Leptin receptor expression in hindbrain Glp-1 neurons regulates food intake and energy balance in mice. - Wikidata - awgldk - TriplyDB

Leptin receptor expression in hindbrain Glp-1 neurons regulates food intake and energy balance in mice.

Leptin receptor expression in hindbrain Glp-1 neurons regulates food intake and energy balance in mice.

http://www.wikidata.org/entity/Q42741829

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Secret talk between adipose tissue and central nervous system via secreted factors-an emerging frontier in the neurodegenerative research From neuroanatomy to behavior: central integration of peripheral signals regulating feeding behavior Brain regulation of energy balance and body weight.Central nervous control of energy and glucose balance: focus on the central melanocortin system Neural control of energy balance: translating circuits to therapies Physiology of proglucagon peptides: role of glucagon and GLP-1 in health and disease Glucagon-like peptide 1 interacts with ghrelin and leptin to regulate glucose metabolism and food intake through vagal afferent neuron signaling Leptin-inhibited PBN neurons enhance responses to hypoglycemia in negative energy balance PPARγ in vagal neurons regulates high-fat diet induced thermogenesis Dynamic expression of transcription factor Brn3b during mouse cranial nerve development Developmental specification of metabolic circuitry Neuronal GLP1R mediates liraglutide's anorectic but not glucose-lowering effect.Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.FOXO1 in the ventromedial hypothalamus regulates energy balance The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss.Targeted leptin receptor blockade: role of ventral tegmental area and nucleus of the solitary tract leptin receptors in body weight homeostasis.Long term exendin-4 treatment reduces food intake and body weight and alters expression of brain homeostatic and reward markers.Intestinal deletion of leptin signaling alters activity of nutrient transporters and delayed the onset of obesity in mice.Deletion of leptin signaling in vagal afferent neurons results in hyperphagia and obesity Role of oxytocin signaling in the regulation of body weight.Neuroanatomy of body weight control: lessons learned from leptin.Set points, settling points and some alternative models: theoretical options to understand how genes and environments combine to regulate body adiposity.Limited impact on glucose homeostasis of leptin receptor deletion from insulin- or proglucagon-expressing cells Endogenous leptin receptor signaling in the medial nucleus tractus solitarius affects meal size and potentiates intestinal satiation signals.Ablation of intact hypothalamic and/or hindbrain TrkB signaling leads to perturbations in energy balance.Food intake reductions and increases in energetic responses by hindbrain leptin and melanotan II are enhanced in mice with POMC-specific PTP1B deficiency.Neurochemical characterization of body weight-regulating leptin receptor neurons in the nucleus of the solitary tract.Central Leptin Regulation of Obesity and Fertility.Leptin action through hypothalamic nitric oxide synthase-1-expressing neurons controls energy balance Area postrema undergoes dynamic postnatal changes in mice and humans FGF21 Mediates Endocrine Control of Simple Sugar Intake and Sweet Taste Preference by the Liver Cannabinoid receptor 1 in the vagus nerve is dispensable for body weight homeostasis but required for normal gastrointestinal motility.Functional heterogeneity of arcuate nucleus pro-opiomelanocortin neurons: implications for diverging melanocortin pathways.Selective leptin resistance revisited.FGF21 regulates metabolism and circadian behavior by acting on the nervous system Neuronal circuits that regulate feeding behavior and metabolism.Leptin Controls Parasympathetic Wiring of the Pancreas during Embryonic Life.A critical view of the use of genetic tools to unveil neural circuits: the case of leptin action in reproduction Blockade of the cerebral aqueduct in rats provides evidence of antagonistic leptin responses in the forebrain and hindbrain.C1q/TNF-related protein 4 (CTRP4) is a unique secreted protein with two tandem C1q domains that functions in the hypothalamus to modulate food intake and body weight.

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P2860

Leptin receptor expression in hindbrain Glp-1 neurons regulates food intake and energy balance in mice.

http://www.wikidata.org/entity/Q42741829

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Joel K Elmquist

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Michael M Scott

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P2860

Positional cloning of the mouse obese gene and its human homologue

Selective deletion of leptin receptor in neurons leads to obesity

The homeobox gene Phox2b is essential for the development of autonomic neural crest derivatives

Evidence that the caudal brainstem is a target for the inhibitory effect of leptin on food intake

Expression and interactions of the two closely related homeobox genes Phox2a and Phox2b during neurogenesis

Leptin and the regulation of body weight in mammals

Weight-reducing effects of the plasma protein encoded by the obese gene

Congenital leptin deficiency is associated with severe early-onset obesity in humans

Identification and expression cloning of a leptin receptor, OB-R

Expression of Phox2b by brainstem neurons involved in chemosensory integration in the adult rat.

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