Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment.
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Restoration of tissue factor pathway inhibitor-2 in a human glioblastoma cell line triggers caspase-mediated pathway and apoptosisRole of AIF in human coronary artery endothelial cell apoptosisp57(Kip2) cooperates with Nurr1 in developing dopamine cellsApoptosis-inducing factor and calpain upregulation in glutamate-induced injury of rat spiral ganglion neuronsDifferential sensitivity to apoptosome apparatus activation in non-small cell lung carcinoma and the lungCross-talk between NFkB and the PI3-kinase/AKT pathway can be targeted in primary effusion lymphoma (PEL) cell lines for efficient apoptosis.Delayed activation of caspase-independent apoptosis during heart failure in transgenic mice overexpressing caspase inhibitor CrmAAmeloblastic carcinoma of the mandible: Report of a case and review.Detection of mitochondria-mediated apoptosis signaling molecules in ameloblastomas.T-cell ubiquitin ligand affects cell death through a functional interaction with apoptosis-inducing factor, a key factor of caspase-independent apoptosis.Genes associated with sodium fluoride-induced human osteoblast apoptosisTargeting GSK-3: a promising approach for cancer therapy?Inhibition of Ephrin B3-mediated survival signaling contributes to increased cell death response of non-small cell lung carcinoma cells after combined treatment with ionizing radiation and PKC 412.Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathways.Proteomics and pathway analysis identifies JNK signaling as critical for high linear energy transfer radiation-induced apoptosis in non-small lung cancer cellsRole of glutathione in cancer progression and chemoresistance.The Yin and Yang of redox regulation.Apoptosis induction in renal cell carcinoma by TRAIL and gamma-radiation is impaired by deficient caspase-9 cleavage.Visfatin induces the apoptosis of endothelial progenitor cells via the induction of pro-inflammatory mediators through the NF-κB pathway.Detecting the effect of targeted anti-cancer medicines on single cancer cells using a poly-silicon wire ion sensor integrated with a confined sensitive window.Capsaicin may induce breast cancer cell death through apoptosis-inducing factor involving mitochondrial dysfunction.Critical role for hyperpolarization-activated cyclic nucleotide-gated channel 2 in the AIF-mediated apoptosis.Thioredoxin reductase 1 knockdown enhances selenazolidine cytotoxicity in human lung cancer cells via mitochondrial dysfunctionOpposing effects of hMOF and SIRT1 on H4K16 acetylation and the sensitivity to the topoisomerase II inhibitor etoposide.Proinsulin C-peptide regulates ribosomal RNA expressionThe heat shock protein antagonist 17-AAG potentiates the activity of enzastaurin against malignant human glioma cells.Apoptosis of non-small-cell lung cancer cell lines after paclitaxel treatment involves the BH3-only proapoptotic protein Bim.Comprehensive review on betulin as a potent anticancer agent.Staurosporine induces endothelial cell apoptosis via focal adhesion kinase dephosphorylation and focal adhesion disassembly independent of focal adhesion kinase proteolysis.Dasatinib synergizes with JSI-124 to inhibit growth and migration and induce apoptosis of malignant human glioma cells.Tumor cytotoxicity by endothelial cells. Impairment of the mitochondrial system for glutathione uptake in mouse B16 melanoma cells that survive after in vitro interaction with the hepatic sinusoidal endothelium.Trauma-hemorrhagic shock mesenteric lymph induces endothelial apoptosis that involves both caspase-dependent and caspase-independent mechanisms.Combinatorial action of the HDAC inhibitor trichostatin A and etoposide induces caspase-mediated AIF-dependent apoptotic cell death in non-small cell lung carcinoma cells.The cell cycle inhibitor p57(Kip2) promotes cell death via the mitochondrial apoptotic pathway.
P2860
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P2860
Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Mitochondrial dysfunction is a ...... ant to conventional treatment.
@en
type
label
Mitochondrial dysfunction is a ...... ant to conventional treatment.
@en
prefLabel
Mitochondrial dysfunction is a ...... ant to conventional treatment.
@en
P2860
P50
P356
P1433
P1476
Mitochondrial dysfunction is a ...... ant to conventional treatment.
@en
P2093
Bertrand Joseph
Rolf Lewensohn
P2860
P2888
P356
10.1038/SJ.ONC.1205018
P407
P577
2002-01-01T00:00:00Z
P5875
P6179
1008956560