Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury? - Wikidata - awgldk - TriplyDB

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

http://www.wikidata.org/entity/Q42044272

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Ischemic A/D transition of mitochondrial complex I and its role in ROS generation Identification of S-nitrosated mitochondrial proteins by S-nitrosothiol difference in gel electrophoresis (SNO-DIGE): implications for the regulation of mitochondrial function by reversible S-nitrosation Nitric oxide-associated protein 1 (NOA1) is necessary for oxygen-dependent regulation of mitochondrial respiratory complexes.Mitochondrial complex I deactivation is related to superoxide production in acute hypoxia.Hypoxia and aerobic metabolism adaptations of human endothelial cells.On the mechanism(s) of membrane permeability transition in liver mitochondria of lamprey, Lampetra fluviatilis L.: insights from cadmium Regulation of mitochondrial processes by protein S-nitrosylation.NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats Initiation of electron transport chain activity in the embryonic heart coincides with the activation of mitochondrial complex 1 and the formation of supercomplexes.The composition of plant mitochondrial supercomplexes changes with oxygen availability.Selective inhibition of deactivated mitochondrial complex I by biguanides Cells lacking Rieske iron-sulfur protein have a reactive oxygen species-associated decrease in respiratory complexes I and IV.Regional skeletal muscle remodeling and mitochondrial dysfunction in right ventricular heart failure.PP2A ligand ITH12246 protects against memory impairment and focal cerebral ischemia in mice.Conformational change of mitochondrial complex I increases ROS sensitivity during ischemia.Acidosis overrides oxygen deprivation to maintain mitochondrial function and cell survival.Characterisation of the active/de-active transition of mitochondrial complex I.Essential regions in the membrane domain of bacterial complex I (NDH-1): the machinery for proton translocation.Chemical modifications of respiratory complex I for structural and functional studies.Molecular mechanism and physiological role of active-deactive transition of mitochondrial complex I.Modulation of the conformational state of mitochondrial complex I as a target for therapeutic intervention.Benzothiazepine CGP37157 and its isosteric 2'-methyl analogue provide neuroprotection and block cell calcium entry Structure of mammalian respiratory complex I ND3, ND1 and 39kDa subunits are more exposed in the de-active form of bovine mitochondrial complex I.The deactive form of respiratory complex I from mammalian mitochondria is a Na+/H+ antiporter.VLDL Induced Modulation of Nitric Oxide Signalling and Cell Redox Homeostasis in HUVEC.Effects of metformin and other biguanides on oxidative phosphorylation in mitochondria.Effect of monovalent cations on the kinetics of hypoxic conformational change of mitochondrial complex I.'SNO'-Storms Compromise Protein Activity and Mitochondrial Metabolism in Neurodegenerative Disorders.Exercise training in Tgαq*44 mice during the progression of chronic heart failure: cardiac vs. peripheral (soleus muscle) impairments to oxidative metabolism.Intrauterine growth restriction impairs right ventricular response to hypoxia in adult male rats.Structure of the Deactive State of Mammalian Respiratory Complex I.Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure.Mitochondrial electron transport chain in heavy metal-induced neurotoxicity: effects of cadmium, mercury, and copper.Nonylphenol and Octylphenol Differently Affect Cell Redox Balance by Modulating the Nitric Oxide Signaling.Hypoxia and IF₁ Expression Promote ROS Decrease in Cancer Cells Kinetic control of oxygen consumption during contractions in self-perfused skeletal muscle Cryo-EM structures of complex I from mouse heart mitochondria in two biochemically defined states

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Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

http://www.wikidata.org/entity/Q42044272

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh

2009年學術文章

@zh-hant

name

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@en

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@nl

type

label

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@en

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@nl

prefLabel

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@en

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@nl

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JBC.M109.054346

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Journal of Biological Chemistry

P1476

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

@en

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Alexander Galkin

http://www.w3.org/2001/XMLSchema#string

Nanci Frakich

http://www.w3.org/2001/XMLSchema#string

Salvador Moncada

http://www.w3.org/2001/XMLSchema#string

P2860

How mitochondria produce reactive oxygen species

Nitric oxide inhibits mitochondrial NADH:ubiquinone reductase activity through peroxynitrite formation

Inactivation of aconitase and oxoglutarate dehydrogenase in skeletal muscle in vitro by superoxide anions and/or nitric oxide

Energy converting NADH:quinone oxidoreductase (complex I).

Active/de-active transition of respiratory complex I in bacteria, fungi, and animals.

Endogenous NO regulates superoxide production at low oxygen concentrations by modifying the redox state of cytochrome c oxidase

S-nitrosation of mitochondrial complex I depends on its structural conformation.

Identification of the mitochondrial ND3 subunit as a structural component involved in the active/deactive enzyme transition of respiratory complex I.

Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates.

Peroxynitrous acid--where is the hydroxyl radical?

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36055-36061

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scholarly article

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10.1074/JBC.M109.054346

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52

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284

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Michael R. Duchen

Andrey Y Abramov

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2009-10-27T00:00:00Z

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19861410

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