Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism. - Wikidata - awgldk - TriplyDB

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

http://www.wikidata.org/entity/Q46031528

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From mechanosensitivity to inflammatory responses: new players in the pathology of glaucoma Redefining the classification of AMPA-selective ionotropic glutamate receptors Pax6-positive Müller glia cells express cell cycle markers but do not proliferate after photoreceptor injury in the mouse retina Chronic and acute models of retinal neurodegeneration TrkA activity are neuroprotective whereas p75NTR activity is neurotoxic through a paracrine mechanism Interaction and antagonistic roles of NF-κB and Hes6 in the regulation of cortical neurogenesis.Beneficial effects of secretory leukocyte protease inhibitor after spinal cord injury.ProNGF induces TNFalpha-dependent death of retinal ganglion cells through a p75NTR non-cell-autonomous signaling pathway Reactive microglia and macrophage facilitate the formation of Müller glia-derived retinal progenitors.Etanercept, a widely used inhibitor of tumor necrosis factor-α (TNF-α), prevents retinal ganglion cell loss in a rat model of glaucoma.Glucocorticoid receptors in the retina, Müller glia and the formation of Müller glia-derived progenitors.The reactivity, distribution and abundance of Non-astrocytic Inner Retinal Glial (NIRG) cells are regulated by microglia, acute damage, and IGF1 Steady-state levels of retinal 24S-hydroxycholesterol are maintained by glial cells intervention after elevation of intraocular pressure in the rat.Structural and functional neuroprotection in glaucoma: role of galantamine-mediated activation of muscarinic acetylcholine receptors Neuroprotective effect of lovastatin by inhibiting NMDA receptor1 in 6-hydroxydopamine treated PC12 cells Tumor necrosis factor alpha has an early protective effect on retinal ganglion cells after optic nerve crush.In glaucoma the upregulated truncated TrkC.T1 receptor isoform in glia causes increased TNF-alpha production, leading to retinal ganglion cell death.Reactive retinal microglia, neuronal survival, and the formation of retinal folds and detachments.A cell-permeable phosphine-borane complex delays retinal ganglion cell death after axonal injury through activation of the pro-survival extracellular signal-regulated kinases 1/2 pathway.The role of N-methyl-D-aspartate receptor activation in homocysteine-induced death of retinal ganglion cells Calcium-permeable AMPA receptors in the retina.Genetic deletion of TNF receptor suppresses excitatory synaptic transmission via reducing AMPA receptor synaptic localization in cortical neurons.Lovastatin induces neuroprotection by inhibiting inflammatory cytokines in 6-hydroxydopamine treated microglia cells Cell swelling contributes to thickening of low-dose N-methyl-D-aspartate-induced retinal edema.REDD2-mediated inhibition of mTOR promotes dendrite retraction induced by axonal injury The Paradoxical Signals of Two TrkC Receptor Isoforms Supports a Rationale for Novel Therapeutic Strategies in ALS.Activation of glucocorticoid receptors in Müller glia is protective to retinal neurons and suppresses microglial reactivity.Intrinsically photosensitive retinal ganglion cells are resistant to N-methyl-D-aspartic acid excitotoxicity.Neuroinflammation in glaucoma: soluble tumor necrosis factor alpha and the connection with excitotoxic damage.Retinal glial responses to optic nerve crush are attenuated in Bax-deficient mice and modulated by purinergic signaling pathways.Insulin-like growth factor I (IGF-I)-induced chronic gliosis and retinal stress lead to neurodegeneration in a mouse model of retinopathy Small Interfering RNA Targeted to ASPP2 Promotes Progression of Experimental Proliferative Vitreoretinopathy.Pharmacologic inhibition of reactive gliosis blocks TNF-α-mediated neuronal apoptosis.Optineurin insufficiency impairs IRF3 but not NF-κB activation in immune cells.The cell and molecular biology of glaucoma: mechanisms of retinal ganglion cell death.AMPA-receptor trafficking and injury-induced cell death.Cell non-autonomous functions of homeoproteins in neuroprotection in the brain.Gene therapy for retinal ganglion cell neuroprotection in glaucoma.Thinking of Co²⁺-staining explant tissue or cultured cells? How to make it reliable and specific.Autocrine and paracrine interactions and neuroprotection in glaucoma.The role of Müller glia and microglia in glaucoma.

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P2860

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

http://www.wikidata.org/entity/Q46031528

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

@zh-my

2009年学术文章

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2009年學術文章

@yue

2009年學術文章

@zh-hant

name

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@en

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@nl

type

label

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@en

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@nl

prefLabel

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@en

Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism.

@nl

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JNEUROSCI.0831-09.2009

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Journal of Neuroscience

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Excitotoxic death of retinal neurons in vivo occurs via a non-cell-autonomous mechanism

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Adriana Di Polo

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Frédéric Lebrun-Julien

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Ingrid Osswald

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Kathleen Dickson

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Laure Duplan

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Philip A Barker

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Philippe Bourgeois

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Przemyslaw Sapieha

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Vincent Pernet

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5536-5545

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scholarly article

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10.1523/JNEUROSCI.0831-09.2009

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