Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
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Is inflammation a consequence of extracellular hyperosmolarity?The role of hyperosmotic stress in inflammation and diseaseMechanisms Underlying Dysregulation of Electrolyte Absorption in Inflammatory Bowel Disease-Associated DiarrheaSte20-related proline/alanine-rich kinase (SPAK) regulated transcriptionally by hyperosmolarity is involved in intestinal barrier function.Bacterial protein signals are associated with Crohn's disease.Immunodeficiency and autoimmune enterocolopathy linked to NFAT5 haploinsufficiencyCaV1.3 channels and intracellular calcium mediate osmotic stress-induced N-terminal c-Jun kinase activation and disruption of tight junctions in Caco-2 CELL MONOLAYERS.Sodium channel γENaC mediates IL-17 synergized high salt induced inflammatory stress in breast cancer cells.Inhibitory phosphorylation of GSK-3β by AKT, PKA, and PI3K contributes to high NaCl-induced activation of the transcription factor NFAT5 (TonEBP/OREBP)NFAT5/STAT3 interaction mediates synergism of high salt with IL-17 towards induction of VEGF-A expression in breast cancer cellsc-Jun NH2-terminal kinase-2 mediates osmotic stress-induced tight junction disruption in the intestinal epithelium.Hyperosmotic stress stimulates autophagy via polycystin-2.Hyperosmotic stress induces nuclear factor-kappaB activation and interleukin-8 production in human intestinal epithelial cells.Role of NFAT5 in inflammatory disorders associated with osmotic stress.Protein Kinase R Mediates the Inflammatory Response Induced by Hyperosmotic Stress.Electrolyte and acid-base disorders in inflammatory bowel disease.New therapeutic targets in ulcerative colitis: the importance of ion transporters in the human colon.Halophilic archaea in the human intestinal mucosa.Hyperosmotic stress contributes to mouse colonic inflammation through the methylation of protein phosphatase 2A.Preferential phosphorylation of focal adhesion kinase tyrosine 861 is critical for mediating an anti-apoptotic response to hyperosmotic stress.Faecal osmolality and electrolyte concentrations in chronic diarrhoea: do they provide diagnostic clues?Toward a Reasoned Classification of Diseases Using Physico-Chemical Based Phenotypes.Plasma and skeletal muscle electrolytes in patients with Crohn's disease.SLC6A14, an amino acid transporter, modifies the primary CF defect in fluid secretion
P2860
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P2860
Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年論文
@yue
1982年論文
@zh-hant
1982年論文
@zh-hk
1982年論文
@zh-mo
1982年論文
@zh-tw
1982年论文
@wuu
1982年论文
@zh
1982年论文
@zh-cn
name
Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
@en
type
label
Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
@en
prefLabel
Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
@en
P2093
P2860
P356
P1433
P1476
Comparison of the composition of faecal fluid in Crohn's disease and ulcerative colitis.
@en
P2093
Bernstein J
Gnaedinger A
P2860
P304
P356
10.1136/GUT.23.4.326
P407
P577
1982-04-01T00:00:00Z