CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation.
about
Recent advances in understanding cardiac contractility in health and diseaseCaMKII induces permeability transition through Drp1 phosphorylation during chronic β-AR stimulationIsoproterenol Promotes Rapid Ryanodine Receptor Movement to Bridging Integrator 1 (BIN1)-Organized Dyads.Reductions in the Cardiac Transient Outward K+ Current Ito Caused by Chronic β-Adrenergic Receptor Stimulation Are Partly Rescued by Inhibition of Nuclear Factor κB.Assessing structural and functional responses of murine hearts to acute and sustained β-adrenergic stimulation in vivo.Calcium/Calmodulin-Dependent Protein Kinase II Activity Persists During Chronic β-Adrenoceptor Blockade in Experimental and Human Heart Failure.Mechanistic and therapeutic perspectives for cardiac arrhythmias: beyond ion channels.Altered Mitochondrial Metabolism and Mechanosensation in the Failing Heart: Focus on Intracellular Calcium Signaling.CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α.Molecular and cellular neurocardiology: development, and cellular and molecular adaptations to heart disease.New insights into the complex effects of KChIP2 on calcium transientsApocynin prevents isoproterenol-induced cardiac hypertrophy in rat.Dioxygen and Metabolism; Dangerous Liaisons in Cardiac Function and Disease.CRABP1 protects the heart from isoproterenol-induced acute and chronic remodeling.Altered Repolarization Reserve in Failing Rabbit Ventricular Myocytes: Calcium and β-Adrenergic Effects on Delayed- and Inward-Rectifier Potassium Currents.Inhibition of calcium/calmodulin-dependent kinase II restores contraction and relaxation in isolated cardiac muscle from type 2 diabetic rats.Interplay Between Sub-Cellular Alterations of Calcium Release and T-Tubular Defects in Cardiac Diseases
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P2860
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca
@nl
CaMKIIδ mediates β-adrenergic ...... onic β-adrenergic stimulation.
@en
type
label
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca
@nl
CaMKIIδ mediates β-adrenergic ...... onic β-adrenergic stimulation.
@en
prefLabel
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca
@nl
CaMKIIδ mediates β-adrenergic ...... onic β-adrenergic stimulation.
@en
P2093
P2860
P50
P1476
CaMKIIδ mediates β-adrenergic ...... ronic β-adrenergic stimulation
@en
P2093
Andrew Willeford
Charles B B Gray
Haiyun Ling
Jeffrey R Erickson
Joan Heller Brown
Jonathan L Respress
Michael Grimm
Satyam Sarma
P2860
P304
P356
10.1016/J.YJMCC.2015.06.007
P577
2015-06-14T00:00:00Z