Administration of green tea or caffeine enhances the disappearance of UVB-induced patches of mutant p53 positive epidermal cells in SKH-1 mice. - Wikidata - awgldk - TriplyDB

Administration of green tea or caffeine enhances the disappearance of UVB-induced patches of mutant p53 positive epidermal cells in SKH-1 mice.

Administration of green tea or caffeine enhances the disappearance of UVB-induced patches of mutant p53 positive epidermal cells in SKH-1 mice.

http://www.wikidata.org/entity/Q42476661

about

Cancer prevention with promising natural products: mechanisms of action and molecular targets A multi-targeted approach to suppress tumor-promoting inflammation Advances in chemical carcinogenesis: a historical review and prospective Stochastic fate of p53-mutant epidermal progenitor cells is tilted toward proliferation by UV B during preneoplasia.The enigmatic effects of caffeine in cell cycle and cancer ATR-Chk1 pathway inhibition promotes apoptosis after UV treatment in primary human keratinocytes: potential basis for the UV protective effects of caffeine.Inhibition of UVB-induced nonmelanoma skin cancer: a path from tea to caffeine to exercise to decreased tissue fat Tea, coffee, and caffeine and early-onset basal cell carcinoma in a case-control study.Botanical antioxidants in the prevention of photocarcinogenesis and photoaging.A preliminary study of the safety of red light phototherapy of tissues harboring cancer.Effects of high-fat diets rich in either omega-3 or omega-6 fatty acids on UVB-induced skin carcinogenesis in SKH-1 mice.Tea polyphenols for health promotion Inhibition of carcinogenesis by tea constituents Effect of caffeine on the ATR/Chk1 pathway in the epidermis of UVB-irradiated mice.Ultraviolet B regulation of transcription factor families: roles of nuclear factor-kappa B (NF-kappaB) and activator protein-1 (AP-1) in UVB-induced skin carcinogenesis Effect of caffeine on UVB-induced carcinogenesis, apoptosis, and the elimination of UVB-induced patches of p53 mutant epidermal cells in SKH-1 mice.Nitric oxide as a target of complementary and alternative medicines to prevent and treat inflammation and cancer.Natural agents: cellular and molecular mechanisms of photoprotection.The role of hair follicles in the percutaneous absorption of caffeine.In Vitro Chemopreventive Properties of Green Tea, Rooibos and Honeybush Extracts in Skin Cells.Tea polyphenols for the prevention of UVB-induced skin cancer.Follicular penetration of topically applied caffeine via a shampoo formulation.Natural products targeting the p53-MDM2 pathway and mutant p53: Recent advances and implications in cancer medicine

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P2860

Administration of green tea or caffeine enhances the disappearance of UVB-induced patches of mutant p53 positive epidermal cells in SKH-1 mice.

http://www.wikidata.org/entity/Q42476661

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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name

Administration of green tea or ...... epidermal cells in SKH-1 mice.

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Administration of green tea or ...... epidermal cells in SKH-1 mice.

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type

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Administration of green tea or ...... epidermal cells in SKH-1 mice.

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Administration of green tea or ...... epidermal cells in SKH-1 mice.

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Administration of green tea or ...... epidermal cells in SKH-1 mice.

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P2093

Allan H Conney

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Chung S Yang

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Jian-Guo Xie

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Jie Liao

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Qing-Yun Peng

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Yao-Ping Lu

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You-Rong Lou

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P2860

Early p53 alterations in mouse skin carcinogenesis by UVB radiation: immunohistochemical detection of mutant p53 protein in clusters of preneoplastic epidermal cells.

Escaping the stem cell compartment: sustained UVB exposure allows p53-mutant keratinocytes to colonize adjacent epidermal proliferating units without incurring additional mutations

Preferential binding of ATR protein to UV-damaged DNA.

Topical applications of caffeine or (-)-epigallocatechin gallate (EGCG) inhibit carcinogenesis and selectively increase apoptosis in UVB-induced skin tumors in mice

Cell cycle checkpoints and their impact on anticancer therapeutic strategies.

Frequent clones of p53-mutated keratinocytes in normal human skin

The Xenopus Chk1 protein kinase mediates a caffeine-sensitive pathway of checkpoint control in cell-free extracts

Mutation hotspots due to sunlight in the p53 gene of nonmelanoma skin cancers.

Solar ultraviolet radiation effects on biological systems.

ATR is not required for p53 activation but synergizes with p53 in the replication checkpoint.

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1465-1472

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10.1093/CARCIN/BGI086

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26

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15817611

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