Requirement for RAR-mediated gene repression in skeletal progenitor differentiation. - Wikidata - awgldk - TriplyDB

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

http://www.wikidata.org/entity/Q42917201

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Transcriptional co-operativity between distant retinoic acid response elements in regulation of Cyp26A1 inducibility Increased dietary intake of vitamin A promotes aortic valve calcification in vivo Tumor necrosis factor alpha and epidermal growth factor act additively to inhibit matrix gene expression by chondrocyte.A pathway to bone: signaling molecules and transcription factors involved in chondrocyte development and maturation Fibroblast growth factor (FGF) 18 signals through FGF receptor 3 to promote chondrogenesis Incomplete penetrance and phenotypic variability characterize Gdf6-attributable oculo-skeletal phenotypes Dominant-negative retinoic acid receptors elicit epidermal defects through a non-canonical pathway Egr-1 inhibits the expression of extracellular matrix genes in chondrocytes by TNFalpha-induced MEK/ERK signalling Retinoic acid receptors are required for skeletal growth, matrix homeostasis and growth plate function in postnatal mouse Nondestructive Techniques to Evaluate the Characteristics and Development of Engineered Cartilage.Retinoic acid actions through mammalian nuclear receptors.The transcription factor ATF3 is upregulated during chondrocyte differentiation and represses cyclin D1 and A gene transcription Wnt/beta-catenin and retinoic acid receptor signaling pathways interact to regulate chondrocyte function and matrix turnover.Hyaluronan oligosaccharide-induced activation of transcription factors in bovine articular chondrocytes.Constitutive activation of MKK6 in chondrocytes of transgenic mice inhibits proliferation and delays endochondral bone formation Endogenous retinoids in mammalian growth plate cartilage: analysis and roles in matrix homeostasis and turnover Sox9-regulated miRNA-574-3p inhibits chondrogenic differentiation of mesenchymal stem cells Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists.Active repression by unliganded retinoid receptors in development: less is sometimes more Multifaceted signaling regulators of chondrogenesis: Implications in cartilage regeneration and tissue engineering Common mutations in ALK2/ACVR1, a multi-faceted receptor, have roles in distinct pediatric musculoskeletal and neural orphan disorders.Mutant Hoxd13 induces extra digits in a mouse model of synpolydactyly directly and by decreasing retinoic acid synthesis.Cells and biomaterials in cartilage tissue engineering.The roles of endogenous retinoid signaling in organ and appendage regeneration.Regulation of Sox9 activity by crosstalk with nuclear factor-kappaB and retinoic acid receptors.Regulation of retinoic acid synthetic enzymes by WT1 and HDAC inhibitors in 293 cells.Signaling pathways regulating cartilage growth plate formation and activity.Zebrafish retinoic acid receptors function as context-dependent transcriptional activators.Targeted stimulation of retinoic acid receptor-γ mitigates the formation of heterotopic ossification in an established blast-related traumatic injury model.Genetic and pharmacological inhibition of retinoic acid receptor γ function promotes endochondral bone formation.BMP action in skeletogenesis involves attenuation of retinoid signaling.Effectiveness and mode of action of a combination therapy for heterotopic ossification with a retinoid agonist and an anti-inflammatory agent.Mechanisms underlying the synergistic enhancement of self-assembled neocartilage treated with chondroitinase-ABC and TGF-β1.Low-density expansion protects human synovium-derived stem cells from replicative senescence: a preliminary study.Palovarotene Inhibits Heterotopic Ossification and Maintains Limb Mobility and Growth in Mice With the Human ACVR1(R206H) Fibrodysplasia Ossificans Progressiva (FOP) Mutation Sox9 Expression in Amniotes: Species-Specific Differences in the Formation of Digits.The retinal pigment epithelium of the eye regulates the development of scleral cartilage.p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.Rho/ROCK and MEK/ERK activation by transforming growth factor-alpha induces articular cartilage degradation.Astrocyte-derived retinoic acid: a novel regulator of blood-brain barrier function in multiple sclerosis.

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P2860

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

http://www.wikidata.org/entity/Q42917201

description

2002 nî lūn-bûn

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2002年論文

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2002年论文

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2002年论文

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2002年论文

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name

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@en

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@nl

type

label

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@en

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@nl

prefLabel

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@en

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

@nl

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Journal of Cell Biology

P1476

Requirement for RAR-mediated gene repression in skeletal progenitor differentiation.

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P2093

Andrea D Weston

http://www.w3.org/2001/XMLSchema#string

Joseph Torchia

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Roshantha A S Chandraratna

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T Michael Underhill

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P2860

Phosphorylation of SOX9 by cyclic AMP-dependent protein kinase A enhances SOX9's ability to transactivate a Col2a1 chondrocyte-specific enhancer

Signal-dependent nuclear export of a histone deacetylase regulates muscle differentiation

Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor

Characterization of the structure and function of a novel MAP kinase kinase (MKK6)

Structures of a histone deacetylase homologue bound to the TSA and SAHA inhibitors

Embryonic retinoic acid synthesis is essential for early mouse post-implantation development

Campomelic dysplasia and autosomal sex reversal caused by mutations in an SRY-related gene

Autosomal sex reversal and campomelic dysplasia are caused by mutations in and around the SRY-related gene SOX9

The retinoic acid-inactivating enzyme CYP26 is essential for establishing an uneven distribution of retinoic acid along the anterio-posterior axis within the mouse embryo

The retinoic acid-metabolizing enzyme, CYP26A1, is essential for normal hindbrain patterning, vertebral identity, and development of posterior structures.

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10.1083/JCB.200112029

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1

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158

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