Host-range restriction of vaccinia virus E3L-specific deletion mutants.
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Replication of subgenomic hepatitis C virus replicons in mouse fibroblasts is facilitated by deletion of interferon regulatory factor 3 and expression of liver-specific microRNA 122Potential for Improving Potency and Specificity of Reovirus Oncolysis with Next-Generation Reovirus VariantsHost-range restriction of vaccinia virus E3L deletion mutant can be overcome in vitro, but not in vivo, by expression of the influenza virus NS1 proteinPoxvirus tropismA survey of host range genes in poxvirus genomesGeneration of a complete single-gene knockout bacterial artificial chromosome library of cowpox virus and identification of its essential genesReplication of modified vaccinia virus Ankara in primary chicken embryo fibroblasts requires expression of the interferon resistance gene E3L.Vaccinia virus E3 protein prevents the antiviral action of ISG15Both carboxy- and amino-terminal domains of the vaccinia virus interferon resistance gene, E3L, are required for pathogenesis in a mouse model.Biological function of the vaccinia virus Z-DNA-binding protein E3L: gene transactivation and antiapoptotic activity in HeLa cells.Antisense RNA: function and fate of duplex RNA in cells of higher eukaryotes.C7L family of poxvirus host range genes inhibits antiviral activities induced by type I interferons and interferon regulatory factor 1.Complementation of vaccinia virus lacking the double-stranded RNA-binding protein gene E3L by human cytomegalovirusRoles of vaccinia virus genes E3L and K3L and host genes PKR and RNase L during intratracheal infection of C57BL/6 mice.Genomes of the parapoxviruses ORF virus and bovine papular stomatitis virus.Recombinant modified vaccinia virus Ankara generating excess early double-stranded RNA transiently activates protein kinase R and triggers enhanced innate immune responses.Modified vaccinia virus Ankara triggers type I IFN production in murine conventional dendritic cells via a cGAS/STING-mediated cytosolic DNA-sensing pathwayVirus infection rapidly activates the P58(IPK) pathway, delaying peak kinase activation to enhance viral replication.Identification of 10 cowpox virus proteins that are necessary for induction of hemorrhagic lesions (red pocks) on chorioallantoic membranes.Poxvirus decapping enzymes enhance virulence by preventing the accumulation of dsRNA and the induction of innate antiviral responsesComparative analysis of poxvirus orthologues of the vaccinia virus E3 protein: modulation of protein kinase R activity, cytokine responses, and virus pathogenicityEctopic expression of vaccinia virus E3 and K3 cannot rescue ectromelia virus replication in rabbit RK13 cellsDouble-stranded RNA is a trigger for apoptosis in vaccinia virus-infected cells.The amino terminus of the vaccinia virus E3 protein is necessary to inhibit the interferon responseDistinct gene expression profiling after infection of immature human monocyte-derived dendritic cells by the attenuated poxvirus vectors MVA and NYVAC.A Single Amino Acid Dictates Protein Kinase R Susceptibility to Unrelated Viral Antagonists.Antagonism of the protein kinase R pathway by the guinea pig cytomegalovirus US22-family gene gp145.The infectious bursal disease virus RNA-binding VP3 polypeptide inhibits PKR-mediated apoptosisExpression of the E3L gene of vaccinia virus in transgenic mice decreases host resistance to vaccinia virus and Leishmania major infections.Vaccinia virus subverts a mitochondrial antiviral signaling protein-dependent innate immune response in keratinocytes through its double-stranded RNA binding protein, E3.Vaccinia viruses with mutations in the E3L gene as potential replication-competent, attenuated vaccines: intra-nasal vaccinationDouble-stranded RNA binding by the human cytomegalovirus PKR antagonist TRS1Vaccinia virus E3 suppresses expression of diverse cytokines through inhibition of the PKR, NF-kappaB, and IRF3 pathways.Involvement of the cellular phosphatase DUSP1 in vaccinia virus infection.Dendritic cell subtypes as primary targets of vaccines: the emerging role and cross-talk of pattern recognition receptors.Vaccinia virus K1L and C7L inhibit antiviral activities induced by type I interferons.Myxoma Virus dsRNA Binding Protein M029 Inhibits the Type I IFN-Induced Antiviral State in a Highly Species-Specific Fashion.Silencing suppressors: viral weapons for countering host cell defenses.Suppression of proinflammatory signal transduction and gene expression by the dual nucleic acid binding domains of the vaccinia virus E3L proteins.Role of viral factor E3L in modified vaccinia virus ankara infection of human HeLa Cells: regulation of the virus life cycle and identification of differentially expressed host genes.
P2860
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P2860
Host-range restriction of vaccinia virus E3L-specific deletion mutants.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Host-range restriction of vaccinia virus E3L-specific deletion mutants.
@en
type
label
Host-range restriction of vaccinia virus E3L-specific deletion mutants.
@en
prefLabel
Host-range restriction of vaccinia virus E3L-specific deletion mutants.
@en
P2093
P356
P1433
P1476
Host-range restriction of vaccinia virus E3L-specific deletion mutants.
@en
P2093
B L Jacobs
E B Kauffman
E Paoletti
H Martinez
J Tartaglia
M E Perkus
P2888
P356
10.1007/BF00370005
P577
1996-01-01T00:00:00Z
P6179
1021911686