Benzyl isothiocyanate-induced apoptosis in human breast cancer cells is initiated by reactive oxygen species and regulated by Bax and Bak.
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NADPH oxidase as a therapeutic target for oxalate induced injury in kidneysCancer chemoprevention with dietary isothiocyanates mature for clinical translational researchPUMA decreases the growth of prostate cancer PC-3 cells independent of p53.Withaferin A inhibits experimental epithelial-mesenchymal transition in MCF-10A cells and suppresses vimentin protein level in vivo in breast tumors.Mechanism of action of phenethylisothiocyanate and other reactive oxygen species-inducing anticancer agentsInhibition of human breast cancer xenograft growth by cruciferous vegetable constituent benzyl isothiocyanateBenzyl isothiocyanate (BITC) and phenethyl isothiocyanate (PEITC)-mediated generation of reactive oxygen species causes cell cycle arrest and induces apoptosis via activation of caspase-3, mitochondria dysfunction and nitric oxide (NO) in human ostep53-Independent apoptosis by benzyl isothiocyanate in human breast cancer cells is mediated by suppression of XIAP expressionBreast cancer cell growth inhibition by phenethyl isothiocyanate is associated with down-regulation of oestrogen receptor-alpha36Withaferin A-induced apoptosis in human breast cancer cells is mediated by reactive oxygen species.Notch2 activation is protective against anticancer effects of zerumbone in human breast cancer cellsPhenethyl isothiocyanate inhibits oxidative phosphorylation to trigger reactive oxygen species-mediated death of human prostate cancer cellsSelective depletion of mutant p53 by cancer chemopreventive isothiocyanates and their structure-activity relationshipsCritical role of p53 upregulated modulator of apoptosis in benzyl isothiocyanate-induced apoptotic cell deathBenzyl isothiocyanate causes FoxO1-mediated autophagic death in human breast cancer cells.Differential response of normal (PrEC) and cancerous human prostate cells (PC-3) to phenethyl isothiocyanate-mediated changes in expression of antioxidant defense genes.Reactive oxygen species-dependent apoptosis by gugulipid extract of Ayurvedic medicine plant Commiphora mukul in human prostate cancer cells is regulated by c-Jun N-terminal kinase.Withaferin A causes FOXO3a- and Bim-dependent apoptosis and inhibits growth of human breast cancer cells in vivo.Ayurvedic medicine constituent withaferin a causes G2 and M phase cell cycle arrest in human breast cancer cellsLutein protects against methotrexate-induced and reactive oxygen species-mediated apoptotic cell injury of IEC-6 cells.Benzyl isothiocyanate inhibits oncogenic actions of leptin in human breast cancer cells by suppressing activation of signal transducer and activator of transcription 3.The role of polycomb group protein Bmi-1 and Notch4 in breast cancer stem cell inhibition by benzyl isothiocyanate.Benzyl isothiocyanate inhibits epithelial-mesenchymal transition in cultured and xenografted human breast cancer cellsMacroautophagy: the key ingredient to a healthy diet?Bim contributes to phenethyl isothiocyanate-induced apoptosis in breast cancer cells.D,L-sulforaphane-induced apoptosis in human breast cancer cells is regulated by the adapter protein p66Shc.Glucosinolate-derived isothiocyanates impact mitochondrial function in fungal cells and elicit an oxidative stress response necessary for growth recoveryThe molecular basis that unifies the metabolism, cellular uptake and chemopreventive activities of dietary isothiocyanates.Notch2 activation by benzyl isothiocyanate impedes its inhibitory effect on breast cancer cell migrationShort-form RON overexpression augments benzyl isothiocyanate-induced apoptosis in human breast cancer cellsZerumbone causes Bax- and Bak-mediated apoptosis in human breast cancer cells and inhibits orthotopic xenograft growth in vivo.Benzyl Isothiocyanate Inhibits Prostate Cancer Development in the Transgenic Adenocarcinoma Mouse Prostate (TRAMP) Model, Which Is Associated with the Induction of Cell Cycle G1 Arrest.Critical role for reactive oxygen species in apoptosis induction and cell migration inhibition by diallyl trisulfide, a cancer chemopreventive component of garlicIn vitro and in vivo effects of phenethyl isothiocyanate treatment on vimentin protein expression in cancer cells.Benzyl isothiocyanate targets mitochondrial respiratory chain to trigger reactive oxygen species-dependent apoptosis in human breast cancer cells.Role of reactive oxygen intermediates in cellular responses to dietary cancer chemopreventive agents.Autophagy fails to alter withaferin A-mediated lethality in human breast cancer cells.Apoptosis and necrosis of human breast cancer cells by an aqueous extract of garden cress (Lepidium sativum) seedsSynthesis and anticancer activity comparison of phenylalkyl isoselenocyanates with corresponding naturally occurring and synthetic isothiocyanates.Downregulation of Mcl-1 through inhibition of translation contributes to benzyl isothiocyanate-induced cell cycle arrest and apoptosis in human leukemia cells.
P2860
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P2860
Benzyl isothiocyanate-induced apoptosis in human breast cancer cells is initiated by reactive oxygen species and regulated by Bax and Bak.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Benzyl isothiocyanate-induced ...... and regulated by Bax and Bak.
@en
type
label
Benzyl isothiocyanate-induced ...... and regulated by Bax and Bak.
@en
prefLabel
Benzyl isothiocyanate-induced ...... and regulated by Bax and Bak.
@en
P2093
P1476
Benzyl isothiocyanate-induced ...... and regulated by Bax and Bak.
@en
P2093
Shivendra V Singh
Victor Vogel
P304
P356
10.1158/1535-7163.MCT-06-0396
P577
2006-11-01T00:00:00Z