mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia. - Wikidata - awgldk - TriplyDB

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

http://www.wikidata.org/entity/Q41770752

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mTOR/p70S6K signaling distinguishes routine, maintenance-level autophagy from autophagic cell death during influenza A infection Erythropoietin: new directions for the nervous system Cutting through the complexities of mTOR for the treatment of stroke Targeting disease through novel pathways of apoptosis and autophagy Shedding new light on neurodegenerative diseases through the mammalian target of rapamycin Disruptive environmental chemicals and cellular mechanisms that confer resistance to cell death Upregulation of glutamate transporter GLT-1 by mTOR-Akt-NF-кB cascade in astrocytic oxygen-glucose deprivation Caspase-3 activation as a bifurcation point between plasticity and cell death.Rapamycin and interleukin-1β impair brain-derived neurotrophic factor-dependent neuron survival by modulating autophagy.Hepatitis C virus NS5A activates the mammalian target of rapamycin (mTOR) pathway, contributing to cell survival by disrupting the interaction between FK506-binding protein 38 (FKBP38) and mTOR Prion protein interaction with stress-inducible protein 1 enhances neuronal protein synthesis via mTOR PRAS40 is an integral regulatory component of erythropoietin mTOR signaling and cytoprotection The mTOR kinase inhibitor rapamycin decreases iNOS mRNA stability in astrocytes.Spatial and cellular characterization of mTORC1 activation after spinal cord injury reveals biphasic increase mainly attributed to microglia/macrophages.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.Activation of the mTOR pathway by low levels of xenoestrogens in breast epithelial cells from high-risk women.Cardiovascular disease and mTOR signaling Inhibition of mTOR Pathway by Rapamycin Reduces Brain Damage in Rats Subjected to Transient Forebrain Ischemia.A Critical Kinase Cascade in Neurological Disorders: PI 3-K, Akt, and mTOR.Maternal Body Weight and Gestational Diabetes Differentially Influence Placental and Pregnancy Outcomes Oxidant stress and signal transduction in the nervous system with the PI 3-K, Akt, and mTOR cascade Wnt1 inducible signaling pathway protein 1 (WISP1) targets PRAS40 to govern β-amyloid apoptotic injury of microglia.mTOR: on target for novel therapeutic strategies in the nervous system.Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.Therapeutic approaches to epileptogenesis--hope on the horizon.mTOR kinase, a key player in the regulation of glial functions: relevance for the therapy of multiple sclerosis.Exploring novel therapeutic targets in GIST: focus on the PI3K/Akt/mTOR pathway.Role of autophagy in the pathogenesis of multiple sclerosis.Ghrelin and gastrointestinal stromal tumors.Sunitinib induces apoptosis in pheochromocytoma tumor cells by inhibiting VEGFR2/Akt/mTOR/S6K1 pathways through modulation of Bcl-2 and BAD.Deficiency of Factor VII activating protease alters the outcome of ischemic stroke in mice.Ischemic post-conditioning facilitates brain recovery after stroke by promoting Akt/mTOR activity in nude rats Effects of rapamycin on cerebral oxygen supply and consumption during reperfusion after cerebral ischemia.Mammalian target of rapamycin cell signaling pathway contributes to the protective effects of ischemic postconditioning against stroke.Loss of GSK-3 Causes Abnormal Astrogenesis and Behavior in Mice.The mechanistic target of rapamycin (mTOR) pathway and S6 Kinase mediate diazoxide preconditioning in primary rat cortical neurons.MicroRNA-145 as one negative regulator of astrogliosis.MicroRNA-27a Promotes Inefficient Lysosomal Clearance in the Hippocampi of Rats Following Chronic Brain Hypoperfusion.Ischemia-Induced Changes of PRAS40 and p-PRAS40 Immunoreactivities in the Gerbil Hippocampal CA1 Region After Transient Cerebral Ischemia.Role of mTORC1 Controlling Proteostasis after Brain Ischemia.

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P2860

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

http://www.wikidata.org/entity/Q41770752

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

2009年论文

@zh

2009年论文

@zh-cn

name

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

@en

type

label

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

@en

prefLabel

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

@en

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JBC.M109.033100

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Journal of Biological Chemistry

P1476

mTOR/S6 kinase pathway contributes to astrocyte survival during ischemia.

@en

P2093

Isaac García-Yébenes

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José Manuel Pérez-Ortiz

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Mario Pende

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María Dolores Pastor

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Noelia Fradejas

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Pedro Tranque

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Silvia Mora-Lee

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Soledad Calvo

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P2860

The mTOR/PI3K and MAPK pathways converge on eIF4B to control its phosphorylation and activity

FGF-2 protects small cell lung cancer cells from apoptosis through a complex involving PKCepsilon, B-Raf and S6K2

Regulation of mTOR function in response to hypoxia by REDD1 and the TSC1/TSC2 tumor suppressor complex

S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

TOR signaling in growth and metabolism

TSC2 mediates cellular energy response to control cell growth and survival

SKAR is a specific target of S6 kinase 1 in cell growth control

mTOR and S6K1 mediate assembly of the translation preinitiation complex through dynamic protein interchange and ordered phosphorylation events

Hierarchical phosphorylation of the translation inhibitor 4E-BP1

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