Activation systems for latent matrix metalloproteinase-2 are upregulated immediately after focal cerebral ischemia.
about
Implications of MMP9 for Blood Brain Barrier Disruption and Hemorrhagic Transformation Following Ischemic StrokeAging and the neurovascular unitInhibition of MMP-2 but not MMP-9 influences inner ear spiral ganglion neurons in vitro.Molecular insights and therapeutic targets for blood-brain barrier disruption in ischemic stroke: critical role of matrix metalloproteinases and tissue-type plasminogen activatorHemostasis and alterations of the central nervous systemPlasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Relationship of neurovascular elements to neuron injury during ischemia.Matrix metalloproteinase-9 mediates hypoxia-induced vascular leakage in the brain via tight junction rearrangement.Infrared optical imaging of matrix metalloproteinases (MMPs) up regulation following ischemia reperfusion is ameliorated by hypothermia.Hypoxia-ischemia or excitotoxin-induced tissue plasminogen activator- dependent gelatinase activation in mice neonate brain microvessels.Inward remodeling of resistance arteries requires reactive oxygen species-dependent activation of matrix metalloproteinases.Activation of matrix metalloproteinases following anti-Aβ immunotherapy; implications for microhemorrhage occurrence.Bystander Effect Fuels Human Induced Pluripotent Stem Cell-Derived Neural Stem Cells to Quickly Attenuate Early Stage Neurological Deficits After Stroke.Microglial cell activation is a source of metalloproteinase generation during hemorrhagic transformation.The neurovascular unit, matrix proteases, and innate inflammation.Extracellular matrix and matrix receptors in blood-brain barrier formation and stroke.Diverse roles of matrix metalloproteinases and tissue inhibitors of metalloproteinases in neuroinflammation and cerebral ischemia.Immunologic privilege in the central nervous system and the blood-brain barrier.The rapid decrease in astrocyte-associated dystroglycan expression by focal cerebral ischemia is protease-dependent.Virchow's triad: the vascular basis of cerebral injuryUltrastructural and temporal changes of the microvascular basement membrane and astrocyte interface following focal cerebral ischemia.Inflammation and the neurovascular unit in the setting of focal cerebral ischemia.The fibrinolytic system and the regulation of lung epithelial cell proteolysis, signaling, and cellular viability.2-Chloroethanol Induced Upregulation of Matrix Metalloproteinase-2 in Primary Cultured Rat Astrocytes Via MAPK Signal Pathways.The neurovascular unit in the setting of stroke.Review: molecular pathogenesis of blood-brain barrier breakdown in acute brain injury.Matrix metalloproteinases and small artery remodeling.Targeting reactive nitrogen species: a promising therapeutic strategy for cerebral ischemia-reperfusion injury.Matrix metalloproteinases, new insights into the understanding of neurodegenerative disorders.Neuropathic pain: role of inflammation, immune response, and ion channel activity in central injury mechanisms.Spinal cord injury induced neuropathic pain: Molecular targets and therapeutic approaches.Matrix metalloproteinases as therapeutic targets for stroke.Matrix metalloproteinases in cerebral ischemia.New approach to identify ischemic stroke patients at risk to develop hemorrhagic transformation.Targeted drug delivery to ischemic stroke via chlorotoxin-anchored, lexiscan-loaded nanoparticles.The expression of matrix metalloproteinase-13 is increased in vessels with blood-brain barrier impairment in a stroke-prone hypertensive model.Cardiac ischemia-reperfusion injury induces matrix metalloproteinase-2 expression through the AP-1 components FosB and JunB.Gelatinase activity imaged by activatable cell-penetrating peptides in cell-based and in vivo models of stroke.Involvement of matrix metalloproteinases-2 and -9 in the formation of a lacuna-like cerebral cavity.Post-ischemic treatment with azithromycin protects ganglion cells against retinal ischemia/reperfusion injury in the rat.
P2860
Q26766038-5AE3653C-AF18-484A-88E0-5207C3331F6DQ27026843-00D7D538-7233-4CB1-B3A7-686582B060F6Q30414848-F380CB74-30AA-4AB8-B398-9C75C47E04B5Q33823616-A23B92F0-DBD2-4599-86A0-4C5B47C93B63Q33842161-C23B30E9-521D-47C3-B874-8DC596827321Q34018715-E3E28916-1E7E-4200-9775-BF05EE007248Q34038281-40D2BCF4-99D7-43B6-89E7-9961F790A5C1Q34170971-04606FD2-9357-46FB-ABF1-E8B505C525DCQ34319977-48B857B4-2872-41E9-A593-D95516414F82Q34937315-B2386A21-563B-4E29-9176-26A9E3322995Q35056901-2D4C83B3-C30D-4731-8181-098019B51D3DQ35240865-561A5967-48C1-4524-B91C-6B81920B6AC5Q35777502-2E7AE7D2-76EF-4BBF-9D4E-B653353F0D39Q35939162-C848C5E5-221F-4A3E-96EE-1D69F9CA98DCQ35990775-47D8AEE7-8500-47D3-81B3-BB387B5B858EQ36351793-BB3145B8-DB40-4C38-970E-C098C993FCADQ36644315-C7FE866D-13E4-4087-A582-141ACBCE6661Q36684162-C62F5A2B-881E-44EB-A163-4D7C4666AC67Q36986793-2DFFC1E4-79CE-4F28-AFD2-505D16A7DF89Q37223437-9313BF6C-AC18-48AC-B8E1-37660F3A85C1Q37264260-6D7DCC56-58B8-4A60-A44B-8D63E367F571Q37281874-D0943908-F547-4794-A10F-EC9123586A15Q37286882-23BB3068-4BB6-4BF2-B71F-5ED0A666075DQ37553275-21FA5DBC-CBD6-440D-8E49-8E067A4787D9Q37696584-E4E3827D-D051-48A5-91B9-A2AE5795E750Q37800133-516CC16D-A4C9-4FCB-A23E-84CAE4B1B8DAQ37962503-BEA96715-8E2D-4D68-AC88-C66841E63477Q38030379-1043F5C3-9FBF-43F5-B0A1-8901314DFC6EQ38151704-AA0611D0-AE9B-412B-BAB0-44E2789F5B80Q38247950-A676DDED-AEFC-46E4-B233-98155899A180Q38318458-7C91A8AF-55C4-4910-980D-C10FA03E9536Q38444452-30C978BE-DD5C-4DF5-9ABB-7BB2AA448BDEQ40028510-28447095-FF62-4D00-84A1-450B5D63B7ACQ41362038-E195CB4D-4824-4BCE-AE12-950430EDB3A1Q41654070-498C9D54-F3DA-4209-A122-30BABF089DE4Q43799003-95498EFF-90EB-4343-9AF6-9A45F3E61027Q46044860-9E87B1B9-A9C7-4CA5-BBCC-D5CC3498E59FQ46110252-48253EA7-0BE6-47FF-B806-3EF12303F3A2Q46150981-F6F0479B-5C36-4637-8C41-6DF56779CDFFQ47119067-531DBED6-C38A-46C7-8E3E-639420AE75A0
P2860
Activation systems for latent matrix metalloproteinase-2 are upregulated immediately after focal cerebral ischemia.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Activation systems for latent ...... after focal cerebral ischemia.
@en
Activation systems for latent ...... after focal cerebral ischemia.
@nl
type
label
Activation systems for latent ...... after focal cerebral ischemia.
@en
Activation systems for latent ...... after focal cerebral ischemia.
@nl
prefLabel
Activation systems for latent ...... after focal cerebral ischemia.
@en
Activation systems for latent ...... after focal cerebral ischemia.
@nl
P2093
P2860
P1476
Activation systems for latent ...... after focal cerebral ischemia
@en
P2093
Andrew P Mazar
Dae-Il Chang
Gregory J del Zoppo
Jacinta Lucero
Naohisa Hosomi
Takeo Abumiya
P2860
P304
P356
10.1097/01.WCB.0000091765.61714.30
P50
P577
2003-12-01T00:00:00Z
P6179
1017723260