Anti-Abeta antibody treatment promotes the rapid recovery of amyloid-associated neuritic dystrophy in PDAPP transgenic mice.
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Active and passive immunotherapy for neurodegenerative disordersImaging of Leukocyte Trafficking in Alzheimer's DiseaseDevelopment of positron emission tomography β-amyloid plaque imaging agentsThe dendritic hypothesis for Alzheimer's disease pathophysiology.Spines, plasticity, and cognition in Alzheimer's model miceTransgenic mice overexpressing reticulon 3 develop neuritic abnormalities.Advances in light microscopy for neuroscience.Robust amyloid clearance in a mouse model of Alzheimer's disease provides novel insights into the mechanism of amyloid-beta immunotherapyAlzheimer disease models and human neuropathology: similarities and differencesContribution of reactive oxygen species to cerebral amyloid angiopathy, vasomotor dysfunction, and microhemorrhage in aged Tg2576 mice.Focused ultrasound for blood-brain disruption and delivery of therapeutic molecules into the brain.Anti-Amyloid Effects of Small Molecule Aβ-Binding Agents in PS1/APP MiceAmyloid beta induces the morphological neurodegenerative triad of spine loss, dendritic simplification, and neuritic dystrophies through calcineurin activation.Beneficial effect of human anti-amyloid-beta active immunization on neurite morphology and tau pathologyIn vivo characterization of a bigenic fluorescent mouse model of Alzheimer's disease with neurodegenerationSynaptic activity becomes excitotoxic in neurons exposed to elevated levels of platelet-activating factor.Calpain activation promotes BACE1 expression, amyloid precursor protein processing, and amyloid plaque formation in a transgenic mouse model of Alzheimer diseaseAntioxidants have a rapid and long-lasting effect on neuritic abnormalities in APP:PS1 mice.Cholinotrophic basal forebrain system alterations in 3xTg-AD transgenic mice.Brain imaging of nicotinic receptors in Alzheimer's disease.Microglia mediate the clearance of soluble Abeta through fluid phase macropinocytosis.Curcumin labels amyloid pathology in vivo, disrupts existing plaques, and partially restores distorted neurites in an Alzheimer mouse model.Tau passive immunotherapy in mutant P301L mice: antibody affinity versus specificityPassive (amyloid-β) immunotherapy attenuates monoaminergic axonal degeneration in the AβPPswe/PS1dE9 mice.Interleukin-1 receptor 1 knockout has no effect on amyloid deposition in Tg2576 mice and does not alter efficacy following Abeta immunotherapy.Two-photon microscopy for chemical neuroscience.Amyloid accelerates tau propagation and toxicity in a model of early Alzheimer's diseaseT-cell-mediated disruption of the neuronal microtubule network: correlation with early reversible axonal dysfunction in acute experimental autoimmune encephalomyelitis.Impairments in experience-dependent scaling and stability of hippocampal place fields limit spatial learning in a mouse model of Alzheimer's disease.A Peephole into the Brain: Neuropathological Features of Alzheimer's Disease Revealed by in vivo Two-Photon ImagingConstitutive modeling of mouse carotid arteries using experimentally measured microstructural parameters.Diosgenin is an exogenous activator of 1,25D₃-MARRS/Pdia3/ERp57 and improves Alzheimer's disease pathologies in 5XFAD miceImmune clearance of attenuated rabies virus results in neuronal survival with altered gene expression.Cholinergic forebrain degeneration in the APPswe/PS1DeltaE9 transgenic mouse.Plaque-Associated Local Toxicity Increases over the Clinical Course of Alzheimer Disease.Two-photon imaging of synaptic plasticity and pathology in the living mouse brain.Amyloid plaque and neurofibrillary tangle pathology in a regulatable mouse model of Alzheimer's disease.Beta-amyloid deposition and functional impairment in the retina of the APPswe/PS1DeltaE9 transgenic mouse model of Alzheimer's disease.Fusion antibody for Alzheimer's disease with bidirectional transport across the blood-brain barrier and abeta fibril disaggregation.Protecting the synapse: evidence for a rational strategy to treat HIV-1 associated neurologic disease.
P2860
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P2860
Anti-Abeta antibody treatment promotes the rapid recovery of amyloid-associated neuritic dystrophy in PDAPP transgenic mice.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Anti-Abeta antibody treatment ...... ophy in PDAPP transgenic mice.
@en
type
label
Anti-Abeta antibody treatment ...... ophy in PDAPP transgenic mice.
@en
prefLabel
Anti-Abeta antibody treatment ...... ophy in PDAPP transgenic mice.
@en
P2093
P2860
P356
P1476
Anti-Abeta antibody treatment ...... ophy in PDAPP transgenic mice.
@en
P2093
Brian J Bacskai
Chester A Mathis
Jesse M Skoch
John R Cirrito
Kelly A Simmons
Kelly R Bales
Robert P Brendza
Steven M Paul
William E Klunk
P2860
P304
P356
10.1172/JCI23269
P407
P577
2005-02-01T00:00:00Z