Apoptosis induced by withdrawal of interleukin-3 (IL-3) from an IL-3-dependent hematopoietic cell line is associated with repartitioning of intracellular calcium and is blocked by enforced Bcl-2 oncoprotein production.
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Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxesEndoplasmic reticulum stress: cell life and death decisions.IRS-4 mediates protein kinase B signaling during insulin stimulation without promoting antiapoptosisTransformation of hematopoietic cells and activation of JAK2-V617F by IL-27R, a component of a heterodimeric type I cytokine receptorTemporal changes in chromatin, intracellular calcium, and poly(ADP-ribose) polymerase during Sindbis virus-induced apoptosis of neuroblastoma cellsER Stress-Mediated Signaling: Action Potential and Ca(2+) as Key PlayersRegulation of endothelial monocyte-activating polypeptide II release by apoptosisThe functions of cytokines and their uses in toxicologyCaMKII is involved in cadmium activation of MAPK and mTOR pathways leading to neuronal cell deathSurvival signaling by C-RAF: mitochondrial reactive oxygen species and Ca2+ are critical targetsCalcium signaling and cytotoxicity.Role of the Bcl-2 gene family in prostate cancer progression and its implications for therapeutic intervention.Impact of conditioning hyperglycemic on myocardial infarction rats: Cardiac cell survival factors.Bcl-2 and Bcl-X(L) block thapsigargin-induced nitric oxide generation, c-Jun NH(2)-terminal kinase activity, and apoptosisThe effects of calcitonin on the development of and Ca2+ levels in heat-shocked bovine preimplantation embryos in vitro.Bcl-X(L) affects Ca(2+) homeostasis by altering expression of inositol 1,4,5-trisphosphate receptorsMultiple functions of BCL-2 family proteins.Odontoblast-targeted Bcl-2 overexpression impairs dentin formation.A reversible component of mitochondrial respiratory dysfunction in apoptosis can be rescued by exogenous cytochrome c.Small-molecule anticancer compounds selectively target the hemopexin domain of matrix metalloproteinase-9Bcl-2 decreases the free Ca2+ concentration within the endoplasmic reticulum.Apoptosis and tumorigenesis in human cholangiocarcinoma cells. Involvement of Fas/APO-1 (CD95) and calmodulinRegulation and function of Bcl-2 during differentiation-induced cell death in HL-60 promyelocytic cells.Immunohistochemical analysis of Mcl-1 and Bcl-2 proteins in normal and neoplastic lymph nodesBcl-2 family in inter-organelle modulation of calcium signaling; roles in bioenergetics and cell survival.Commitment to apoptosis is associated with changes in mitochondrial biogenesis and activity in cell lines conditionally immortalized with simian virus 40.Bcl-2 and the regulation of programmed cell death.R-Ras promotes apoptosis caused by growth factor deprivation via a Bcl-2 suppressible mechanism.Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosisMaintenance of calcium homeostasis in the endoplasmic reticulum by Bcl-2Towards predictive models of stem cell fate.Bcl-2 functionally interacts with inositol 1,4,5-trisphosphate receptors to regulate calcium release from the ER in response to inositol 1,4,5-trisphosphate.Changes in endoplasmic reticulum luminal environment affect cell sensitivity to apoptosis.Lyn, Jak2, and Raf-1 kinases are critical for the antiapoptotic effect of interleukin 5, whereas only Raf-1 kinase is essential for eosinophil activation and degranulation.Increased sensitivity to apoptotic stimuli in c-abl-deficient progenitor B-cell linesBcl-2 immunoreactive neurons are differentially distributed in subregions of the amygdala and hippocampus of the adult macaque.TRPC1-mediated inhibition of 1-methyl-4-phenylpyridinium ion neurotoxicity in human SH-SY5Y neuroblastoma cells.Regulation of Bcl-xL expression in human keratinocytes by cell-substratum adhesion and the epidermal growth factor receptor.Channel formation by antiapoptotic protein Bcl-2.Resuscitation with 100%, compared with 21%, oxygen following brief, repeated periods of apnea can protect vulnerable neonatal brain regions from apoptotic injury
P2860
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P2860
Apoptosis induced by withdrawal of interleukin-3 (IL-3) from an IL-3-dependent hematopoietic cell line is associated with repartitioning of intracellular calcium and is blocked by enforced Bcl-2 oncoprotein production.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Apoptosis induced by withdrawa ...... Bcl-2 oncoprotein production.
@en
type
label
Apoptosis induced by withdrawa ...... Bcl-2 oncoprotein production.
@en
prefLabel
Apoptosis induced by withdrawa ...... Bcl-2 oncoprotein production.
@en
P2093
P1476
Apoptosis induced by withdrawa ...... Bcl-2 oncoprotein production.
@en
P2093
P304
P407
P577
1993-03-01T00:00:00Z