4-1BB-mediated immunotherapy of rheumatoid arthritis. - Wikidata - awgldk - TriplyDB

4-1BB-mediated immunotherapy of rheumatoid arthritis.

4-1BB-mediated immunotherapy of rheumatoid arthritis.

http://www.wikidata.org/entity/Q45078356

about

Early rheumatoid arthritis is characterized by a distinct and transient synovial fluid cytokine profile of T cell and stromal cell origin.The role of costimulatory receptors of the tumour necrosis factor receptor family in atherosclerosis Novel immunotherapies for hematologic malignancies Trial watch: IDO inhibitors in cancer therapy 4-1BB Agonists: Multi-Potent Potentiators of Tumor Immunity IDO2: A Pathogenic Mediator of Inflammatory Autoimmunity CD137 and CD137L signals are main drivers of type 1, cell-mediated immune responses Galectin-9 controls the therapeutic activity of 4-1BB-targeting antibodies Cytokine-mediated disruption of lymphocyte trafficking, hemopoiesis, and induction of lymphopenia, anemia, and thrombocytopenia in anti-CD137-treated mice Therapeutic potential of anti-CD137 (4-1BB) monoclonal antibodies.Expression of 4-1BB and 4-1BBL in thymocytes during thymus regeneration Mechanisms of self-nonself discrimination and possible clinical relevance.Adjuvantive effects of anti-4-1BB agonist Ab and 4-1BBL DNA for a HIV-1 Gag DNA vaccine: different effects on cellular and humoral immunity.Disease exacerbation of multiple sclerosis is characterized by loss of terminally differentiated autoregulatory CD8+ T cells.MicroRNAs profiling in murine models of acute and chronic asthma: a relationship with mRNAs targets.CD8+ Tregs in lupus, autoimmunity, and beyond.Role of 4-1BB receptor in the control played by CD8(+) T cells on IFN-gamma production by Mycobacterium tuberculosis antigen-specific CD4(+) T Cells Agonistic Anti-CD137 Monoclonal Antibody Treatment Induces CD11bGr-1 Myeloid-derived Suppressor Cells.Common gamma chain cytokines promote rapid in vitro expansion of allo-specific human CD8+ suppressor T cells 4-1BB (CD137), an inducible costimulatory receptor, as a specific target for cancer therapy CD8+ Treg cells suppress CD8+ T cell-responses by IL-10-dependent mechanism during H5N1 influenza virus infection.4-1BB signaling breaks the tolerance of maternal CD8+ T cells that are reactive with alloantigens.CD137-mediated immunotherapy for allergic asthma Exposure of a distinct PDCA-1+ (CD317) B cell population to agonistic anti-4-1BB (CD137) inhibits T and B cell responses both in vitro and in vivo.CD137 expression is induced by Epstein-Barr virus infection through LMP1 in T or NK cells and mediates survival promoting signals.Lipopolysaccharide-induced depressive-like behavior is mediated by indoleamine 2,3-dioxygenase activation in mice.CD8(+) T Cells Are Required For Glatiramer Acetate Therapy in Autoimmune Demyelinating Disease.Immune checkpoint receptors in regulating immune reactivity in rheumatic disease.CD137 facilitates the resolution of acute DSS-induced colonic inflammation in mice Small intestine CD11c+ CD8+ T cells suppress CD4+ T cell-induced immune colitis.Indoleamine 2,3-dioxygenase as a modifier of pathogenic inflammation in cancer and other inflammation-associated diseases.The tumor necrosis factor superfamily of cytokines in the inflammatory myopathies: potential targets for therapy.CD8+ T cell dysfunction and increase in murine gammaherpesvirus latent viral burden in the absence of 4-1BB ligand Deficiency for costimulatory receptor 4-1BB protects against obesity-induced inflammation and metabolic disorders Inhibition of 4-1BBL-regulated TLR response in macrophages ameliorates endotoxin-induced sepsis in mice.Hu.4-1BB-Fc fusion protein inhibits allergic inflammation and airway hyperresponsiveness in a murine model of asthma Reverse signaling through the costimulatory ligand CD137L in epithelial cells is essential for natural killer cell-mediated acute tissue inflammation.Heat Shock Protein 60 in Eggs Specifically Induces Tregs and Reduces Liver Immunopathology in Mice with Schistosomiasis Japonica CD137 stimulation delivers an antigen-independent growth signal for T lymphocytes with memory phenotype Altered tryptophan metabolism as a paradigm for good and bad aspects of immune privilege in chronic inflammatory diseases.

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4-1BB-mediated immunotherapy of rheumatoid arthritis.

http://www.wikidata.org/entity/Q45078356

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@en

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@nl

type

label

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@en

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@nl

prefLabel

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@en

4-1BB-mediated immunotherapy of rheumatoid arthritis.

@nl

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Nature Medicine

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4-1BB-mediated immunotherapy of rheumatoid arthritis

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P2093

Beom K Choi

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Byoung S Kwon

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Dass S Vinay

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Hye Y Park

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Jae H Choi

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Jae H Suh

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Su K Seo

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Young H Kim

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P2860

Prevention of allogeneic fetal rejection by tryptophan catabolism

The molecular pathogenesis of collagen-induced arthritis in mice--a model for rheumatoid arthritis.

Effector CD8 T cells possess suppressor function after 4-1BB and Toll-like receptor triggering

CD137 costimulatory T cell receptor engagement reverses acute disease in lupus-prone NZB x NZW F1 mice

Tryptophan catabolism and regulation of adaptive immunity.

Anti-4-1BB monoclonal antibodies abrogate T cell-dependent humoral immune responses in vivo through the induction of helper T cell anergy.

Inhibition of allogeneic T cell proliferation by indoleamine 2,3-dioxygenase-expressing dendritic cells: mediation of suppression by tryptophan metabolites.

4-1BB costimulatory signals preferentially induce CD8+ T cell proliferation and lead to the amplification in vivo of cytotoxic T cell responses

Therapeutic activity of agonistic monoclonal antibodies against CD40 in a chronic autoimmune inflammatory process.

Pathogenesis of the joint disease of rheumatoid arthritis.

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10.1038/NM1107

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10

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1034622205

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15448685

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