Comparison of the in vitro transforming activities of human papillomavirus types.
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The region of the HPV E7 oncoprotein homologous to adenovirus E1a and Sv40 large T antigen contains separate domains for Rb binding and casein kinase II phosphorylationRHBDF2 mutations are associated with tylosis, a familial esophageal cancer syndromeBinding of integrin alpha6beta4 to plectin prevents plectin association with F-actin but does not interfere with intermediate filament bindingHPV16 E6 and E7 proteins cooperate to immortalize human foreskin keratinocytesPapillomavirus E6 oncoproteins.Intermediate filament-membrane attachments function synergistically with actin-dependent contacts to regulate intercellular adhesive strengthNonradioactive RNA in situ hybridization detection of human papillomavirus 16-E7 transcripts in squamous cell carcinomas of the uterine cervix using confocal laser scan microscopy.Interaction of the human papillomavirus type 16 E6 oncoprotein with wild-type and mutant human p53 proteinsThe E7 oncoprotein associates with Mi2 and histone deacetylase activity to promote cell growth.Recurrent respiratory papillomatosis: an overview of current thinking and treatmentGene expression profile of cervical and skin tissues from human papillomavirus type 16 E6 transgenic mice.Continued expression of HPV-16 E7 protein is required for maintenance of the transformed phenotype of cells co-transformed by HPV-16 plus EJ-ras.Human papillomavirus 16 oncoprotein E7 stimulates UBF1-mediated rDNA gene transcription, inhibiting a p53-independent activity of p14ARFComplex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product.Human papillomavirus type 16 cooperates with activated ras and fos oncogenes in the hormone-dependent transformation of primary mouse cells.Dual expression of Epstein-Barr virus, latent membrane protein-1 and human papillomavirus-16 E6 transform primary mouse embryonic fibroblasts through NF-κB signalingDifferential effects of human papillomavirus type 6, 16, and 18 DNAs on immortalization and transformation of human cervical epithelial cellsThe E6 and E7 genes of human papillomavirus type 6 have weak immortalizing activity in human epithelial cells.Structure-function analysis of the human papillomavirus type 16 E7 oncoprotein.Targeted degradation of the retinoblastoma protein by human papillomavirus E7-E6 fusion proteins.Properties of p53 mutations detected in primary and secondary cervical cancers suggest mechanisms of metastasis and involvement of environmental carcinogens.The transforming and suppressor functions of p53 alleles: effects of mutations that disrupt phosphorylation, oligomerization and nuclear translocation.Characterization of in vivo expression of the human papillomavirus type 16 E4 protein in cervical biopsy tissues.Constitutive expression of c-myc oncogene confers hormone independence and enhanced growth-factor responsiveness on cells transformed by human papilloma virus type 16.The upstream regulatory region of human papillomavirus type 31 is insensitive to glucocorticoid induction.HLA class I binding promiscuity of the CD8 T-cell epitopes of human papillomavirus type 16 E6 protein.The constitutively expressed octamer binding protein OTF-1 and a novel octamer binding protein expressed specifically in cervical cells bind to an octamer-related sequence in the human papillomavirus 16 enhancer.The HPV 16 genome induces carcinomas and T-cell lymphomas in transgenic miceTranscription of the transforming genes of the oncogenic human papillomavirus-16 is stimulated by tumor promotors through AP1 binding sites.Effect of human papillomavirus type 16 oncogenes on MAP kinase activity.Both conserved region 1 (CR1) and CR2 of the human papillomavirus type 16 E7 oncogene are required for induction of epidermal hyperplasia and tumor formation in transgenic mice.Human papillomavirus type 16 E6 and E7 oncogenes abrogate radiation-induced DNA damage responses in vivo through p53-dependent and p53-independent pathways.Human papillomavirus type 18 DNA and E6-E7 mRNA are detected in squamous cell carcinoma and adenocarcinoma of the lung.Nuclear proto-oncogene products transactivate the human papillomavirus type 16 promoter.Human papillomavirus type 18 is associated with less apoptosis in fibroblast tumours than human papillomavirus type 16.Synergy between the Mos/mitogen-activated protein kinase pathway and loss of p53 function in transformation and chromosome instability.Elderly Japanese women with cervical carcinoma show higher proportions of both intermediate-risk human papillomavirus types and p53 mutationsHuman papillomavirus type 11 E2 proteins repress the homologous E6 promoter by interfering with the binding of host transcription factors to adjacent elements.Human papillomaviruses: are we ready to type?Human papillomavirus infection as a prognostic factor in oropharyngeal squamous cell carcinomas treated in a prospective phase II clinical trial.
P2860
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P2860
Comparison of the in vitro transforming activities of human papillomavirus types.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Comparison of the in vitro transforming activities of human papillomavirus types.
@en
type
label
Comparison of the in vitro transforming activities of human papillomavirus types.
@en
prefLabel
Comparison of the in vitro transforming activities of human papillomavirus types.
@en
P2093
P2860
P1433
P1476
Comparison of the in vitro transforming activities of human papillomavirus types
@en
P2093
P2860
P304
P356
10.1002/J.1460-2075.1988.TB03013.X
P407
P50
P577
1988-06-01T00:00:00Z