ANG II receptor blockade prevents ventricular hypertrophy and ANF gene expression with pressure overload in mice.
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The knockout of miR-143 and -145 alters smooth muscle cell maintenance and vascular homeostasis in mice: correlates with human diseaseA-kinase-anchoring protein-Lbc anchors IκB kinase β to support interleukin-6-mediated cardiomyocyte hypertrophyPressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient miceThrombospondin-4 regulates fibrosis and remodeling of the myocardium in response to pressure overloadAnti-remodeling effects of rapamycin in experimental heart failure: dose response and interaction with angiotensin receptor blockadeRac1 and RhoA differentially regulate angiotensinogen gene expression in stretched cardiac fibroblastsIL-33 and ST2 comprise a critical biomechanically induced and cardioprotective signaling systemCardioprotective effect of nicorandil, a mitochondrial ATP-sensitive potassium channel opener, prolongs survival in HSPB5 R120G transgenic miceUltrasound based assessment of coronary artery flow and coronary flow reserve using the pressure overload model in mice.Comprehensive cardiovascular magnetic resonance of myocardial mechanics in mice using three-dimensional cine DENSE.Lrrc10 is required for early heart development and function in zebrafishPhosphatase-resistant gap junctions inhibit pathological remodeling and prevent arrhythmias.Platelet TGF-β1 contributions to plasma TGF-β1, cardiac fibrosis, and systolic dysfunction in a mouse model of pressure overload.Effects of carvedilol on structural and functional outcomes and plasma biomarkers in the mouse transverse aortic constriction heart failure model.Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets?The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis.Insulin-like growth factor-1 enhances ventricular hypertrophy and function during the onset of experimental cardiac failureOverexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction.Animal models of cardiovascular diseasesAdministration of a Synthetic Peptide Derived from the E-domain Region of Mechano-Growth Factor Delays Decompensation Following Myocardial Infarction.Hypertrophic growth in cardiac myocytes is mediated by Myc through a Cyclin D2-dependent pathway.The role of renin angiotensin system antagonists in the prevention of doxorubicin and trastuzumab induced cardiotoxicityANG II modulation of cardiac growth and remodeling in immature fetal sheep.Apolipoprotein J/clusterin induction in myocarditis: A localized response gene to myocardial injuryTlr4 Deficiency Protects against Cardiac Pressure Overload Induced HyperinflammationTlr2 deficiency does not limit the development of left ventricular hypertrophy in a model of transverse aortic constriction induced pressure overloadInhibition of angiotensin II Gq signaling augments beta-adrenergic receptor mediated effects in a renal artery stenosis model of high blood pressureMAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets.Receptor-specific in vivo desensitization by the G protein-coupled receptor kinase-5 in transgenic mice.Enhanced myocardial relaxation in vivo in transgenic mice overexpressing the beta2-adrenergic receptor is associated with reduced phospholamban proteinConducting the G-protein Coupled Receptor (GPCR) Signaling Symphony in Cardiovascular Diseases: New Therapeutic ApproachesIsoproterenol-induced myocardial injury and diastolic dysfunction in mice: structural and functional correlates.Evidence for a role of mast cells in the evolution to congestive heart failure.Regulation of RNA polymerase III transcription during hypertrophic growth.Gap junction remodeling and spironolactone-dependent reverse remodeling in the hypertrophied heart.Differential patterns of replacement and reactive fibrosis in pressure and volume overload are related to the propensity for ischaemia and involve resistin.Angiotensin II in cardiac pressure-overload hypertrophy in fetal sheep.How does pressure overload cause cardiac hypertrophy and dysfunction? High-ouabain affinity cardiac Na+ pumps are crucial.
P2860
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P2860
ANG II receptor blockade prevents ventricular hypertrophy and ANF gene expression with pressure overload in mice.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh-hant
name
ANG II receptor blockade preve ...... ith pressure overload in mice.
@en
ANG II receptor blockade preve ...... ith pressure overload in mice.
@nl
type
label
ANG II receptor blockade preve ...... ith pressure overload in mice.
@en
ANG II receptor blockade preve ...... ith pressure overload in mice.
@nl
prefLabel
ANG II receptor blockade preve ...... ith pressure overload in mice.
@en
ANG II receptor blockade preve ...... ith pressure overload in mice.
@nl
P2093
P1476
ANG II receptor blockade preve ...... ith pressure overload in mice.
@en
P2093
P304
P356
10.1152/AJPHEART.1994.266.6.H2468
P407
P433
P577
1994-06-01T00:00:00Z