Participation of tumor necrosis factor in the mediation of gram negative bacterial lipopolysaccharide-induced injury in rabbits.
about
Lipopolysaccharide is in close proximity to each of the proteins in its membrane receptor complex. transfer from CD14 to TLR4 and MD-2Increased expression of the interleukin 1 receptor on blood neutrophils of humans with the sepsis syndromeResilience to bacterial infection: difference between species could be due to proteins in serumTezosentan-induced attenuation of lung injury in endotoxemic sheep is associated with reduced activation of protein kinase C.Role of surface-exposed loops of Haemophilus influenzae protein P2 in the mitogen-activated protein kinase cascadeProbing lipase/esterase libraries for lipid A hydrolases--discovery of biocatalysts for the detoxification of bacterially-expressed recombinant protein.The effect of lipopolysaccharide, interleukin-1 and tumour necrosis factor on the hepatic accumulation of 5-hydroxytryptamine and platelets in the mouse.Staphylococcus epidermidis induces complement activation, tumor necrosis factor and interleukin-1, a shock-like state and tissue injury in rabbits without endotoxemia. Comparison to Escherichia coli.Concentrations of interleukin 6 and tumour necrosis factor in serum and stools of children with Shigella dysenteriae 1 infection.Comparative analysis of lipopolysaccharide-induced tumor necrosis factor alpha activity in serum and lethality in mice and rabbits pretreated with the staphylococcal superantigen toxic shock syndrome toxin 1.Dissociation of lipopolysaccharide (LPS)-inducible gene expression in murine macrophages pretreated with smooth LPS versus monophosphoryl lipid A.Endotoxin-mediated endothelial cell injury and activation: role of soluble CD14.Tumor necrosis factor alpha (TNF-alpha) production in mice immunized with Escherichia coli: correlation with mortality after lethal challenge and production of a native inhibitor of TNF-alpha activityA critical look at cardiovascular translational research.Lipopolysaccharide induction of tissue factor expression in rabbitsLipopolysaccharide (LPS) recognition in macrophages. Participation of LPS-binding protein and CD14 in LPS-induced adaptation in rabbit peritoneal exudate macrophagesRole of T cells and gamma interferon during induction of hypersensitivity to lipopolysaccharide by toxic shock syndrome toxin 1 in miceThe T cell STAT signaling network is reprogrammed within hours of bacteremia via secondary signalsAnti-inflammatory therapies in sepsis and septic shock.A CAT reporter construct allows ultrasensitive estimation of TNF synthesis, and suggests that the TNF gene has been silenced in non-macrophage cell lines.Efficacy of antilipopolysaccharide and anti-tumor necrosis factor monoclonal antibodies in a neutropenic rat model of Pseudomonas sepsisAdministration of endotoxin, tumor necrosis factor, or interleukin 1 to rats activates skeletal muscle branched-chain alpha-keto acid dehydrogenase.Adaptation to bacterial lipopolysaccharide controls lipopolysaccharide-induced tumor necrosis factor production in rabbit macrophagesThe acute splanchnic and peripheral tissue metabolic response to endotoxin in humans.Role of tumor necrosis factor-alpha in the pathophysiologic alterations after hepatic ischemia/reperfusion injury in the rat.Tumor necrosis factor alpha and gamma interferon, but not hemorrhage or pathogen burden, dictate levels of protective fibrin deposition during infectionTumour necrosis factor alpha secretion in leporine endotoxaemia: role of the liver and effects of hepatic ischaemia.Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.Tumor necrosis factor-alpha, interleukin-6, and interleukin-8 secretion and the acute-phase response in patients with bacterial and tuberculous osteomyelitis.Tumor necrosis factor participates in the pathogenesis of acute immune complex alveolitis in the ratDistinctive cytokines as biomarkers predicting fatal outcome of severe Staphylococcus aureus bacteremia in miceCytokine modulation in sepsis and septic shock.Prolonged and effective blockade of tumor necrosis factor activity through adenovirus-mediated gene transferBacterial LPS up-regulated TLR3 expression is critical for antiviral response in human monocytes: evidence for negative regulation by CYLDCytokine induction by lipopolysaccharide (LPS) corresponds to lethal toxicity and is inhibited by nontoxic Rhodobacter capsulatus LPS.Intraperitoneal injection of tetracyclines protects mice from lethal endotoxemia downregulating inducible nitric oxide synthase in various organs and cytokine and nitrate secretion in blood.Participation of lipopolysaccharide-binding protein in lipopolysaccharide-dependent macrophage activation.TNF-alpha dependent production of inducible nitric oxide is involved in PGE(1) protection against acute liver injury.Human macrophages acquire a hyporesponsive state of tumor necrosis factor alpha production in response to successive Mycobacterium avium serovar 4 stimulationAntiendotoxin activity of cationic peptide antimicrobial agents.
P2860
Q24290990-16AE666B-A4FC-49FF-9B68-778016A7D1F9Q24630405-E5A82CEA-087A-4C5F-A2C8-CEA14E70778EQ24654161-DC604312-9A94-4512-BD68-45C072443CE3Q24811647-3BFBC3D3-7DA7-4FAF-B807-BDB83FBD5F11Q30164772-51711F69-8A4E-444E-81AA-05D34A192B49Q33197502-035BAC1D-E2A9-4F61-B20F-7B94D393785EQ33369202-A95387C0-E2AB-4FDB-AEF1-8D838B82C6ABQ33389219-D78F48C0-DA31-4D35-AE37-65776F6B435EQ33494319-69BB000A-2370-4833-8444-7C9121F91486Q33554336-ABD857D9-65CE-4812-BA1C-14DBEC131A33Q33599125-AA778D26-6B6C-4F7B-9B8C-7F61E568A405Q33604205-9F7EFE94-8A0E-45B5-A431-8404261F5E6FQ33608875-DEF88501-840F-44F8-AF07-5D651B3C8F55Q33774783-013D25AD-A053-44BA-8D23-DEE8676B5EB2Q33867595-34401436-C203-4FAE-B6E6-64136EBC5E44Q33906270-1AA82284-F0A8-4B91-A7D5-B7955C0028ACQ34006170-CC4A4144-AA12-4BB8-A299-2F07328B66D1Q34060206-DE04A6DF-BED1-4133-AF6C-CEC073D9B59BQ34074976-AEDBA866-AD28-4195-880B-8874C34E0228Q34183889-92D336F8-97F5-4B8E-B59D-ED39D484AEC4Q34198040-47FE12D0-7975-4974-AF9B-9646E1C26476Q34240870-1E0979C3-19AE-44EE-BA13-6A0ADE53E6A4Q34247741-64BE909B-3E52-4608-A63D-7AD5D9174397Q34253403-CCF8915E-477F-454D-8F93-E0177614156CQ34253619-69AFF4A0-D48D-416D-A87E-4B2B338FC3BEQ34334346-59FC67AE-97A5-43C2-8F64-C2D26CCBE631Q34395347-124202E9-A179-45A5-BA48-24FFFEE2D594Q34498927-A9C446A0-F5D6-4429-A34E-2B49872A0473Q34554218-28C5DAEF-6182-4FB5-8C9A-D76F0EB965A5Q34581136-B006CC45-EDCB-4829-BC9D-217BC4B80B55Q34632004-731C72B0-D604-4640-9707-9732F2F739A0Q34768553-25AD0444-5A54-4665-A3D7-12B12789F6F3Q34926866-0BA9068D-CBC9-4696-BAD5-7EEBA58513CCQ35011162-5CDA64EF-4BEF-4C20-9186-31794DCD6693Q35107961-C3BB2BEB-D5E0-49A9-B24A-22FD684FEDF2Q35131293-24F0476B-F8E9-447D-A0BF-0F58F244EE1EQ35335499-785D8171-BF26-4F96-BC03-2CDF05AE445EQ35361740-7D199EAD-8B81-43C7-BDC3-C0777E5044FBQ35411519-CF20CBB9-5C52-479C-8ACF-5849DF58B1D1Q35530704-9BDB58D4-49DF-4767-9FE9-F563FE5E8195
P2860
Participation of tumor necrosis factor in the mediation of gram negative bacterial lipopolysaccharide-induced injury in rabbits.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Participation of tumor necrosi ...... ide-induced injury in rabbits.
@en
type
label
Participation of tumor necrosi ...... ide-induced injury in rabbits.
@en
prefLabel
Participation of tumor necrosi ...... ide-induced injury in rabbits.
@en
P2093
P2860
P356
P1476
Participation of tumor necrosi ...... ide-induced injury in rabbits.
@en
P2093
J C Mathison
R J Ulevitch
P2860
P304
P356
10.1172/JCI113540
P407
P577
1988-06-01T00:00:00Z