Defective humoral responses and extensive intravascular apoptosis are associated with fatal outcome in Ebola virus-infected patients. - Wikidata - awgldk - TriplyDB

Defective humoral responses and extensive intravascular apoptosis are associated with fatal outcome in Ebola virus-infected patients.

Defective humoral responses and extensive intravascular apoptosis are associated with fatal outcome in Ebola virus-infected patients.

http://www.wikidata.org/entity/Q44089836

about

High prevalence of both humoral and cellular immunity to Zaire ebolavirus among rural populations in Gabon Animal models for Ebola and Marburg virus infections Assembly of Ebola virus matrix protein VP40 is regulated by latch-like properties of N and C terminal tails Human fatal zaire ebola virus infection is associated with an aberrant innate immunity and with massive lymphocyte apoptosis Current ebola vaccines Clinical management of filovirus-infected patients Intracellular events and cell fate in filovirus infection Filovirus research in Gabon and equatorial Africa: the experience of a research center in the heart of Africa Quantifying Poverty as a Driver of Ebola Transmission Marburgvirus genomics and association with a large hemorrhagic fever outbreak in Angola Emerging targets and novel approaches to Ebola virus prophylaxis and treatment Ebola haemorrhagic fever Ebolavirus vaccines for humans and apes Antibody-dependent enhancement of Ebola virus infection Clinical development of Ebola vaccines The Role of Cytokines and Chemokines in Filovirus Infection Filoviruses: One of These Things is (not) Like the Other Ebola Virus Disease (The Killer Virus): Another Threat to Humans and Bioterrorism: Brief Review and Recent Updates Mouse models for filovirus infections Neutralizing ebolavirus: structural insights into the envelope glycoprotein and antibodies targeted against it Ebolavirus VP35 uses a bimodal strategy to bind dsRNA for innate immune suppression Structure of the Ebola virus glycoprotein bound to an antibody from a human survivor Structure of the Ebola VP35 interferon inhibitory domain Structural basis for dsRNA recognition and interferon antagonism by Ebola VP35 Marburg Virus VP35 Can Both Fully Coat the Backbone and Cap the Ends of dsRNA for Interferon Antagonism The Ebola Virus Interferon Antagonist VP24 Directly Binds STAT1 and Has a Novel, Pyramidal Fold Ebolavirus VP35 Coats the Backbone of Double-Stranded RNA for Interferon Antagonism [Haemostasis dysregulation in filovirus infections].Chloroquine could be used for the treatment of filoviral infections and other viral infections that emerge or emerged from viruses requiring an acidic pH for infectivity Early immune responses accompanying human asymptomatic Ebola infections Gender-dependent specific immune response during chronic human Schistosomiasis haematobia Neglected filoviruses Diagnosis of Ebola Virus Disease: Past, Present, and Future Deaths, late deaths, and role of infecting dose in Ebola virus disease in Sierra Leone: retrospective cohort study Interferon-γ Inhibits Ebola Virus Infection Broad-Range Antiviral Activity of Hydrogen Sulfide Against Highly Pathogenic RNA Viruses Production of Potent Fully Human Polyclonal Antibodies against Ebola Zaire Virus in Transchromosomal Cattle Immune protection of nonhuman primates against Ebola virus with single low-dose adenovirus vectors encoding modified GPs A review of Phase I trials of Ebola virus vaccines: what can we learn from the race to develop novel vaccines?Progress towards the treatment of Ebola haemorrhagic fever.

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Defective humoral responses and extensive intravascular apoptosis are associated with fatal outcome in Ebola virus-infected patients.

http://www.wikidata.org/entity/Q44089836

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