Different host defences are required to protect mice from primary systemic vs pulmonary infection with the facultative intracellular bacterial pathogen, Francisella tularensis LVS.
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Immunotherapy for tularemiaEvasion of IFN-γ signaling by Francisella novicida is dependent upon Francisella outer membrane protein CIntranasal immunization with an archaeal lipid mucosal vaccine adjuvant and delivery formulation protects against a respiratory pathogen challengeTLR-dependent control of Francisella tularensis infection and host inflammatory responsesCurrent status of vaccine development for tularemia preparedness.Identification of genes contributing to the virulence of Francisella tularensis SCHU S4 in a mouse intradermal infection modelDifferential ability of novel attenuated targeted deletion mutants of Francisella tularensis subspecies tularensis strain SCHU S4 to protect mice against aerosol challenge with virulent bacteria: effects of host background and route of immunization.Intranasal interleukin-12 treatment for protection against respiratory infection with the Francisella tularensis live vaccine strain.Genome-wide RNAi screen in IFN-γ-treated human macrophages identifies genes mediating resistance to the intracellular pathogen Francisella tularensis.Generation of a convalescent model of virulent Francisella tularensis infection for assessment of host requirements for survival of tularemiaHost-pathogen interactions and immune evasion strategies in Francisella tularensis pathogenicityMouse models of aerosol-acquired tularemia caused by Francisella tularensis types A and B.Administration of a synthetic TLR4 agonist protects mice from pneumonic tularemia.Francisella tularensis subsp. tularensis induces a unique pulmonary inflammatory response: role of bacterial gene expression in temporal regulation of host defense responsesThe francisella tularensis proteome and its recognition by antibodies.Mac-1+ cells are the predominant subset in the early hepatic lesions of mice infected with Francisella tularensis.Coactivating signals for the hepatic lymphocyte gamma interferon response to Francisella tularensisVaccines against Francisella tularensis--past, present and future.Prior infection with Type A Francisella tularensis antagonizes the pulmonary transcriptional response to an aerosolized Toll-like receptor 4 agonist.Oral immunization of mice with the live vaccine strain (LVS) of Francisella tularensis protects mice against respiratory challenge with virulent type A F. tularensis.Francisella tularensis: taxonomy, genetics, and Immunopathogenesis of a potential agent of biowarfare.An Immature Myeloid/Myeloid-Suppressor Cell Response Associated with Necrotizing Inflammation Mediates Lethal Pulmonary Tularemia.Alpha-1 antitrypsin is markedly decreased following pulmonary F. tularensis challengeInflammasome-Independent NLRP3 Restriction of a Protective Early Neutrophil Response to Pulmonary Tularemia.Toll-like receptor 2 controls the gamma interferon response to Francisella tularensis by mouse liver lymphocytes.Neutrophils play an important role in host resistance to respiratory infection with Acinetobacter baumannii in mice.Interleukin-6 is essential for primary resistance to Francisella tularensis live vaccine strain infection.Phenotypic Characterization of a Novel Virulence-Factor Deletion Strain of Burkholderia mallei That Provides Partial Protection against Inhalational Glanders in MiceB1a cells enhance susceptibility to infection with virulent Francisella tularensis via modulation of NK/NKT cell responses.Respiratory Francisella tularensis live vaccine strain infection induces Th17 cells and prostaglandin E2, which inhibits generation of gamma interferon-positive T cells.Initial delay in the immune response to Francisella tularensis is followed by hypercytokinemia characteristic of severe sepsis and correlating with upregulation and release of damage-associated molecular patterns.Mast cells inhibit intramacrophage Francisella tularensis replication via contact and secreted products including IL-4.F. novicida-Infected A. castellanii Does Not Enhance Bacterial Virulence in Mice.Neutrophils are essential for rapid clearance of Enterococcus faecium in miceFrancisella inhibits STAT1-mediated signaling in macrophages and prevents activation of antigen-specific T cells.Interleukin-17 protects against the Francisella tularensis live vaccine strain but not against a virulent F. tularensis type A strain.T cells from lungs and livers of Francisella tularensis-immune mice control the growth of intracellular bacteriaFrancisella tularensis directly interacts with the endothelium and recruits neutrophils with a blunted inflammatory phenotype.Humoral and cell-mediated immunity to the intracellular pathogen Francisella tularensis.Francisella tularensis induces extensive caspase-3 activation and apoptotic cell death in the tissues of infected mice.
P2860
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P2860
Different host defences are required to protect mice from primary systemic vs pulmonary infection with the facultative intracellular bacterial pathogen, Francisella tularensis LVS.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Different host defences are re ...... n, Francisella tularensis LVS.
@en
Different host defences are re ...... n, Francisella tularensis LVS.
@nl
type
label
Different host defences are re ...... n, Francisella tularensis LVS.
@en
Different host defences are re ...... n, Francisella tularensis LVS.
@nl
prefLabel
Different host defences are re ...... n, Francisella tularensis LVS.
@en
Different host defences are re ...... n, Francisella tularensis LVS.
@nl
P2093
P356
P1476
Different host defences are re ...... n, Francisella tularensis LVS.
@en
P2093
J Wayne Conlan
Rhonda KuoLee
P304
P356
10.1006/MPAT.2001.0489
P577
2002-03-01T00:00:00Z