Reproductive malformation of the male offspring following maternal exposure to estrogenic chemicals.
about
Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutationsThe toxic origins of diseaseIn utero exposure to bisphenol-A and anogenital distance of male offspringBasic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case studyEstrogenic chemicals in plastic and oral contraceptives disrupt development of the fetal mouse prostate and urethra.Exposure to a low dose of bisphenol A during fetal life or in adulthood alters maternal behavior in miceBiotransformations of bisphenol A in a mammalian model: answers and new questions raised by low-dose metabolic fate studies in pregnant CD1 miceAn extensive new literature concerning low-dose effects of bisphenol A shows the need for a new risk assessmentEstrogen and xenoestrogens in breast cancerHormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responsesThe politics of plastics: the making and unmaking of bisphenol a "safety"Endocrine-disrupting chemicals: an Endocrine Society scientific statementWhy public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol ADirect evidence revealing structural elements essential for the high binding ability of bisphenol A to human estrogen-related receptor-gammaEstradiol and Bisphenol A stimulate androgen receptor and estrogen receptor gene expression in fetal mouse prostate mesenchyme cellsIn vivo effects of bisphenol A in laboratory rodent studiesIn vitro and in vivo estrogenicity of UV screensEarly cerebral activities of the environmental estrogen bisphenol A appear to act via the somatostatin receptor subtype sst(2)Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levelsSurvivorship patterns of larval amphibians exposed to low concentrations of atrazineLarge effects from small exposures. I. Mechanisms for endocrine-disrupting chemicals with estrogenic activityEffect of bisphenol A on murine immune function: modulation of interferon-gamma, IgG2a, and disease symptoms in NZB X NZW F1 miceNon-monotonic dose-response relationships and endocrine disruptors: a qualitative method of assessmentDirected Differentiation of Human Embryonic Stem Cells into Prostate Organoids In Vitro and its Perturbation by Low-Dose Bisphenol A ExposureToluene at environmentally relevant low levels disrupts differentiation of astrocyte precursor cellsAdverse effects in adulthood resulting from low-level dioxin exposure in juvenile zebrafishProceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: executive summaryEvidence that bisphenol A (BPA) can be accurately measured without contamination in human serum and urine, and that BPA causes numerous hazards from multiple routes of exposureDisruption of androgen receptor signaling in males by environmental chemicalsPCB-153 shows different dynamics of mobilisation from differentiated rat adipocytes during lipolysis in comparison with PCB-28 and PCB-118Bisphenol A: Perinatal exposure and body weightOf Oestrogens and Sperm: A Review of the Roles of Oestrogens and Oestrogen Receptors in Male Reproduction.Components of plastic: experimental studies in animals and relevance for human health.Bisphenol A interferes with synaptic remodeling.Bisphenol AF is a full agonist for the estrogen receptor ERalpha but a highly specific antagonist for ERbetaOestrogenic and antiandrogenic chemicals in the environment: effects on male reproductive health.Contributions of androgen and estrogen to fetal programming of ovarian dysfunction.Childhood obesity and environmental chemicals.Endocrine disruptor issues in Japan.Expressional Changes of Water Transport-related Molecules in the Efferent Ductules and Initial Segment of Mouse Treated with Bisphenol A-Containing Drinking Water for Two Generations
P2860
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P2860
Reproductive malformation of the male offspring following maternal exposure to estrogenic chemicals.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh-hant
name
Reproductive malformation of t ...... osure to estrogenic chemicals.
@en
Reproductive malformation of t ...... osure to estrogenic chemicals.
@nl
type
label
Reproductive malformation of t ...... osure to estrogenic chemicals.
@en
Reproductive malformation of t ...... osure to estrogenic chemicals.
@nl
prefLabel
Reproductive malformation of t ...... osure to estrogenic chemicals.
@en
Reproductive malformation of t ...... osure to estrogenic chemicals.
@nl
P2860
P1476
Reproductive malformation of t ...... osure to estrogenic chemicals.
@en
P2093
P2860
P356
10.1046/J.1525-1373.2000.22402.X
P577
2000-06-01T00:00:00Z