Potassium- and acetylcholine-induced vasorelaxation in mice lacking endothelial nitric oxide synthase.
about
Endothelial cell dysfunction and the vascular complications associated with type 2 diabetes: assessing the health of the endotheliumMultiple mechanisms of vascular smooth muscle relaxation by the activation of proteinase-activated receptor 2 in mouse mesenteric arteriolesNeuronal nitric oxide synthase (NOS) regulates leukocyte-endothelial cell interactions in endothelial NOS deficient miceTwenty-five years since the discovery of endothelium-derived relaxing factor (EDRF): does a dysfunctional endothelium contribute to the development of type 2 diabetes?Interaction between nitric oxide signaling and gap junctions: effects on vascular functionTRPV4-mediated endothelial Ca2+ influx and vasodilation in response to shear stress.Cell-cell communication in the vessel wall.Cigarette smoking impairs Na+-K+-ATPase activity in the human coronary microcirculationEndothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouseEndothelium-derived reactive oxygen species: their relationship to endothelium-dependent hyperpolarization and vascular tone.Role of endothelium-derived hyperpolarizing factor in endothelial dysfunction during diabetes.Spironolactone treatment attenuates vascular dysfunction in type 2 diabetic mice by decreasing oxidative stress and restoring NO/GC signalingPathophysiology of hypertension in the absence of nitric oxide/cyclic GMP signaling.KCa 3.1 channels maintain endothelium-dependent vasodilatation in isolated perfused kidneys of spontaneously hypertensive rats after chronic inhibition of NOS.Crucial role of nitric oxide synthases system in endothelium-dependent hyperpolarization in mice.Transient receptor potential vanilloid type 4-deficient mice exhibit impaired endothelium-dependent relaxation induced by acetylcholine in vitro and in vivoCalcium-activated potassium channels and endothelial dysfunction: therapeutic options?A role for nitroxyl (HNO) as an endothelium-derived relaxing and hyperpolarizing factor in resistance arteries.Compensatory Vasodilator Mechanisms in the Ophthalmic Artery of Endothelial Nitric Oxide Synthase Gene Knockout Mice.Comparison of the pharmacological properties of EDHF-mediated vasorelaxation in guinea-pig cerebral and mesenteric resistance vessels.Determinants of renal microvascular response to ACh: afferent and efferent arteriolar actions of EDHF.Hyperpolarization of murine small caliber mesenteric arteries by activation of endothelial proteinase-activated receptor 2.Endothelium-derived hyperpolarizing factor as an in vivo back-up mechanism in the cutaneous microcirculation in old mice.Contributions of endothelium-derived relaxing factors to control of hindlimb blood flow in the mouse in vivo.Hyperglycaemia disrupts conducted vasodilation in the resistance vasculature of db/db mice.
P2860
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P2860
Potassium- and acetylcholine-induced vasorelaxation in mice lacking endothelial nitric oxide synthase.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Potassium- and acetylcholine-i ...... thelial nitric oxide synthase.
@en
type
label
Potassium- and acetylcholine-i ...... thelial nitric oxide synthase.
@en
prefLabel
Potassium- and acetylcholine-i ...... thelial nitric oxide synthase.
@en
P2860
P356
P1476
Potassium- and acetylcholine-i ...... thelial nitric oxide synthase.
@en
P2860
P304
P356
10.1038/SJ.BJP.0703144
P407
P577
2000-03-01T00:00:00Z