Central inflammation and sickness-like behavior induced by the food contaminant deoxynivalenol: a PGE2-independent mechanism.
about
Advances in deoxynivalenol toxicity mechanisms: the brain as a targetThe food-contaminant deoxynivalenol modifies eating by targeting anorexigenic neurocircuitryEffect of deoxynivalenol and other Type B trichothecenes on the intestine: a reviewLeptin is required for hypothalamic regulation of miRNAs targeting POMC 3'UTR.Deoxynivalenol (Vomitoxin)-Induced Cholecystokinin and Glucagon-Like Peptide-1 Release in the STC-1 Enteroendocrine Cell Model Is Mediated by Calcium-Sensing Receptor and Transient Receptor Potential Ankyrin-1 ChannelMurine Anorectic Response to Deoxynivalenol (Vomitoxin) Is Sex-Dependent.High Sensitivity of Aged Mice to Deoxynivalenol (Vomitoxin)-Induced Anorexia Corresponds to Elevated Proinflammatory Cytokine and Satiety Hormone Responses.Anorexia induction by the trichothecene deoxynivalenol (vomitoxin) is mediated by the release of the gut satiety hormone peptide YYPeptide YY3-36 and 5-hydroxytryptamine mediate emesis induction by trichothecene deoxynivalenol (vomitoxin).The role of biomarkers in evaluating human health concerns from fungal contaminants in food.From the gut to the brain: journey and pathophysiological effects of the food-associated trichothecene mycotoxin deoxynivalenol.Obesity Impacts Fever and Sickness Behavior During Acute Systemic Inflammation.Does Dietary Deoxynivalenol Modulate the Acute Phase Reaction in Endotoxaemic Pigs?--Lessons from Clinical Signs, White Blood Cell Counts, and TNF-Alpha.Risks to human and animal health related to the presence of deoxynivalenol and its acetylated and modified forms in food and feedRelationships between body temperatures and inflammation indicators under physiological and pathophysiological conditions in pigs exposed to systemic lipopolysaccharide and dietary deoxynivalenol.
P2860
Q27009156-60A68895-8C78-448B-A36A-23017133F7AFQ28743954-FD61052A-67C1-4D59-88C1-1E3DE8BAB636Q33736548-2E02A4AE-CD97-4429-9D77-A8722C2DF7E6Q35575425-6740C2F1-D491-4F14-9888-4A196034031FQ35975380-5535E452-AE49-447D-8002-A28B137AA132Q35996593-47A5D320-2807-4AF4-B9F5-F92CCCF32EA6Q36226879-72EC2465-93A3-49EF-BC99-9396BDA407DBQ36752713-CDC664E8-4379-4069-9F4F-B755C469321AQ36769372-6009816A-7669-4C5F-B428-28B626851B5FQ38014991-23F9FEA6-0F66-42B6-B648-25A5EE2D424BQ38101247-13D79FA0-8AD9-4F8D-A269-AC0047D6947FQ38738358-76D81BE2-ED45-47C4-A3D5-7A2E92ABCA80Q40857599-3B671B8A-B856-4020-8A30-DE9FBDE2E579Q47160469-B03BF07E-DC29-4BC1-A32E-80BD77596BC1Q48161606-2B290762-922C-4542-AD30-F65074147402
P2860
Central inflammation and sickness-like behavior induced by the food contaminant deoxynivalenol: a PGE2-independent mechanism.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Central inflammation and sickn ...... a PGE2-independent mechanism.
@en
Central inflammation and sickn ...... a PGE2-independent mechanism.
@nl
type
label
Central inflammation and sickn ...... a PGE2-independent mechanism.
@en
Central inflammation and sickn ...... a PGE2-independent mechanism.
@nl
prefLabel
Central inflammation and sickn ...... a PGE2-independent mechanism.
@en
Central inflammation and sickn ...... a PGE2-independent mechanism.
@nl
P2093
P2860
P356
P1476
Central inflammation and sickn ...... a PGE2-independent mechanism.
@en
P2093
André Jean
Bruno Lebrun
Catherine Tardivel
Clémence Girardet
Jean-Denis Troadec
Julien Roux
Jérôme Trouslard
Lourdes Mounien
Marion S Bonnet
Medhi Sadoud
P2860
P304
P356
10.1093/TOXSCI/KFR219
P577
2011-08-26T00:00:00Z