pathogenesis.High-resolution human cytomegalovirus transcriptome Human cytomegalovirus activation of ERK and myeloid cell leukemia-1 protein correlates with survival of latently infected cells.PNPASE and RNA trafficking into mitochondria.Landscape of post-transcriptional gene regulation during hepatitis C virus infection West Nile virus noncoding subgenomic RNA contributes to viral evasion of the type I interferon-mediated antiviral response.GRIM-19-mediated translocation of STAT3 to mitochondria is necessary for TNF-induced necroptosis.

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P2860

Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

http://www.wikidata.org/entity/Q40125595

description

2007 nî lūn-bûn

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2007年论文

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2007年论文

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name

Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

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Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

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Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

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Complex I binding by a virally encoded RNA regulates mitochondria-induced cell death.

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Andrew A Davies

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Gavin W Wilkinson

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John H Sinclair

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P2860

Identification of GRIM-19, a novel cell death-regulatory gene induced by the interferon-beta and retinoic acid combination, using a genetic approach

GRIM-19, a death-regulatory gene product, suppresses Stat3 activity via functional interaction

GRIM-19, a cell death regulatory protein, is essential for assembly and function of mitochondrial complex I

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

The pathophysiology of mitochondrial cell death

Mitochondrial disorders: clinical and genetic features.

Latency and reactivation of human cytomegalovirus.

Absence of IE1 p72 protein function during low-multiplicity infection by human cytomegalovirus results in a broad block to viral delayed-early gene expression.

Mitochondria: releasing power for life and unleashing the machineries of death.

Mitochondrial dysfunction, apoptotic cell death, and Alzheimer's disease.

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1345-1348

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10.1126/SCIENCE.1142984

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316

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Brian P McSharry

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6295251

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