Redundant pathways for negative feedback regulation of bile acid production. - Wikidata - awgldk - TriplyDB

Redundant pathways for negative feedback regulation of bile acid production.

Redundant pathways for negative feedback regulation of bile acid production.

http://www.wikidata.org/entity/Q44025689

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Synergistic activation of the human orphan nuclear receptor SHP gene promoter by basic helix-loop-helix protein E2A and orphan nuclear receptor SF-1 Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis Tissue-specific expression of betaKlotho and fibroblast growth factor (FGF) receptor isoforms determines metabolic activity of FGF19 and FGF21 An autoregulatory feedback loop between Mdm2 and SHP that fine tunes Mdm2 and SHP stability Bile acids activate fibroblast growth factor 19 signaling in human hepatocytes to inhibit cholesterol 7alpha-hydroxylase gene expression Involvement of corepressor complex subunit GPS2 in transcriptional pathways governing human bile acid biosynthesis Promotion of liver regeneration/repair by farnesoid X receptor in both liver and intestine in mice Endocrine fibroblast growth factors 15/19 and 21: from feast to famine Bile acid transporters in health and disease Bile acids: regulation of apoptosis by ursodeoxycholic acid Bile acids: regulation of synthesis Endotoxin leads to rapid subcellular re-localization of hepatic RXRalpha: A novel mechanism for reduced hepatic gene expression in inflammation Recent advances in understanding and managing cholestasis Bile acid receptors and nonalcoholic fatty liver disease Controversies in the Mechanism of Total Parenteral Nutrition Induced Pathology Bile acid metabolism and signaling The bile acid sensor FXR protects against dyslipidemia and aortic plaques development induced by the HIV protease inhibitor ritonavir in mice Identification of a potent synthetic FXR agonist with an unexpected mode of binding and activation Structural insights into gene repression by the orphan nuclear receptor SHP Cytochrome P450s in the synthesis of cholesterol and bile acids--from mouse models to human diseases.Nuclear Receptors as Therapeutic Targets in Liver Disease: Are We There Yet?Regulation of fibroblast growth factor 15/19 and 21 on metabolism: in the fed or fasted state The farnesoid X receptor controls gene expression in a ligand- and promoter-selective fashion Glucocorticoid signaling is perturbed by the atypical orphan receptor and corepressor SHP Bile acid signaling in metabolic disease and drug therapy Conjugated bile acid-activated S1P receptor 2 is a key regulator of sphingosine kinase 2 and hepatic gene expression MAFG is a transcriptional repressor of bile acid synthesis and metabolism Ileal bile acid-binding protein, functionally associated with the farnesoid X receptor or the ileal bile acid transporter, regulates bile acid activity in the small intestine Small heterodimer partner-targeting therapy inhibits systemic inflammatory responses through mitochondrial uncoupling protein 2 The fetoprotein transcription factor (FTF) gene is essential to embryogenesis and cholesterol homeostasis and is regulated by a DR4 element The orphan nuclear receptor SHP acts as a negative regulator in inflammatory signaling triggered by Toll-like receptors Farnesoid X receptor is essential for normal glucose homeostasis Rifampicin Does not Significantly Affect the Expression of Small Heterodimer Partner in Primary Human Hepatocytes Role of nuclear receptor SHP in metabolism and cancer.Combined deletion of Fxr and Shp in mice induces Cyp17a1 and results in juvenile onset cholestasis.The small heterodimer partner is a gonadal gatekeeper of sexual maturation in male mice.Structural and biochemical basis for selective repression of the orphan nuclear receptor liver receptor homolog 1 by small heterodimer partner.Exposure to the synthetic FXR agonist GW4064 causes alterations in gene expression and sublethal hepatotoxicity in eleutheroembryo medaka (Oryzias latipes)Nuclear receptor SHP activates miR-206 expression via a cascade dual inhibitory mechanism.Mechanisms for increased expression of cholesterol 7alpha-hydroxylase (Cyp7a1) in lactating rats

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Redundant pathways for negative feedback regulation of bile acid production.

http://www.wikidata.org/entity/Q44025689

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2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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2002年學術文章

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Redundant pathways for negative feedback regulation of bile acid production.

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Redundant pathways for negative feedback regulation of bile acid production.

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Redundant pathways for negative feedback regulation of bile acid production.

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Redundant pathways for negative feedback regulation of bile acid production.

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Redundant pathways for negative feedback regulation of bile acid production.

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Redundant pathways for negative feedback regulation of bile acid production.

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Developmental Cell

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Redundant pathways for negative feedback regulation of bile acid production.

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Chang Soo Kim

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David D Moore

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Donnie Bundman

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Li Wang

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Ping Wei

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Richard A Heyman

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Steven S Chua

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Sundararajah Thevananther

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Yoon-Kwang Lee

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Yunqing Han

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10.1016/S1534-5807(02)00187-9

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