Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
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H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failureOsteopontin: role in extracellular matrix deposition and myocardial remodeling post-MI.Matrix metalloproteinases cleave the beta2-adrenergic receptor in spontaneously hypertensive rats.Homocysteine to hydrogen sulfide or hypertension.Lack of ataxia telangiectasia mutated kinase induces structural and functional changes in the heart: role in β-adrenergic receptor-stimulated apoptosisMetalloproteinase in myocardial adaptation and maladaptation.Synergism between arrhythmia and hyperhomo-cysteinemia in structural heart disease.β-Adrenergic Receptor-Stimulated Cardiac Myocyte Apoptosis: Role of β1 Integrins.Mechanotransduction Mechanisms for Intraventricular Diastolic Vortex Forces and Myocardial Deformations: Part 2.ECM remodeling in hypertensive heart disease.Superoxide-dependent cathepsin activation is associated with hypertensive myocardial remodeling and represents a target for angiotensin II type 1 receptor blocker treatment.Imperatorin derivative OW1 inhibits the upregulation of TGF-β and MMP-2 in renovascular hypertension-induced cardiac remodeling.Nitrotyrosinylation, remodeling and endothelial-myocyte uncoupling in iNOS, cystathionine beta synthase (CBS) knockouts and iNOS/CBS double knockout mice.Inhibition of matrix metalloproteinases improves left ventricular function in mice lacking osteopontin after myocardial infarction.An emerging role of degrading proteinases in hypertension and the metabolic syndrome: autodigestion and receptor cleavage.THE AUTODIGESTION HYPOTHESIS AND RECEPTOR CLEAVAGE IN DIABETES AND HYPERTENSION.Management of cardiac fibrosis in diabetic rats; the role of peroxisome proliferator activated receptor gamma (PPAR-gamma) and calcium channel blockers (CCBs)Activation of GABA-A receptor ameliorates homocysteine-induced MMP-9 activation by ERK pathway.Cardiosome mediated regulation of MMP9 in diabetic heart: role of mir29b and mir455 in exercise.Mitochondrial pathways to cardiac recovery: TFAM.Tumor necrosis factor-related weak inducer of apoptosis augments matrix metalloproteinase 9 (MMP-9) production in skeletal muscle through the activation of nuclear factor-kappaB-inducing kinase and p38 mitogen-activated protein kinase: a potential rProteinase activity and receptor cleavage: mechanism for insulin resistance in the spontaneously hypertensive rat.Apoptosis in the left ventricle of chronic volume overload causes endocardial endothelial dysfunction in rats.Two MMP-2 promoter polymorphisms (-790T/G and -735C/T) in chronic heart failure.Protease-activated receptor and endothelial-myocyte uncoupling in chronic heart failure.Pioglitazone prevents cardiac remodeling in high-fat, high-calorie-induced Type 2 diabetes mellitus.Soluble CD74 Reroutes MIF/CXCR4/AKT-Mediated Survival of Cardiac Myofibroblasts to Necroptosis
P2860
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P2860
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@en
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@nl
type
label
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@en
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@nl
prefLabel
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@en
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@nl
P2093
P1476
Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength.
@en
P2093
P304
P356
10.1016/S0167-5273(01)00449-1
P407
P577
2001-07-01T00:00:00Z