Type I IFN signaling is crucial for host resistance against different species of pathogenic bacteria. - Wikidata - awgldk - TriplyDB

Type I IFN signaling is crucial for host resistance against different species of pathogenic bacteria.

Type I IFN signaling is crucial for host resistance against different species of pathogenic bacteria.

http://www.wikidata.org/entity/Q43596491

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Type I interferon protects against pneumococcal invasive disease by inhibiting bacterial transmigration across the lung Inflammasome/IL-1β Responses to Streptococcal Pathogens Immunomodulatory functions of type I interferons Critical role of type I interferon-induced macrophage necroptosis during infection with Salmonella enterica serovar Typhimurium Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia T(H)17 cells promote microbial killing and innate immune sensing of DNA via interleukin 26 Type I interferon: friend or foe?Interferon protects mice against inhalation anthrax Type I interferon induces necroptosis in macrophages during infection with Salmonella enterica serovar Typhimurium Age-enhanced endoplasmic reticulum stress contributes to increased Atg9A inhibition of STING-mediated IFN-β production during Streptococcus pneumoniae infection Salmonella Suppresses the TRIF-Dependent Type I Interferon Response in Macrophages.Identification of host cytosolic sensors and bacterial factors regulating the type I interferon response to Legionella pneumophila Forced IFIT-2 expression represses LPS induced TNF-alpha expression at posttranscriptional levels.Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes Streptococcus pneumoniae nasopharyngeal colonization induces type I interferons and interferon-induced gene expression.The bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferons Type I Interferon Induction by Neisseria gonorrhoeae: Dual Requirement of Cyclic GMP-AMP Synthase and Toll-like Receptor 4.Borrelia burgdorferi RNA induces type I and III interferons via Toll-like receptor 7 and contributes to production of NF-κB-dependent cytokines Bifidobacterium bifidum actively changes the gene expression profile induced by Lactobacillus acidophilus in murine dendritic cells.Induction and termination of inflammatory signaling in group B streptococcal sepsis.Type I interferon signaling in hematopoietic cells is required for survival in mouse polymicrobial sepsis by regulating CXCL10.A fluorescence reporter model defines "Tip-DCs" as the cellular source of interferon β in murine listeriosis Induction of type I and type III interferons by Borrelia burgdorferi correlates with pathogenesis and requires linear plasmid 36.Mucosal immunotherapy for protection from pneumonic infection with Francisella tularensis Mast cells elicit proinflammatory but not type I interferon responses upon activation of TLRs by bacteria Bacteria-specific neutrophil dysfunction associated with interferon-stimulated gene expression in the acute respiratory distress syndrome.MyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infection Type I interferons mediate the innate cytokine response to recombinant fowlpox virus but not the induction of plasmacytoid dendritic cell-dependent adaptive immunity.XBP-1 couples endoplasmic reticulum stress to augmented IFN-beta induction via a cis-acting enhancer in macrophages.Type I IFN receptor regulates neutrophil functions and innate immunity to Leishmania parasites.Type I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.An essential role for IFN-β in the induction of IFN-stimulated gene expression by LPS in macrophages.Essential role of interleukin-1 signaling in host defenses against group B streptococcus.Murine gut microbiota is defined by host genetics and modulates variation of metabolic traits Avian resistance to Campylobacter jejuni colonization is associated with an intestinal immunogene expression signature identified by mRNA sequencing.Role of Toll-like receptor 13 in innate immune recognition of group B streptococci.The interleukin-1β/CXCL1/2/neutrophil axis mediates host protection against group B streptococcal infection.Negative regulation of type I IFN expression by OASL1 permits chronic viral infection and CD8⁺ T-cell exhaustion.Streptococcus pneumoniae DNA initiates type I interferon signaling in the respiratory tract MPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.

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Type I IFN signaling is crucial for host resistance against different species of pathogenic bacteria.

http://www.wikidata.org/entity/Q43596491

description

2007 nî lūn-bûn

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2007年の論文

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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JIMMUNOL.178.5.3126

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Journal of Immunology

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Type I IFN signaling is crucia ...... pecies of pathogenic bacteria.

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Alessia Ruggeri

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Angelina Midiri

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Carmelo Biondo

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Concetta Beninati

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Francesco Tomasello

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Giancarlo Macrì

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Giuseppe Mancuso

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Giuseppe Teti

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Maria Gambuzza

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Roberta Galbo

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3126-3133

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scholarly article

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10.4049/JIMMUNOL.178.5.3126

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2007-03-01T00:00:00Z

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