Cellular energetics in the preconditioned state: protective role for phosphotransfer reactions captured by 18O-assisted 31P NMR.
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Defective metabolic signaling in adenylate kinase AK1 gene knock-out hearts compromises post-ischemic coronary reflowAdenylate kinase and AMP signaling networks: metabolic monitoring, signal communication and body energy sensingMolecular system bioenergics of the heart: experimental studies of metabolic compartmentation and energy fluxes versus computer modelingDefects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's diseaseEnergetic communication between mitochondria and nucleus directed by catalyzed phosphotransfer.Modular organization of cardiac energy metabolism: energy conversion, transfer and feedback regulation.Sensitivity analysis of flux determination in heart by H₂ ¹⁸O -provided labeling using a dynamic Isotopologue model of energy transfer pathways.The role of mitochondria in protection of the heart by preconditioning.Proteomic changes in the heart of diet-induced pre-diabetic mice.Cellular remodeling in heart failure disrupts K(ATP) channel-dependent stress tolerance(31)P NMR correlation maps of (18)O/ (16)O chemical shift isotopic effects for phosphometabolite labeling studies.Dynamic phosphometabolomic profiling of human tissues and transgenic models by 18O-assisted ³¹P NMR and mass spectrometryDecline of Phosphotransfer and Substrate Supply Metabolic Circuits Hinders ATP Cycling in Aging Myocardium.Cardiac KATP channels in health and disease.Cardiac system bioenergetics: metabolic basis of the Frank-Starling law.18O-assisted dynamic metabolomics for individualized diagnostics and treatment of human diseases.Prostate cancer: correlation of MR imaging and MR spectroscopy with pathologic findings after radiation therapy-initial experience.Philosophical basis and some historical aspects of systems biology: from Hegel to Noble - applications for bioenergetic research.Mapping hypoxia-induced bioenergetic rearrangements and metabolic signaling by 18O-assisted 31P NMR and 1H NMR spectroscopyNucleotide-gated KATP channels integrated with creatine and adenylate kinases: amplification, tuning and sensing of energetic signals in the compartmentalized cellular environment.Bidirectionality and compartmentation of metabolic fluxes are revealed in the dynamics of isotopomer networks.Compartmentation of membrane processes and nucleotide dynamics in diffusion-restricted cardiac cell microenvironment.Cellular compartmentation of energy metabolism: creatine kinase microcompartments and recruitment of B-type creatine kinase to specific subcellular sites.Rearrangement of energetic and substrate utilization networks compensate for chronic myocardial creatine kinase deficiency.Ischemic preconditioning vs adenosine vs prostaglandin E1 for protection against liver ischemia/reperfusion injuryElectron spray ionization mass spectrometry and 2D 31P NMR for monitoring 18O/16O isotope exchange and turnover rates of metabolic oligophosphates.Where have the fluxes gone?The combination of ischemic preconditioning and liver Bcl-2 overexpression is a suitable strategy to prevent liver and lung damage after hepatic ischemia-reperfusionK(ATP) channels process nucleotide signals in muscle thermogenic response.Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.Tandem function of nucleotide binding domains confers competence to sulfonylurea receptor in gating ATP-sensitive K+ channels.Coupling of cell energetics with membrane metabolic sensing. Integrative signaling through creatine kinase phosphotransfer disrupted by M-CK gene knock-out.Adenylate kinase AK1 knockout heart: energetics and functional performance under ischemia-reperfusion.ATP-sensitive K(+) channel activation by nitric oxide and protein kinase G in rabbit ventricular myocytes.Failing atrial myocardium: energetic deficits accompany structural remodeling and electrical instability.Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotection.Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart.Impaired intracellular energetic communication in muscles from creatine kinase and adenylate kinase (M-CK/AK1) double knock-out mice.Regular exercise is associated with a protective metabolic phenotype in the rat heart.Potassium channel openers are uncoupling protonophores: implication in cardioprotection.
P2860
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P2860
Cellular energetics in the preconditioned state: protective role for phosphotransfer reactions captured by 18O-assisted 31P NMR.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@en
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@nl
type
label
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@en
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@nl
prefLabel
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@en
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@nl
P2093
P2860
P356
P1476
Cellular energetics in the pre ...... tured by 18O-assisted 31P NMR.
@en
P2093
P2860
P304
44812-44819
P356
10.1074/JBC.M104425200
P407
P577
2001-10-02T00:00:00Z