Increased pentose phosphate pathway flux after clinical traumatic brain injury: a [1,2-13C2]glucose labeling study in humans.
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Stable isotope-resolved metabolomics and applications for drug developmentMetabolic crisis in severely head-injured patients: is ischemia just the tip of the iceberg?Insights into the metabolic response to traumatic brain injury as revealed by (13)C NMR spectroscopyCerebral metabolism following traumatic brain injury: new discoveries with implications for treatmentEnhanced neuronal glucose transporter expression reveals metabolic choice in a HD Drosophila modelUsing bioconductor package BiGGR for metabolic flux estimation based on gene expression changes in brainGlycolysis and the pentose phosphate pathway after human traumatic brain injury: microdialysis studies using 1,2-(13)C2 glucose.Lactate shuttling and lactate use as fuel after traumatic brain injury: metabolic considerations(13)C-labelled microdialysis studies of cerebral metabolism in TBI patients.Glucose metabolism following human traumatic brain injury: methods of assessment and pathophysiological findings.Glycolysis and the significance of lactate in traumatic brain injury.Glucose and Intermediary Metabolism and Astrocyte-Neuron Interactions Following Neonatal Hypoxia-Ischemia in Rat.Structural imaging of mild traumatic brain injury may not be enough: overview of functional and metabolic imaging of mild traumatic brain injury.Dietary branched chain amino acids ameliorate injury-induced cognitive impairment.Blast-related brain injury: imaging for clinical and research applications: report of the 2008 st. Louis workshop.Metabolic and histologic effects of sodium pyruvate treatment in the rat after cortical contusion injuryBrain metabolism is significantly impaired at blood glucose below 6 mM and brain glucose below 1 mM in patients with severe traumatic brain injury.Lactate and the lactate-to-pyruvate molar ratio cannot be used as independent biomarkers for monitoring brain energetic metabolism: a microdialysis study in patients with traumatic brain injuriesCompartmentalized Cerebral Metabolism of [1,6-(13)C]Glucose Determined by in vivo (13)C NMR Spectroscopy at 14.1 T.Animal modelling of traumatic brain injury in preclinical drug development: where do we go from here?Glucose administration after traumatic brain injury exerts some benefits and no adverse effects on behavioral and histological outcomes.The return of metabolism: biochemistry and physiology of the pentose phosphate pathwayQuantitative importance of the pentose phosphate pathway determined by incorporation of 13C from [2-13C]- and [3-13C]glucose into TCA cycle intermediates and neurotransmitter amino acids in functionally intact neurons.Density-Dependent Metabolic Heterogeneity in Human Mesenchymal Stem Cells.(13)C metabolic flux analysis in neurons utilizing a model that accounts for hexose phosphate recycling within the pentose phosphate pathway.Microdialysis: is it ready for prime time?Characterization of glucose-related metabolic pathways in differentiated rat oligodendrocyte lineage cellsGlucose metabolism via the pentose phosphate pathway, glycolysis and Krebs cycle in an orthotopic mouse model of human brain tumors(13)C MRS and LC-MS Flux Analysis of Tumor Intermediary Metabolism.Differential influence of arterial blood glucose on cerebral metabolism following severe traumatic brain injuryGlucose administration after traumatic brain injury improves cerebral metabolism and reduces secondary neuronal injury.Lactate uptake by the injured human brain: evidence from an arteriovenous gradient and cerebral microdialysis study.Evaluation of the Effect of Glibenclamide in Patients With Diffuse Axonal Injury Due to Moderate to Severe Head TraumaPotential non-hypoxic/ischemic causes of increased cerebral interstitial fluid lactate/pyruvate ratio: a review of available literature.Studies of isolated global brain ischaemia: I. Overview of irreversible brain injury and evolution of a new concept - redefining the time of brain death.Cerebral microdialysis in traumatic brain injury and subarachnoid hemorrhage: state of the art.Metabolic evaluations of cancer metabolism by NMR-based stable isotope tracer methodologies.A comprehensive metabolic profile of cultured astrocytes using isotopic transient metabolic flux analysis and C-labeled glucose.Cerebral Lactate Metabolism After Traumatic Brain Injury.Mammalian Circadian Period, But Not Phase and Amplitude, Is Robust Against Redox and Metabolic Perturbations.
P2860
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P2860
Increased pentose phosphate pathway flux after clinical traumatic brain injury: a [1,2-13C2]glucose labeling study in humans.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Increased pentose phosphate pa ...... cose labeling study in humans.
@en
Increased pentose phosphate pa ...... cose labeling study in humans.
@nl
type
label
Increased pentose phosphate pa ...... cose labeling study in humans.
@en
Increased pentose phosphate pa ...... cose labeling study in humans.
@nl
prefLabel
Increased pentose phosphate pa ...... cose labeling study in humans.
@en
Increased pentose phosphate pa ...... cose labeling study in humans.
@nl
P2093
P2860
P50
P356
P1476
Increased pentose phosphate pa ...... cose labeling study in humans.
@en
P2093
Daniel F Kelly
David A Hovda
Joshua R Dusick
Stefan M Lee
W N Paul Lee
P2860
P304
P356
10.1038/SJ.JCBFM.9600458
P577
2007-02-07T00:00:00Z