Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear. - Wikidata - awgldk - TriplyDB

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

http://www.wikidata.org/entity/Q45215137

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Endocannabinoid-mediated long-term plasticity requires cAMP/PKA signaling and RIM1alpha Pharmacological treatment of PTSD - established and new approaches Cannabinoids in health and disease Smoking tobacco along with marijuana increases symptoms of cannabis dependence Modulation of Fear Extinction by Stress, Stress Hormones and Estradiol: A Review.From Pavlov to PTSD: the extinction of conditioned fear in rodents, humans, and anxiety disorders.Amygdala microcircuits controlling learned fear Neural and cellular mechanisms of fear and extinction memory formation Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disorders Amygdala FAAH and anandamide: mediating protection and recovery from stress Translational evidence for a role of endocannabinoids in the etiology and treatment of posttraumatic stress disorder The neurobiological bases of memory formation: from physiological conditions to psychopathology 2-arachidonoylglycerol signaling impairs short-term fear extinction.PTSD: from neurobiology to pharmacological treatments Extinction circuits for fear and addiction overlap in prefrontal cortex Mechanisms of fear extinction The role of CB1 receptors in psychostimulant addiction Mechanisms to medicines: elucidating neural and molecular substrates of fear extinction to identify novel treatments for anxiety disorders Endocannabinoid-mediated synaptic plasticity and addiction-related behavior Neural mechanisms of extinction learning and retrieval The CB1 inverse agonist AM251, but not the CB1 antagonist AM4113, enhances retention of contextual fear conditioning in rats Precondition of right frontal region with anodal tDCS can restore the fear memory impairment induced by ACPA in male mice.Common Polymorphisms in the Age of Research Domain Criteria (RDoC): Integration and Translation.Developmental regulation of fear learning and anxiety behavior by endocannabinoids.Does Cannabis Cause, Exacerbate or Ameliorate Psychiatric Disorders? An Oversimplified Debate Discussed Stress and Fear Extinction.Interaction between the cholecystokinin and endogenous cannabinoid systems in cued fear expression and extinction retention Can fear extinction be enhanced? A review of pharmacological and behavioral findings.Differential Expression of Brain Cannabinoid Receptors between Repeatedly Stressed Males and Females may Play a Role in Age and Gender-Related Difference in Traumatic Brain Injury: Implications from Animal Studies.Learning and memory during sleep and anesthesia.Conditioned fear associated phenotypes as robust, translational indices of trauma-, stressor-, and anxiety-related behaviors Recent progress in understanding the pathophysiology of post-traumatic stress disorder: implications for targeted pharmacological treatment.Conditioned fear extinction and reinstatement in a human fear-potentiated startle paradigm How the neurocircuitry and genetics of fear inhibition may inform our understanding of PTSD.Pharmacological enhancement of fear reduction: preclinical models.Pharmacological elevation of anandamide impairs short-term memory by altering the neurophysiology in the hippocampus.Seeking a spotless mind: extinction, deconsolidation, and erasure of fear memory The endocannabinoid system in anxiety, fear memory and habituation.Effects of the novel endocannabinoid uptake inhibitor, LY2183240, on fear-potentiated startle and alcohol-seeking behaviors in mice selectively bred for high alcohol preference.Endocannabinoid system and psychiatry: in search of a neurobiological basis for detrimental and potential therapeutic effects.

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P2860

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

http://www.wikidata.org/entity/Q45215137

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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name

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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type

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Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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prefLabel

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

@en

Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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Neuropsychopharmacology

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Enhancing cannabinoid neurotransmission augments the extinction of conditioned fear.

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Jasmeer P Chhatwal

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Kimberly A Maguschak

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Michael Davis

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P2860

Molecular characterization of human and mouse fatty acid amide hydrolases

Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase

Role of endogenous cannabinoids in synaptic signaling

Behavioral and neural analysis of extinction

The analgesic effects of R(+)-WIN 55,212-2 mesylate, a high affinity cannabinoid agonist, in a rat model of neuropathic pain

Modulation of anxiety through blockade of anandamide hydrolysis

CB1 cannabinoid receptors modulate kinase and phosphatase activity during extinction of conditioned fear in mice.

Localization of the CB1 type cannabinoid receptor in the rat basolateral amygdala: high concentrations in a subpopulation of cholecystokinin-containing interneurons.

The endocannabinoid nervous system: unique opportunities for therapeutic intervention.

Anandamide transport is independent of fatty-acid amide hydrolase activity and is blocked by the hydrolysis-resistant inhibitor AM1172.

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516-524

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10.1038/SJ.NPP.1300655

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3

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30

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Kerry J. Ressler

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8092934

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15637635

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