Response: the role of RUNX1 isoforms in hematopoietic commitment of human pluripotent stem cells.

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Response: the role of RUNX1 isoforms in hematopoietic commitment of human pluripotent stem cells.

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2013 nî lūn-bûn

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2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

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2013年论文

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2013年论文

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name

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@en

Response: the role of RUNX1 is ...... human pluripotent stem cells.

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label

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@en

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@nl

prefLabel

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@en

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@nl

P1476

Response: the role of RUNX1 is ...... human pluripotent stem cells.

@en

P2093

Dan Ran

http://www.w3.org/2001/XMLSchema#string

Dan S Kaufman

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David A Knorr

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Dong-Er Zhang

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Jun-Bao Fan

http://www.w3.org/2001/XMLSchema#string

Kentson Lam

http://www.w3.org/2001/XMLSchema#string

Miao-Chia Lo

http://www.w3.org/2001/XMLSchema#string

Ming Yan

http://www.w3.org/2001/XMLSchema#string

Patrick I Ferrell

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Wei-Jong Shia

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P2860

Isoform-specific potentiation of stem and progenitor cell engraftment by AML1/RUNX1

Multiple functional domains of AML1: PU.1 and C/EBPalpha synergize with different regions of AML1

Runx1 isoforms show differential expression patterns during hematopoietic development but have similar functional effects in adult hematopoietic stem cells

Runx1 deletion or dominant inhibition reduces Cebpa transcription via conserved promoter and distal enhancer sites to favor monopoiesis over granulopoiesis.

RUNX1 and RUNX1-ETO: roles in hematopoiesis and leukemogenesis.

RUNX1a enhances hematopoietic lineage commitment from human embryonic stem cells and inducible pluripotent stem cells

Expansion of functionally defined mouse hematopoietic stem and progenitor cells by a short isoform of RUNX1/AML1.

RUNX1 and its fusion oncoprotein derivative, RUNX1-ETO, induce senescence-like growth arrest independently of replicative stress.

RUNX1 transformation of primary embryonic fibroblasts is revealed in the absence of p53.

Overexpression of an isoform of AML1 in acute leukemia and its potential role in leukemogenesis.

P304

5252-5253

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P31

scholarly article

P356

10.1182/BLOOD-2013-04-494914

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English

P433

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P478

121

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P50

Zhaohui Ye

Linzhao Cheng

P577

2013-06-01T00:00:00Z

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P698

23813938

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P932

3695367

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