Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
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Mechanisms of disease: Helicobacter pylori virulence factorsRegulation of cell migration and morphogenesis by Abl-family kinases: emerging mechanisms and physiological contextsMultifunctional Abl kinases in health and diseaseEffect of Helicobacter pylori on gastric epithelial cellsPolymorphism in the Helicobacter pylori CagA and VacA toxins and diseaseStructure of the Helicobacter pylori CagA oncoprotein bound to the human tumor suppressor ASPP2Order and disorder in large multi-site docking proteins of the Gab family--implications for signalling complex formation and inhibitor design strategiesTarp regulates early Chlamydia-induced host cell survival through interactions with the human adaptor protein SHC1Abelson tyrosine kinase facilitates Salmonella enterica serovar Typhimurium entry into epithelial cells.Helicobacter pylori cytotoxin-associated gene A (CagA) subverts the apoptosis-stimulating protein of p53 (ASPP2) tumor suppressor pathway of the hostInvasion of endothelial cells by tissue-invasive M3 type group A streptococci requires Src kinase and activation of Rac1 by a phosphatidylinositol 3-kinase-independent mechanism.Helicobacter pylori CagA causes mitotic impairment and induces chromosomal instability.Helicobacter pylori type IV secretion apparatus exploits beta1 integrin in a novel RGD-independent mannerDietary factors modulate Helicobacter-associated gastric cancer in rodent modelsEffector prediction in host-pathogen interaction based on a Markov model of a ubiquitous EPIYA motif.Helicobacter pylori induced gastric immunopathology is associated with distinct microbiota changes in the large intestines of long-term infected Mongolian gerbilsHelicobacter pylori CagA triggers expression of the bactericidal lectin REG3γ via gastric STAT3 activationPresence of terminal EPIYA phosphorylation motifs in Helicobacter pylori CagA contributes to IL-8 secretion, irrespective of the number of repeats.A novel inhibitory domain of Helicobacter pylori protein CagA reduces CagA effects on host cell biology.Complex cellular responses of Helicobacter pylori-colonized gastric adenocarcinoma cellsSystematic analysis of phosphotyrosine antibodies recognizing single phosphorylated EPIYA-motifs in CagA of Western-type Helicobacter pylori strains.Helicobacter pylori cag pathogenicity island's role in B7-H1 induction and immune evasion.Paxillin is a novel cellular target for converging Helicobacter pylori-induced cellular signaling.Coiled coil rich proteins (Ccrp) influence molecular pathogenicity of Helicobacter pyloriMolecular mechanisms of gastric epithelial cell adhesion and injection of CagA by Helicobacter pylori.Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system.c-Src and c-Abl kinases control hierarchic phosphorylation and function of the CagA effector protein in Western and East Asian Helicobacter pylori strainsMultistep activation of the Helicobacter pylori effector CagAFragmentation of CagA Reduces Hummingbird Phenotype Induction by Helicobactor pylori.Systematic analysis of phosphotyrosine antibodies recognizing single phosphorylated EPIYA-motifs in CagA of East Asian-type Helicobacter pylori strainsTransgenic expression of Helicobacter pylori CagA induces gastrointestinal and hematopoietic neoplasms in mouse.The beta1 integrin activates JNK independent of CagA, and JNK activation is required for Helicobacter pylori CagA+-induced motility of gastric cancer cellsChlamydia trachomatis tarp is phosphorylated by src family tyrosine kinasesCagA of Helicobacter pylori interacts with and inhibits the serine-threonine kinase PRK2Interaction of Helicobacter pylori with gastric epithelial cells is mediated by the p53 protein family.Cholesterol depletion reduces Helicobacter pylori CagA translocation and CagA-induced responses in AGS cells.The Influence of the Gut Microbiome on Obesity, Metabolic Syndrome and Gastrointestinal Disease.CagA EPIYA polymorphisms in Colombian Helicobacter pylori strains and their influence on disease-associated cellular responses.The impact of autophagic processes on the intracellular fate of Helicobacter pylori: more tricks from an enigmatic pathogen?CagA Phosphorylation in Helicobacter pylori-Infected B Cells Is Mediated by the Nonreceptor Tyrosine Kinases of the Src and Abl Families
P2860
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P2860
Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
@en
type
label
Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
@en
prefLabel
Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
@en
P2093
P2860
P356
P1433
P1476
Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1210139
P407
P577
2006-12-11T00:00:00Z
P5875
P6179
1003589921