Two alternative pathways of double-strand break repair that are kinetically separable and independently modulated.
about
Differential effects of Rad52p overexpression on gene targeting and extrachromosomal homologous recombination in a human cell lineCharacterization of mutations that suppress the temperature-sensitive growth of the hpr1 delta mutant of Saccharomyces cerevisiae.High frequency repeat-induced point mutation (RIP) is not associated with efficient recombination in NeurosporaMultiple pathways of recombination induced by double-strand breaks in Saccharomyces cerevisiaeRsp5, a ubiquitin-protein ligase, is involved in degradation of the single-stranded-DNA binding protein rfa1 in Saccharomyces cerevisiaeTAL nucleases (TALNs): hybrid proteins composed of TAL effectors and FokI DNA-cleavage domainRecombinational repair of gaps in DNA is asymmetric in Ustilago maydis and can be explained by a migrating D-loop modelInvolvement of nucleotide excision and mismatch repair mechanisms in double strand break repairCell cycle and genetic requirements of two pathways of nonhomologous end-joining repair of double-strand breaks in Saccharomyces cerevisiaeRepairing a double-strand chromosome break by homologous recombination: revisiting Robin Holliday's modelGene conversion and deletion frequencies during double-strand break repair in human cells are controlled by the distance between direct repeatsRole of the silkworm argonaute2 homolog gene in double-strand break repair of extrachromosomal DNAApplications of CRISPR-Cas systems in neuroscienceMechanisms and regulation of mitotic recombination in Saccharomyces cerevisiaeMultiple cellular mechanisms prevent chromosomal rearrangements involving repetitive DNAGenetic requirements for the single-strand annealing pathway of double-strand break repair in Saccharomyces cerevisiaeRole of the Rad1 and Rad10 proteins in nucleotide excision repair and recombination.Role of Saccharomyces cerevisiae Msh2 and Msh3 repair proteins in double-strand break-induced recombination.Mutations in homologous recombination genes rescue top3 slow growth in Saccharomyces cerevisiae.A phosphatase complex that dephosphorylates gammaH2AX regulates DNA damage checkpoint recovery.Opposing roles for DNA structure-specific proteins Rad1, Msh2, Msh3, and Sgs1 in yeast gene targeting.RAD1 and RAD10, but not other excision repair genes, are required for double-strand break-induced recombination in Saccharomyces cerevisiae.Mutations in the Saccharomyces cerevisiae CDC1 gene affect double-strand-break-induced intrachromosomal recombinationReappearance from Obscurity: Mammalian Rad52 in Homologous RecombinationNoncanonical views of homology-directed DNA repairMultiple roles of ERCC1-XPF in mammalian interstrand crosslink repairThe SUMO isopeptidase Ulp2p is required to prevent recombination-induced chromosome segregation lethality following DNA replication stressNuclear foci of mammalian recombination proteins are located at single-stranded DNA regions formed after DNA damageEnzymatic activities and DNA substrate specificity of Mycobacterium tuberculosis DNA helicase XPBEvidence for multiple cycles of strand invasion during repair of double-strand gaps in DrosophilaDouble strand break-induced recombination in Chlamydomonas reinhardtii chloroplastsRole of RAD52 epistasis group genes in homologous recombination and double-strand break repairEscherichia coli RecO protein anneals ssDNA complexed with its cognate ssDNA-binding protein: A common step in genetic recombination.The recombination-deficient mutant RPA (rfa1-t11) is displaced slowly from single-stranded DNA by Rad51 protein.Genetic analysis of yeast RPA1 reveals its multiple functions in DNA metabolism.Finding alternative expression quantitative trait loci by exploring sparse model spaceSgs1 and exo1 redundantly inhibit break-induced replication and de novo telomere addition at broken chromosome ends.Rad51 inhibits translocation formation by non-conservative homologous recombination in Saccharomyces cerevisiae.Harnessing the Potential of Human Pluripotent Stem Cells and Gene Editing for the Treatment of Retinal Degeneration.Competitive repair by naturally dispersed repetitive DNA during non-allelic homologous recombination
P2860
Q24514763-E40BA562-CF6B-4F00-8878-26DC7EB865F9Q24533015-C8D3F9D3-3ED7-4752-AED6-99443BA533A7Q24533135-4F8681DC-9C61-4FA4-A50C-FE8BA4CC6FE6Q24548535-057E49D0-2430-49E6-ADAD-5AD7F47E4406Q24554358-4A5F9C43-FD10-4AB5-8FAD-9DA59BD58442Q24603277-8BA28EF3-A889-4C8F-9082-E4BE3DB7EB08Q24605056-7FC2D00E-228B-4A26-ADFE-72A020AE87C0Q24649491-5FE9F22E-33CC-469D-BD63-02B971F61E01Q24649768-6BA22FA4-653F-4A52-A1F6-3416F522F2B7Q24675453-3C8E5222-8671-4D81-811F-7DC7232BF71CQ24803093-8D7C3EE6-FB34-4212-B9EF-7E1355A53663Q25257701-A00DCA08-09B4-4C85-A507-93D41CFA2F31Q26776515-F46FBB3F-AAD5-486B-9926-804D4E069B18Q26864428-91B6BDC7-FA2E-4C22-818C-07DCADABFFAEQ27012760-47E769A7-D165-4F2F-8FC2-D494B844D889Q27929777-FF6B95C7-C159-4402-81A5-6D7E5089A76AQ27931636-00760612-13AE-461A-A453-F7D0027E4F9DQ27931976-13AE6426-E00D-4C40-8B10-F3DF9D908094Q27931991-3D5E3842-AE39-428E-8813-8053A43F7B9EQ27933727-56C968D3-E0F6-4E4C-A0E8-5B373E3DD25FQ27934436-799CBE0D-3278-47E9-98CE-C13FA660A9D0Q27935264-6E982438-052D-4244-BCBD-DA23A35EEE4CQ27938437-97456923-5403-4A6E-8059-0606834FC60FQ28074245-C98A8465-8A03-41C6-8FB4-945BD7152AEDQ28080032-2AD31B5D-DF48-4185-B098-A76F256B32F6Q28288789-D7011CA7-2FAE-4C68-B35E-12632A2F1122Q28477618-3E482F58-27F2-42BB-A29A-D531ED9CB2F8Q28610023-48F66313-62A7-4059-85E7-EE7BF707C34AQ28729923-FA2A1DD1-3515-44CE-B85D-3FA929A577B4Q28768697-0C1CB440-3A52-45D9-8C4D-945A1FD205F8Q29041806-2D15E7CF-A52D-48F1-B13B-974B46F65E23Q29618204-AFED234F-CDB0-47DE-96B4-2E898D8DA4FBQ30870765-7183E9B0-FF0B-4412-AFB0-5BBE76B9D889Q30917944-CF037B08-4D33-4DCB-B870-D6E2579B0D04Q32068528-954C95F7-D2E8-43EF-95F5-02A81C830F09Q33566556-FF0BD927-179D-4E41-861C-A7F38AB4D4DDQ33594452-35A6CB66-C448-424F-9447-9617342337C7Q33649800-B3143290-875A-47DB-84B1-8A5579869C06Q33729610-97148AD5-E8B4-448C-90AC-8E7D6C3DC545Q33769557-53732117-01F0-4A4C-9057-1855C7834631
P2860
Two alternative pathways of double-strand break repair that are kinetically separable and independently modulated.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Two alternative pathways of do ...... e and independently modulated.
@en
type
label
Two alternative pathways of do ...... e and independently modulated.
@en
prefLabel
Two alternative pathways of do ...... e and independently modulated.
@en
P2093
P2860
P356
P1476
Two alternative pathways of do ...... e and independently modulated.
@en
P2093
J Fishman-Lobell
P2860
P304
P356
10.1128/MCB.12.3.1292
P407
P577
1992-03-01T00:00:00Z