A method to monitor replication fork progression in mammalian cells: nucleotide excision repair enhances and homologous recombination delays elongation along damaged DNA.
about
Redundancy of mammalian Y family DNA polymerases in cellular responses to genomic DNA lesions induced by ultraviolet lightMCL-1 localizes to sites of DNA damage and regulates DNA damage responseRoles of the Werner syndrome RecQ helicase in DNA replication.Single cell analysis of human RAD18-dependent DNA post-replication repair by alkaline bromodeoxyuridine comet assay.A mutant allele of the transcription factor IIH helicase gene, RAD3, promotes loss of heterozygosity in response to a DNA replication defect in Saccharomyces cerevisiae.CHK1 activity is required for continuous replication fork elongation but not stabilization of post-replicative gaps after UV irradiationDNA damage responses and their many interactions with the replication fork.Regulation and disregulation of mammalian nucleotide excision repair: a pathway to nongermline breast carcinogenesis.The histone methyltransferase SET8 is required for S-phase progressionEnvironmental exposure and HPV infection may act synergistically to induce lung tumorigenesis in nonsmokers.Separate domains of Rev1 mediate two modes of DNA damage bypass in mammalian cells.PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination.UV stalled replication forks restart by re-priming in human fibroblasts.The identification of translesion DNA synthesis regulators: Inhibitors in the spotlight.Different sets of translesion synthesis DNA polymerases protect from genome instability induced by distinct food-derived genotoxins.Poly (ADP-ribose) polymerase (PARP) is not involved in base excision repair but PARP inhibition traps a single-strand intermediate.The ERCC1/XPF endonuclease is required for completion of homologous recombination at DNA replication forks stalled by inter-strand cross-links.dUTPase inhibition augments replication defects of 5-Fluorouracil.The RecQ helicase WRN is required for normal replication fork progression after DNA damage or replication fork arrest.Genotoxicity of alcohol is linked to DNA replication-associated damage and homologous recombination repair.
P2860
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P2860
A method to monitor replication fork progression in mammalian cells: nucleotide excision repair enhances and homologous recombination delays elongation along damaged DNA.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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name
A method to monitor replicatio ...... elongation along damaged DNA.
@en
type
label
A method to monitor replicatio ...... elongation along damaged DNA.
@en
prefLabel
A method to monitor replicatio ...... elongation along damaged DNA.
@en
P2093
P2860
P356
P1476
A method to monitor replicatio ...... elongation along damaged DNA.
@en
P2093
Anne Lagerqvist
Dag Jenssen
Fredrik Johansson
Klaus Erixon
P2860
P356
10.1093/NAR/GNH154
P577
2004-11-10T00:00:00Z