Effects of type I interferons on Friend retrovirus infection.
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Insufficient natural killer cell responses against retroviruses: how to improve NK cell killing of retrovirus-infected cellsInterferon-alpha subtype 11 activates NK cells and enables control of retroviral infectionAnti-retroviral effects of type I IFN subtypes in vivoSARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.Type I interferon signaling is required for the APOBEC3/Rfv3-dependent neutralizing antibody response but not innate retrovirus restriction.Blockade of type I interferon (IFN) production by retroviral replicating vectors and reduced tumor cell responses to IFN likely contribute to tumor selectivityUnraveling the convoluted biological roles of type I interferons in infection and immunity: a way forward for therapeutics and vaccine design.Tetherin promotes the innate and adaptive cell-mediated immune response against retrovirus infection in vivo.A single nucleotide polymorphism in tetherin promotes retrovirus restriction in vivo.Friend retrovirus drives cytotoxic effectors through Toll-like receptor 3PD-L1 Expression on Retrovirus-Infected Cells Mediates Immune Escape from CD8+ T Cell KillingInnate immune sensing of retroviral infection via Toll-like receptor 7 occurs upon viral entry.hsp70 and a novel axis of type I interferon-dependent antiviral immunity in the measles virus-infected brain.Toll-like receptor 7 inhibits early acute retroviral infection through rapid lymphocyte responsesB lymphocyte activation by coinfection prevents immune control of friend virus infection.Differential requirements of cellular and humoral immune responses for Fv2-associated resistance to erythroleukemia and for regulation of retrovirus-induced myeloid leukemia development.Confounding roles for type I interferons during bacterial and viral pathogenesis.Ribonuclease L is not critical for innate restriction and adaptive immunity against Friend retrovirus infectionNatural killer T cells contribute to the control of acute retroviral infection.Humoral immunity in the Friend retrovirus infection model.Effect of type-I interferon on retroviruses.Dose of Retroviral Infection Determines Induction of Antiviral NK Cell Responses.IFN-α treatment inhibits acute Friend retrovirus replication primarily through the antiviral effector molecule Apobec3.Upon intranasal vesicular stomatitis virus infection, astrocytes in the olfactory bulb are important interferon Beta producers that protect from lethal encephalitisLentiviral Protein Transfer Vectors Are an Efficient Vaccine Platform and Induce a Strong Antigen-Specific Cytotoxic T Cell Response.NK cells improve control of friend virus infection in mice persistently infected with murine cytomegalovirus.Beta interferon controls West Nile virus infection and pathogenesis in mice.Protective role of beta interferon in host defense against influenza A virus.Induction of Type I Interferons by Therapeutic Nanoparticle-Based Vaccination Is Indispensable to Reinforce Cytotoxic CD8+ T Cell Responses During Chronic Retroviral Infection.
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Effects of type I interferons on Friend retrovirus infection.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Effects of type I interferons on Friend retrovirus infection.
@en
Effects of type I interferons on Friend retrovirus infection.
@nl
type
label
Effects of type I interferons on Friend retrovirus infection.
@en
Effects of type I interferons on Friend retrovirus infection.
@nl
prefLabel
Effects of type I interferons on Friend retrovirus infection.
@en
Effects of type I interferons on Friend retrovirus infection.
@nl
P2093
P2860
P1433
P1476
Effects of type I interferons on Friend retrovirus infection.
@en
P2093
Nicole Gerlach
Siegfried Weiss
Simone Schimmer
P2860
P304
P356
10.1128/JVI.80.7.3438-3444.2006
P407
P577
2006-04-01T00:00:00Z