Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
about
Recent Progress in Chronic Neutrophilic Leukemia and Atypical Chronic Myeloid Leukemia.Chronic neutrophilic leukemia: new science and new diagnostic criteria.Gain-of-function mutations in granulocyte colony-stimulating factor receptor (CSF3R) reveal distinct mechanisms of CSF3R activation.Neomorphic PDGFRA extracellular domain driver mutations are resistant to PDGFRA targeted therapies
P2860
Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
description
2017 nî lūn-bûn
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2017年の論文
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2017年論文
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2017年論文
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2017年論文
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2017年論文
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2017年論文
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2017年论文
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2017年论文
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2017年论文
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name
Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
@en
type
label
Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
@en
prefLabel
Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
@en
P2093
P2860
P1433
P1476
Unpaired Extracellular Cysteine Mutations of CSF3R Mediate Gain or Loss of Function.
@en
P2093
Anna Reister Schultz
Beth Wilmot
Bruno C Medeiros
Daniel Bottomly
Haijiao Zhang
Jeffrey W Tyner
Shannon K McWeeney
Sophie Means
Tim Kükenshöner
P2860
P304
P356
10.1158/0008-5472.CAN-17-1052
P407
P577
2017-06-26T00:00:00Z