Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
about
Leukocyte tyrosine kinase functions in pigment cell developmentAnaplastic lymphoma kinase: signalling in development and diseasep130Cas mediates the transforming properties of the anaplastic lymphoma kinaseAnaplastic lymphoma kinase is activated through the pleiotrophin/receptor protein-tyrosine phosphatase beta/zeta signaling pathway: an alternative mechanism of receptor tyrosine kinase activationThe role of the ALK receptor in cancer biologyMolecular mechanisms that underpin EML4-ALK driven cancers and their response to targeted drugsEmerging importance of ALK in neuroblastomaInsulin/IGF-regulated size scaling of neuroendocrine cells expressing the bHLH transcription factor Dimmed in DrosophilaThe Rap1 guanine nucleotide exchange factor C3G is required for preservation of larval muscle integrity in Drosophila melanogasterFAM150A and FAM150B are activating ligands for anaplastic lymphoma kinaseGlobal gene expression profiling of PAX-FKHR fusion-positive alveolar and PAX-FKHR fusion-negative embryonal rhabdomyosarcomasExpression-guided in silico evaluation of candidate cis regulatory codes for Drosophila muscle founder cellsAnaplastic lymphoma kinase activates the small GTPase Rap1 via the Rap1-specific GEF C3G in both neuroblastoma and PC12 cellsIdentification of receptor-tyrosine-kinase-signaling target genes reveals receptor-specific activities and pathway branchpoints during Drosophila developmentGenetic control of fusion pore expansion in the epidermis of Caenorhabditis elegans.The FGF8-related signals Pyramus and Thisbe promote pathfinding, substrate adhesion, and survival of migrating longitudinal gut muscle founder cells.Hand is a direct target of the forkhead transcription factor Biniou during Drosophila visceral mesoderm differentiation.Characterisation of the role of Vrp1 in cell fusion during the development of visceral muscle of Drosophila melanogasterAn evolutionary conserved role for anaplastic lymphoma kinase in behavioral responses to ethanol.The receptor tyrosine kinase Alk controls neurofibromin functions in Drosophila growth and learning.HLH54F is required for the specification and migration of longitudinal gut muscle founders from the caudal mesoderm of DrosophilaThe Drosophila midkine/pleiotrophin homologues Miple1 and Miple2 affect adult lifespan but are dispensable for alk signaling during embryonic gut formation.Anaplastic lymphoma kinase is required for neurogenesis in the developing central nervous system of zebrafish.Pleiotrophin as a central nervous system neuromodulator, evidences from the hippocampus.Distinct genetic programs guide Drosophila circular and longitudinal visceral myoblast fusion.Glycosylated synaptomatrix regulation of trans-synaptic signaling.Anaplastic Lymphoma Kinase Acts in the Drosophila Mushroom Body to Negatively Regulate Sleep.Pathobiology of ALK+ anaplastic large-cell lymphoma.What have we learnt from mouse models of NPM-ALK-induced lymphomagenesis?The Zic family homologue Odd-paired regulates Alk expression in DrosophilaC. elegans anaplastic lymphoma kinase ortholog SCD-2 controls dauer formation by modulating TGF-beta signalingFusion tyrosine kinase mediated signalling pathways in the transformation of haematopoietic cells.Org-1 is required for the diversification of circular visceral muscle founder cells and normal midgut morphogenesis.Receptor tyrosine kinases in Drosophila development.Targeting ALK in neuroblastoma--preclinical and clinical advancements.Fragile X mental retardation protein regulates trans-synaptic signaling in DrosophilaAnaplastic lymphoma kinase: role in cancer pathogenesis and small-molecule inhibitor development for therapy.The midkine family of growth factors: diverse roles in nervous system formation and maintenance.Treating ALK-positive lung cancer--early successes and future challenges.Current status of targeted therapy for anaplastic lymphoma kinase-rearranged non-small cell lung cancer.
P2860
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P2860
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@en
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@nl
type
label
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@en
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@nl
prefLabel
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@en
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion.
@nl
P2093
P2860
P50
P356
P1433
P1476
Jeb signals through the Alk receptor tyrosine kinase to drive visceral muscle fusion
@en
P2093
Camilla Englund
Caroline Grabbe
Christina E Lorén
Fabienne Deleuil
P2860
P2888
P304
P356
10.1038/NATURE01950
P407
P577
2003-10-01T00:00:00Z
P6179
1012399690