Mechanisms underlying the frequency dependence of contraction and [Ca(2+)](i) transients in mouse ventricular myocytes.
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Tumor treating fields perturb the localization of septins and cause aberrant mitotic exitInactivation of Smad5 in endothelial cells and smooth muscle cells demonstrates that Smad5 is required for cardiac homeostasisDisplacement-encoded and manganese-enhanced cardiac MRI reveal that nNOS, not eNOS, plays a dominant role in modulating contraction and calcium influx in the mammalian heart.Role of CaMKIIdelta phosphorylation of the cardiac ryanodine receptor in the force frequency relationship and heart failureTwo-photon molecular excitation imaging of Ca2+ transients in Langendorff-perfused mouse hearts.Regulation of excitation-contraction coupling in mouse cardiac myocytes: integrative analysis with mathematical modellingCross-bridge cycling gives rise to spatiotemporal heterogeneity of dynamic subcellular mechanics in cardiac myocytes probed with atomic force microscopy.A mathematical model of the murine ventricular myocyte: a data-driven biophysically based approach applied to mice overexpressing the canine NCX isoform.Calcium handling in zebrafish ventricular myocytes.Contribution of sarcoplasmic reticulum Ca²+ release and Ca²+ transporters on sarcolemmal channels to Ca²+ transient in fetal mouse heart.ACE-inhibition, but not weight reduction restores cardiomyocyte response to β-adrenergic stimulation in the metabolic syndrome.Redox regulation of cardiomyocyte cell cycling via an ERK1/2 and c-Myc-dependent activation of cyclin D2 transcriptionDiastolic myofilament dysfunction in the failing human heart.Staurosporine inhibits frequency-dependent myofilament desensitization in intact rabbit cardiac trabeculae.Computational analysis of the regulation of Ca(2+) dynamics in rat ventricular myocytesEndocardial endothelium is a key determinant of force-frequency relationship in rat ventricular myocardiumCaMKII effects on inotropic but not lusitropic force frequency responses require phospholamban.No rest for the weary: diastolic calcium homeostasis in the normal and failing myocardium.Impaired calcium homeostasis is associated with sudden cardiac death and arrhythmias in a genetic equivalent mouse model of the human HRC-Ser96Ala variant.Calcium and Excitation-Contraction Coupling in the HeartCalcium imaging at kHz frame rates resolves millisecond timing in neuronal circuits and varicosities.Systolic [Ca2+ ]i regulates diastolic levels in rat ventricular myocytes.A novel computational model of mouse myocyte electrophysiology to assess the synergy between Na+ loading and CaMKII.NFAT-dependent excitation-transcription coupling in heart.Early exercise training after myocardial infarction prevents contractile but not electrical remodelling or hypertrophy.Vagus nerve stimulation inhibits the increase in Ca2+ transient and left ventricular force caused by sympathetic nerve stimulation but has no direct effects alone--epicardial Ca2+ fluorescence studies using fura-2 AM in the isolated innervated beatiNa(+)-Ca2+ exchange function underlying contraction frequency inotropy in the cat myocardium.Pacing-induced calcineurin activation controls cardiac Ca2+ signalling and gene expression.Remodeling of excitation-contraction coupling in transgenic mice expressing ATP-insensitive sarcolemmal KATP channels.Modelling cardiac calcium sparks in a three-dimensional reconstruction of a calcium release unit.Comparison of contraction and calcium handling between right and left ventricular myocytes from adult mouse heart: a role for repolarization waveform.Slowing of cardiomyocyte Ca2+ release and contraction during heart failure progression in postinfarction mice.Regulation of systolic [Ca2+]i and cellular Ca2+ flux balance in rat ventricular myocytes by SR Ca2+, L-type Ca2+ current and diastolic [Ca2+]i.Computer model of action potential of mouse ventricular myocytes.Cardiac FKBP12.6 overexpression protects against triggered ventricular tachycardia in pressure overloaded mouse hearts.Frequency-dependent myofilament Ca2+ desensitization in failing rat myocardiumA modified approach for programmed electrical stimulation in mice: Inducibility of ventricular arrhythmias
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Mechanisms underlying the frequency dependence of contraction and [Ca(2+)](i) transients in mouse ventricular myocytes.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
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name
Mechanisms underlying the frequency dependence of contraction and [Ca
@nl
Mechanisms underlying the freq ...... in mouse ventricular myocytes.
@en
type
label
Mechanisms underlying the frequency dependence of contraction and [Ca
@nl
Mechanisms underlying the freq ...... in mouse ventricular myocytes.
@en
prefLabel
Mechanisms underlying the frequency dependence of contraction and [Ca
@nl
Mechanisms underlying the freq ...... in mouse ventricular myocytes.
@en
P2093
P2860
P1476
Mechanisms underlying the freq ...... in mouse ventricular myocytes.
@en
P2093
Gudrun Antoons
Ines Nevelsteen
Kanigula Mubagwa
Karin R Sipido
P2860
P304
P356
10.1113/JPHYSIOL.2002.025619
P407
P577
2002-09-01T00:00:00Z