Lipopolysaccharide from the periodontal pathogen Porphyromonas gingivalis prevents apoptosis of HL60-derived neutrophils in vitro.
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Gingipain-dependent interactions with the host are important for survival of Porphyromonas gingivalisIntrinsic apoptotic pathways of gingival epithelial cells modulated by Porphyromonas gingivalisRole of Porphyromonas gingivalis HmuY in Immunopathogenesis of Chronic PeriodontitisCommunity-wide transcriptome of the oral microbiome in subjects with and without periodontitisMeningococcal porin PorB prevents cellular apoptosis in a toll-like receptor 2- and NF-kappaB-independent mannerManipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses.Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalisPorphyromonas gingivalis regulates TREM-1 in human polymorphonuclear neutrophils via its gingipains.Functional signatures of oral dysbiosis during periodontitis progression revealed by microbial metatranscriptome analysis.Apoptosis in normal and diseased oral tissues.Major neutrophil functions subverted by Porphyromonas gingivalis.Porphyromonas gingivalis invades human trophoblasts and inhibits proliferation by inducing G1 arrest and apoptosis.Human neutrophils and oral microbiota: a constant tug-of-war between a harmonious and a discordant coexistence.How has neutrophil research improved our understanding of periodontal pathogenesis?Direct effect of streptozotocin on periodontal ligament cells through myeloid cell leukemia-1.Impaired immune tolerance to Porphyromonas gingivalis lipopolysaccharide promotes neutrophil migration and decreased apoptosis.The Porphyromonas gingivalis O antigen is required for inhibition of apoptosis in gingival epithelial cells following bacterial infection.The role of phagocytosis, oxidative burst and neutrophil extracellular traps in the interaction between neutrophils and the periodontal pathogen Porphyromonas gingivalis.Porphyromonas gingivalis, gamma interferon, and a proapoptotic fibronectin matrix form a synergistic trio that induces c-Jun N-terminal kinase 1-mediated nitric oxide generation and cell death.Identification of proteins differentially expressed in human monocytes exposed to Porphyromonas gingivalis and its purified components by high-throughput immunoblotting.Orally administered liposomal lactoferrin inhibits inflammation-related bone breakdown without interrupting orthodontic tooth movement.Induction of Porphyromonas gingivalis GroEL signaling via binding to Toll-like receptors 2 and 4.
P2860
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P2860
Lipopolysaccharide from the periodontal pathogen Porphyromonas gingivalis prevents apoptosis of HL60-derived neutrophils in vitro.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Lipopolysaccharide from the pe ...... -derived neutrophils in vitro.
@en
type
label
Lipopolysaccharide from the pe ...... -derived neutrophils in vitro.
@en
prefLabel
Lipopolysaccharide from the pe ...... -derived neutrophils in vitro.
@en
P2860
P1476
Lipopolysaccharide from the pe ...... -derived neutrophils in vitro.
@en
P2093
D A Murray
J M A Wilton
P2860
P304
P356
10.1128/IAI.71.12.7232-7235.2003
P407
P577
2003-12-01T00:00:00Z