Elevated glucose impairs cAMP-mediated dilation by reducing Kv channel activity in rat small coronary smooth muscle cells.
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Advanced Glycation End Products Impair Voltage-Gated K+ Channels-Mediated Coronary Vasodilation in Diabetic RatsTwenty-five years since the discovery of endothelium-derived relaxing factor (EDRF): does a dysfunctional endothelium contribute to the development of type 2 diabetes?Heart of the matter: coronary dysfunction in metabolic syndrome.Identification and functional characterization of protein kinase A-catalyzed phosphorylation of potassium channel Kv1.2 at serine 449.Relaxation of rat aorta by farrerol correlates with potency to reduce intracellular calcium of VSMCsMetabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilationEnhanced oxidative stress impairs cAMP-mediated dilation by reducing Kv channel function in small coronary arteries of diabetic rats.Vascular control in humans: focus on the coronary microcirculation.Steady-state modulation of voltage-gated K+ channels in rat arterial smooth muscle by cyclic AMP-dependent protein kinase and protein phosphatase 2BContribution of voltage-dependent K⁺ channels to metabolic control of coronary blood flow.RhoA/rock signaling mediates peroxynitrite-induced functional impairment of Rat coronary vesselsRosiglitazone selectively inhibits K(ATP) channels by acting on the K(IR) 6 subunitThe Human Microcirculation: Regulation of Flow and BeyondDistinct and complementary roles for α and β isoenzymes of PKC in mediating vasoconstrictor responses to acutely elevated glucoseAssessment of leptomeningeal collaterals using dynamic CT angiography in patients with acute ischemic stroke.Impaired Ca2+-dependent activation of large-conductance Ca2+-activated K+ channels in the coronary artery smooth muscle cells of Zucker Diabetic Fatty rats.Effects of exercise training and hypercholesterolemia on adenosine activation of voltage-dependent K+ channels in coronary arterioles.Decreased NO signaling leads to enhanced vasoconstrictor responsiveness in skeletal muscle arterioles of the ZDF rat prior to overt diabetes and hypertension.Regulation of coronary blood flow during exercise.Effects of high glucose with or without other metabolic substrates on alpha-adrenergic contractions in rat mesenteric and femoral arteries.Ser1928 phosphorylation by PKA stimulates the L-type Ca2+ channel CaV1.2 and vasoconstriction during acute hyperglycemia and diabetes.Endothelial dysfunction in diabetes: multiple targets for treatment.Role of potassium channels in coronary vasodilation.Peripheral circulation.Alterations of voltage-dependent K(+) channels in the mesenteric artery during the early and chronic phases of diabetes.Potassium Channels in Regulation of Vascular Smooth Muscle Contraction and Growth.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.Critical contribution of KV1 channels to the regulation of coronary blood flow.Rosiglitazone inhibits vascular KATP channels and coronary vasodilation produced by isoprenaline.Contribution of voltage-dependent K+ and Ca2+ channels to coronary pressure-flow autoregulation.Unveiling the Mechanism of Coronary Metabolic Vasodilation: Voltage-Gated Potassium Channels and Hydrogen Peroxide.Ebselen reduces nitration and restores voltage-gated potassium channel function in small coronary arteries of diabetic rats.Pioglitazone inhibits high glucose-induced expression of receptor for advanced glycation end products in coronary artery smooth muscle cells.Hypercholesterolemia abolishes voltage-dependent K+ channel contribution to adenosine-mediated relaxation in porcine coronary arterioles.Adaptative modifications of right coronary myocytes voltage-gated K+ currents in rat with hypoxic pulmonary hypertension.Regulation of voltage-gated potassium channels in vascular smooth muscle during hypertension and metabolic disorders.The Kv7 Channel and Cardiovascular Risk Factors.Shaker-related voltage-gated K+ channel expression and vasomotor function in human coronary resistance arteries.KV channel trafficking and control of vascular tone.Contribution of KV1.5 Channel to Hydrogen Peroxide-Induced Human Arteriolar Dilation and Its Modulation by Coronary Artery Disease.
P2860
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P2860
Elevated glucose impairs cAMP-mediated dilation by reducing Kv channel activity in rat small coronary smooth muscle cells.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
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2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
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name
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@en
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@nl
type
label
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@en
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@nl
prefLabel
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@en
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@nl
P2093
P2860
P1476
Elevated glucose impairs cAMP- ...... coronary smooth muscle cells.
@en
P2093
David D Gutterman
Hongwei Li
Qiang Chai
Yanping Liu
P2860
P304
P356
10.1152/AJPHEART.00226.2003
P577
2003-05-22T00:00:00Z