The role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1.
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Towards a rational design of an asymptomatic clinical herpes vaccine: the old, the new, and the unknownA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationInhibitors of signal peptide peptidase (SPP) affect HSV-1 infectivity in vitro and in vivo.Role of CD8+ T cells and lymphoid dendritic cells in protection from ocular herpes simplex virus 1 challenge in immunized mice.Associations of HLA-A, HLA-B and HLA-C alleles frequency with prevalence of herpes simplex virus infections and diseases across global populations: implication for the development of an universal CD8+ T-cell epitope-based vaccine.Blocking of PDL-1 interaction enhances primary and secondary CD8 T cell response to herpes simplex virus-1 infection.CD8α dendritic cells drive establishment of HSV-1 latencyEffects of thyroid hormone on HSV-1 gene regulation: implications in the control of viral latency and reactivation.Of mice and not humans: how reliable are animal models for evaluation of herpes CD8(+)-T cell-epitopes-based immunotherapeutic vaccine candidates?The herpes simplex virus 1 latency-associated transcript promotes functional exhaustion of virus-specific CD8+ T cells in latently infected trigeminal ganglia: a novel immune evasion mechanismA herpes simplex virus type 1 mutant disrupted for microRNA H2 with increased neurovirulence and rate of reactivation.Adaptive and innate transforming growth factor beta signaling impact herpes simplex virus 1 latency and reactivationCD11c controls herpes simplex virus 1 responses to limit virus replication during primary infection.Phenotypic and functional characterization of herpes simplex virus glycoprotein B epitope-specific effector and memory CD8+ T cells from symptomatic and asymptomatic individuals with ocular herpesInfluence of galectin-9/Tim-3 interaction on herpes simplex virus-1 latencyTwo microRNAs encoded within the bovine herpesvirus 1 latency-related gene promote cell survival by interacting with RIG-I and stimulating NF-κB-dependent transcription and beta interferon signaling pathways.PD-L1-expressing dendritic cells contribute to viral resistance during acute HSV-1 infectionCD8+ T Cells Play a Bystander Role in Mice Latently Infected with Herpes Simplex Virus 1.The herpes simplex virus type 1 latency-associated transcript inhibits phenotypic and functional maturation of dendritic cells.Maintenance of T cell function in the face of chronic antigen stimulation and repeated reactivation for a latent virus infection.Decreased reactivation of a herpes simplex virus type 1 (HSV-1) latency-associated transcript (LAT) mutant using the in vivo mouse UV-B model of induced reactivation.Rabbit and mouse models of HSV-1 latency, reactivation, and recurrent eye diseasesIncreased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2TIM-3 regulates innate immune cells to induce fetomaternal tolerance.Mucosal herpes immunity and immunopathology to ocular and genital herpes simplex virus infections.Current trends in negative immuno-synergy between two sexually transmitted infectious viruses: HIV-1 and HSV-1/2.The Herpes Simplex Virus Latency-Associated Transcript Gene Is Associated with a Broader Repertoire of Virus-Specific Exhausted CD8+ T Cells Retained within the Trigeminal Ganglia of Latently Infected HLA Transgenic RabbitsPD-L1/B7-H1 regulates the survival but not the function of CD8+ T cells in herpes simplex virus type 1 latently infected trigeminal ganglia.Asymptomatic memory CD8+ T cells: from development and regulation to consideration for human vaccines and immunotherapeuticsInterrelationship of Primary Virus Replication, Level of Latency, and Time to Reactivation in the Trigeminal Ganglia of Latently Infected Mice.Overexpression of herpes simplex virus glycoprotein K (gK) alters expression of HSV receptors in ocularly-infected miceThe PD-1/PD-L1 (B7-H1) pathway in chronic infection-induced cytotoxic T lymphocyte exhaustion.Control of HSV-1 latency in human trigeminal ganglia--current overview.Too much of a good thing? Tim-3 and TCR signaling in T cell exhaustion.Inclusion of CD80 in HSV targets the recombinant virus to PD-L1 on DCs and allows productive infection and robust immune responses.Interactions between herpesvirus entry mediator (TNFRSF14) and latency-associated transcript during herpes simplex virus 1 latency.The bitter side of sweet: the role of Galectin-9 in immunopathogenesis of viral infections.Immune response of T cells during herpes simplex virus type 1 (HSV-1) infection.Roles of M1 and M2 Macrophages in Herpes Simplex Virus 1 Infectivity.Coregulatory interactions among CD8α dendritic cells, the latency-associated transcript, and programmed death 1 contribute to higher levels of herpes simplex virus 1 latency.
P2860
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P2860
The role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
The role of LAT in increased C ...... d with herpes simplex virus 1.
@en
The role of LAT in increased C ...... d with herpes simplex virus 1.
@nl
type
label
The role of LAT in increased C ...... d with herpes simplex virus 1.
@en
The role of LAT in increased C ...... d with herpes simplex virus 1.
@nl
prefLabel
The role of LAT in increased C ...... d with herpes simplex virus 1.
@en
The role of LAT in increased C ...... d with herpes simplex virus 1.
@nl
P2093
P2860
P356
P1433
P1476
The role of LAT in increased C ...... d with herpes simplex virus 1.
@en
P2093
Arlene H Sharpe
Clinton Jones
David Gate
Dimosthenis Mantopoulos
Gordon J Freeman
Homayon Ghiasi
Kevin R Mott
Lbachir BenMohamed
Lixin Zheng
Pedram Hamrah
P2860
P304
P356
10.1128/JVI.02290-10
P407
P577
2011-02-09T00:00:00Z