Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent.

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Roles of impaired intracellular calcium cycling in arrhythmogenicity of diabetic mouse model.CaMKII blockade, cardiac conduction, and arrhythmia.Response to the letter from Warren et al.

P2860

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P2860

Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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Calmodulin/CaMKII inhibition i ...... tions when fibrosis is absent.

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Calmodulin/CaMKII inhibition i ...... tions when fibrosis is absent.

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Calmodulin/CaMKII inhibition i ...... tions when fibrosis is absent.

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Antonia J A Raaijmakers

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Bart Kok

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Haruo Honjo

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Helen Driessen

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Hiroki Takanari

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Itsuo Kodama

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John A Jansen

http://www.w3.org/2001/XMLSchema#string

Kaichiro Kamiya

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Leonie van Stuijvenberg

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Magda S C Fontes

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P2860

Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release

Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sites

New therapeutic targets in cardiology: arrhythmias and Ca2+/calmodulin-dependent kinase II (CaMKII)

Identification and expression of delta-isoforms of the multifunctional Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human myocardium

CaMKIIδB mediates aberrant NCX1 expression and the imbalance of NCX1/SERCA in transverse aortic constriction-induced failing heart

Gap junction remodelling in human heart failure is associated with increased interaction of connexin43 with ZO-1.

Cardiac CaM Kinase II genes δ and γ contribute to adverse remodeling but redundantly inhibit calcineurin-induced myocardial hypertrophy.

Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels

Identification of CaMKII phosphorylation sites in Connexin43 by high-resolution mass spectrometry.

Comparison of the IKr blockers moxifloxacin, dofetilide and E-4031 in five screening models of pro-arrhythmia reveals lack of specificity of isolated cardiomyocytes

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10.1093/CVR/CVW173

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111

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Mohamed Boulaksil

M A van der Heijden

Harold V M van Rijen

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2016-06-29T00:00:00Z

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27357638

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Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent.

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P2860

P2860

Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent.

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