Role of the inositol phosphatase SHIP in B cell receptor-induced Ca2+ oscillatory response.
about
Subcellular localization of Grb2 by the adaptor protein Dok-3 restricts the intensity of Ca2+ signaling in B cellsDok-3, a novel adapter molecule involved in the negative regulation of immunoreceptor signalingVav3 modulates B cell receptor responses by regulating phosphoinositide 3-kinase activationPTP-PEST, a scaffold protein tyrosine phosphatase, negatively regulates lymphocyte activation by targeting a unique set of substratesCalcium channels in lymphocytesA dual role for Src homology 2 domain-containing inositol-5-phosphatase (SHIP) in immunity: aberrant development and enhanced function of b lymphocytes in ship -/- miceB cell receptor signal transduction in the GC is short-circuited by high phosphatase activityEssential role for the C-terminal noncatalytic region of SHIP in FcgammaRIIB1-mediated inhibitory signalingRegulation of the Src homology 2 domain-containing inositol 5'-phosphatase (SHIP1) by the cyclic AMP-dependent protein kinaseA balance of Bruton's tyrosine kinase and SHIP activation regulates B cell receptor cluster formation by controlling actin remodeling.Differential roles for the inositol phosphatase SHIP in the regulation of macrophages and lymphocytes.Ig beta tyrosine residues contribute to the control of B cell receptor signaling by regulating receptor internalization.Role of the rasGAP-associated docking protein p62(dok) in negative regulation of B cell receptor-mediated signaling.Akt2 inhibits the activation of NFAT in lymphocytes by modulating calcium release from intracellular storesT cell-specific deletion of the inositol phosphatase SHIP reveals its role in regulating Th1/Th2 and cytotoxic responses.Cbl-b negatively regulates B cell antigen receptor signaling in mature B cells through ubiquitination of the tyrosine kinase Syk.Differential regulation of B cell development, activation, and death by the src homology 2 domain-containing 5' inositol phosphatase (SHIP)The distinctive germinal center phase of IgE+ B lymphocytes limits their contribution to the classical memory response.Aberrant antibody affinity selection in SHIP-deficient B cells.Rictor positively regulates B cell receptor signaling by modulating actin reorganization via ezrin.Pharmacological targeting of phosphoinositide lipid kinases and phosphatases in the immune system: success, disappointment, and new opportunities.The dynamics of protein kinase B regulation during B cell antigen receptor engagement.
P2860
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P2860
Role of the inositol phosphatase SHIP in B cell receptor-induced Ca2+ oscillatory response.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Role of the inositol phosphata ...... ced Ca2+ oscillatory response.
@en
type
label
Role of the inositol phosphata ...... ced Ca2+ oscillatory response.
@en
prefLabel
Role of the inositol phosphata ...... ced Ca2+ oscillatory response.
@en
P2093
P1476
Role of the inositol phosphata ...... uced Ca2+ oscillatory response
@en
P2093
J V Ravetch
M Kurosaki
T Kurosaki
Y Kabuyama
P304
P407
P577
1998-11-01T00:00:00Z