Tim1 and Tim3 are not essential for experimental allergic asthma
about
Galectin-9 enhances cytokine secretion, but suppresses survival and degranulation, in human mast cell lineDefect in regulatory B-cell function and development of systemic autoimmunity in T-cell Ig mucin 1 (Tim-1) mucin domain-mutant mice.T-cell immunoglobulin and mucin domain 1 deficiency eliminates airway hyperreactivity triggered by the recognition of airway cell death.TIM-3 is not essential for development of airway inflammation induced by house dust mite antigens.The role of TIM-containing molecules in airway disease and their potential as therapeutic targets.Developments in the field of allergy in 2011 through the eyes of Clinical and Experimental Allergy.Murine Tim-1 is excluded from the immunological synapseAre TIM proteins involved in asthma development or pathology?
P2860
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P2860
Tim1 and Tim3 are not essential for experimental allergic asthma
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Tim1 and Tim3 are not essential for experimental allergic asthma
@en
Tim1 and Tim3 are not essential for experimental allergic asthma
@nl
type
label
Tim1 and Tim3 are not essential for experimental allergic asthma
@en
Tim1 and Tim3 are not essential for experimental allergic asthma
@nl
prefLabel
Tim1 and Tim3 are not essential for experimental allergic asthma
@en
Tim1 and Tim3 are not essential for experimental allergic asthma
@nl
P2093
P2860
P1476
Tim1 and Tim3 are not essential for experimental allergic asthma
@en
P2093
A N J McKenzie
J L Barlow
S J Ballantyne
P2860
P304
P356
10.1111/J.1365-2222.2011.03728.X
P577
2011-04-07T00:00:00Z