Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injury. - Wikidata - awgldk - TriplyDB

Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injury.

Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injury.

http://www.wikidata.org/entity/Q44582179

about

Cardiac-specific overexpression of caveolin-3 induces endogenous cardiac protection by mimicking ischemic preconditioning Netrin-1 prevents ischemia/reperfusion-induced myocardial infarction via a DCC/ERK1/2/eNOS s1177/NO/DCC feed-forward mechanism Gene therapy for ischemic heart disease Akt/FOXO3a/SIRT1-mediated cardioprotection by n-tyrosol against ischemic stress in rat in vivo model of myocardial infarction: switching gears toward survival and longevity Altered Nitric Oxide System in Cardiovascular and Renal Diseases Beyond Preconditioning: Postconditioning as an Alternative Technique in the Prevention of Liver Ischemia-Reperfusion Injury A New Method to Stabilize C-Kit Expression in Reparative Cardiac Mesenchymal Cells.Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxia Differential effects of selective and non-selective nitric oxide synthase inhibitors on the blood perfusion of ischemia-reperfused myocardium in dogs.Cytoprotective effects of nitrite during in vivo ischemia-reperfusion of the heart and liver Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.Protective effects of nitric oxide synthase 3 and soluble guanylate cyclase on the outcome of cardiac arrest and cardiopulmonary resuscitation in mice.Novel insights into hydrogen sulfide--mediated cytoprotection.The dynamic regulation of microcirculatory conduit function: features relevant to transfusion medicine.eNOS is required for acute in vivo ischemic preconditioning of the heart: effects of ischemic duration and sex.Inorganic nitrite and chronic tissue ischaemia: a novel therapeutic modality for peripheral vascular diseases.Soluble guanylate cyclase-α1 is required for the cardioprotective effects of inhaled nitric oxide.Tetrahydrobiopterin restores diastolic function and attenuates superoxide production in ovariectomized mRen2.Lewis rats High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.Baicalin protects the cardiomyocytes from ER stress-induced apoptosis: inhibition of CHOP through induction of endothelial nitric oxide synthase Integrative computational and experimental approaches to establish a post-myocardial infarction knowledge map.Reactive oxygen species in cardiovascular disease.Disruption of hypoxia-inducible transcription factor-prolyl hydroxylase domain-1 (PHD-1-/-) attenuates ex vivo myocardial ischemia/reperfusion injury through hypoxia-inducible factor-1α transcription factor and its target genes in mice.Neuroglobin-overexpressing transgenic mice are resistant to cerebral and myocardial ischemia.Simvastatin restores ischemic preconditioning in the presence of hyperglycemia through a nitric oxide-mediated mechanism Emergent role of gasotransmitters in ischemia-reperfusion injury.Ischemic postconditioning attenuates liver warm ischemia-reperfusion injury through Akt-eNOS-NO-HIF pathway A metabolomic study of rats with doxorubicin-induced cardiomyopathy and Shengmai injection treatment.Clinical translation of nitrite therapy for cardiovascular diseases.NO-Rich Diet for Lifestyle-Related Diseases.Placental growth factor influences maternal cardiovascular adaptation to pregnancy in mice.Selective β2-adrenoreceptor stimulation attenuates myocardial cell death and preserves cardiac function after ischemia-reperfusion injury.SIRT1 protects against myocardial ischemia-reperfusion injury via activating eNOS in diabetic rats.Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer.Dietary nitrite supplementation protects against myocardial ischemia-reperfusion injury.Efficacy of gold nanoparticles against isoproterenol induced acute myocardial infarction in adult male albino rats.Hypercholesterolemia: its impact on ischemia-reperfusion injury.Induction of activating transcription factor 3 limits survival following infarct-induced heart failure in mice.Acute and chronic cardioprotection by the enkephalin analogue, Eribis peptide 94, is mediated via activation of nitric oxide synthase and adenosine triphosphate-regulated potassium channels Redox signaling pathways involved in neuronal ischemic preconditioning

P2860

Q24336120-CC1FDA2C-F07D-4ACD-AAE0-74F0907B7DA0 Q24624234-DF09E11B-1B59-41C6-A169-F8710EE47B3C Q24632135-AF81953D-46A5-4237-AF9B-C35A6237FF08 Q24647370-8C370142-DA84-42B3-8F0E-0121921E2C12 Q26745608-EDB00189-1E76-4539-B75F-EE4A7E073554 Q26747244-59A973DA-E206-432F-9BD1-F3586DBC273B Q27330396-476CCC1C-8B56-455C-9A59-95061EE44E5A Q28278944-8473D3F3-8942-475B-8DA0-53E0166EACEC Q30452703-A55B745E-A716-4DC6-9AFF-591E6C3DC951 Q33743659-065F6827-6234-4E8D-9FC6-40E2D4BAAC8D Q33772589-8EADDEC3-6747-4CB4-BA0C-23A2DDF27517 Q33822249-9E7399B1-2033-4DD4-AB25-E95F7DF72A25 Q33830124-961F35B9-C434-43D0-8693-B17E7F1B1BC0 Q34060215-ACAE4AB7-260B-497A-9548-9097800A6CD7 Q34092872-E57DE5DC-7B61-4DB5-97B6-F6C4A771D18F Q34530182-78DB1B4E-B8FB-4262-B38B-3E9701098BA6 Q34784680-1116D442-49B3-424E-A7B6-DCF36D997F5D Q35001320-5185EE57-0402-4486-B301-ACB17BC6E17F Q35075534-24D5DB42-C34E-430A-BC54-821106B90B79 Q35092794-C2310AF3-263C-4D6D-B4E5-343C9982C930 Q35126269-6C0F317E-6F12-4E36-B234-D6F8266DA3B2 Q35165194-06C839F3-73CC-4894-AA6E-9464610E73CC Q35173596-A55EB33B-A458-4D15-9CEB-84FE276194BB Q35215113-6F0E5AC8-9F88-4B5F-81EB-4CD5E5979068 Q35230306-D7F22850-C9E1-474E-9E8E-53E89651D660 Q35321111-DA68065D-06A3-4019-A577-DFB795E154A4 Q35540329-59E45976-DECE-461E-8F41-7397BAE81E75 Q35566390-0745B0DF-1A2A-4500-B7CD-13BD2EF5D407 Q35686746-1A726C1D-1820-4241-B8FF-03B508606C1F Q35809045-0FC39646-9C5E-4AD9-AD4C-F124FD1F7415 Q35815335-0FF1D14A-1786-4814-A004-25AF2E7EE56B Q36106686-A0F3A383-D35C-4D32-ADA2-A44B303959A3 Q36200567-0A7618E9-1846-4EE2-97BC-9BBBD5F60D92 Q36229684-D6132CE3-A168-4BA4-8CA7-19DDD2989FF6 Q36277269-5A7F0BDD-AA45-4D1F-ABE0-AD5429A5C84D Q36301960-8D22EAE7-3870-4682-8F53-B4A2AE70FE37 Q36314857-C93E1D59-129E-483A-B829-8C1AA39AB6F6 Q36339385-58630134-059D-46D8-BA43-88B266CF13FA Q36350926-231D1F7F-0824-4E40-832A-C0FBF80393F7 Q36460627-B28FE0D9-0BB5-424E-9D74-2D5B5D50C13A

P2860

Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injury.

http://www.wikidata.org/entity/Q44582179

description

2003 nî lūn-bûn

@nan

2003年の論文

@ja

2003年学术文章

@wuu

2003年学术文章

@zh

2003年学术文章

@zh-cn

2003年学术文章

@zh-hans

2003年学术文章

@zh-my

2003年学术文章

@zh-sg

2003年學術文章

@yue

2003年學術文章

@zh-hant

name

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@en

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@nl

type

label

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@en

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@nl

prefLabel

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@en

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@nl

P1433

Q44582179-714AD8E6-53F4-424B-8947-1EEBA1DFFFC0

P1476

Q44582179-461384E9-2B69-4DF6-A573-A0A3CF25DE54

P2093

Q44582179-028C0D31-F84E-4C84-A8CA-8CD5B0CB691C

Q44582179-0A317B68-AA7D-4E24-8AC0-A635E2671C07

Q44582179-227C22CE-4ACE-4B5C-B0E9-423A33F74B46

Q44582179-2520303D-8B04-49D9-ADBC-097116CA906F

Q44582179-5BFA2E2A-0EB3-47D1-900B-5D0F6BF92DE3

Q44582179-AAF8A02D-FBFC-464A-9DEB-C5CE2F68BF10

Q44582179-C06A696C-EFAE-45CF-8ABA-0DD95934AA51

Q44582179-C7FADFC4-7993-42EC-96EF-CAF96BB1F3FC

Q44582179-E1BF5737-F758-42AF-9DDD-CCDC63D2B3A7

Q44582179-F8D01ECC-3894-46DB-986A-82934FBE1CBA

P2860

Q44582179-14D13758-E879-429E-90DA-EDF347B63ED9

Q44582179-1FF8FF36-1A4C-45F4-BF1C-8968F8DB2F43

Q44582179-2026FA4B-B9AD-4503-98A7-2D860AA811EE

Q44582179-2062D7DB-DB16-4797-99FF-090EF1EBE300

Q44582179-207248A0-9C95-455D-A28A-BD50ADBB3B21

Q44582179-2826AD7F-64E7-46E0-AE73-963275F50D62

Q44582179-2E989A90-852E-4363-AA24-1AE1011C4A9B

Q44582179-4EC2828C-E290-4CED-AF87-F8CBF8AA9FDA

Q44582179-55D1B732-54DA-4E5A-9352-B8AB65490D27

Q44582179-579B3750-AEF5-47AC-8306-72CCBBCF3D1A

P304

Q44582179-1ADC7990-1C54-4040-B0C9-5591983F56C5

P31

Q44582179-0AFE4A5F-F0AF-4A5D-A580-889088917709

P356

Q44582179-54F76BCF-0922-41A8-85FB-13A1B8E3320A

P433

Q44582179-B2E7DF3C-4A42-4C32-BF13-B2C5B4B0F84B

P478

Q44582179-1B8C14E6-B0EE-4D7B-84A2-DC088CFDFFF7

P50

Q44582179-788D1BA5-44DB-44E1-9522-ECC97AC19B11

P577

Q44582179-8D7F8256-42B0-42B0-8FD5-1D981530505A

P5875

Q44582179-06B99E30-1C35-44C1-8FE0-56E8E4CA756E

P698

Q44582179-AC2D3740-EEC2-4FA4-B20B-BAF2D9712502

P921

Q44582179-A178B074-AC4E-422F-A646-985C8282F33B

Q44582179-B065AC9F-AC54-42F0-BFCB-AEEF1EA02DAA

Q44582179-FCE2F255-10F0-44C0-BD99-C267ED7B947D

P356

AJPHEART.00129.2003

P1433

American Journal of Physiology - Heart and Circulatory Physiology

P1476

Endothelial nitric oxide synth ...... myocardial reperfusion injury.

@en

P2093

Aman K Kakkar

http://www.w3.org/2001/XMLSchema#string

David J Lefer

http://www.w3.org/2001/XMLSchema#string

James J M Greer

http://www.w3.org/2001/XMLSchema#string

Michael Hicks

http://www.w3.org/2001/XMLSchema#string

Mitsuhiro Yokoyama

http://www.w3.org/2001/XMLSchema#string

P Derek Ware

http://www.w3.org/2001/XMLSchema#string

Richard H Turnage

http://www.w3.org/2001/XMLSchema#string

Rien van Haperen

http://www.w3.org/2001/XMLSchema#string

Rini de Crom

http://www.w3.org/2001/XMLSchema#string

Seinosuke Kawashima

http://www.w3.org/2001/XMLSchema#string

P2860

Role of the arginine-nitric oxide pathway in the regulation of vascular smooth muscle cell proliferation

The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

Inducible nitric oxide synthase mediates delayed myocardial protection induced by activation of adenosine A(1) receptors: evidence from gene-knockout mice.

Endothelial NO synthase overexpression inhibits lesion formation in mouse model of vascular remodeling.

Nitric oxide donors and cardiovascular agents modulating the bioactivity of nitric oxide: an overview.

Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.

Endothelial nitric oxide synthase overexpression attenuates congestive heart failure in mice.

Hypotension and reduced nitric oxide-elicited vasorelaxation in transgenic mice overexpressing endothelial nitric oxide synthase.

Nitric oxide: an endogenous modulator of leukocyte adhesion.

Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size.

P304

H276-82

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1152/AJPHEART.00129.2003

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

286

http://www.w3.org/2001/XMLSchema#string

P50

P577

2003-09-11T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

10571188

http://www.w3.org/2001/XMLSchema#string

P698

12969888

http://www.w3.org/2001/XMLSchema#string

P921

reperfusion injury