Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb. - Wikidata - awgldk - TriplyDB

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

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Chemistry and antihypertensive effects of tempol and other nitroxides Why is diabetes mellitus a risk factor for contrast-induced nephropathy?Molecular regulation of NKCC2 in the thick ascending limb p22phox in the macula densa regulates single nephron GFR during angiotensin II infusion in rats Angiotensin II-induced hypertension increases plasma membrane Na pump activity by enhancing Na entry in rat thick ascending limbs Effects of Reactive Oxygen Species on Tubular Transport along the Nephron Angiotensin II stimulates thick ascending limb superoxide production via protein kinase C(α)-dependent NADPH oxidase activation Cyp2c44 epoxygenase in the collecting duct is essential for the high K+ intake-induced antihypertensive effect.NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes Endogenous flow-induced superoxide stimulates Na/H exchange activity via PKC in thick ascending limbs.NAD(P)H oxidase and renal epithelial ion transport The proximal tubule in the pathophysiology of the diabetic kidney.HV1 acts as a sodium sensor and promotes superoxide production in medullary thick ascending limb of Dahl salt-sensitive rats Endogenous flow-induced nitric oxide reduces superoxide-stimulated Na/H exchange activity via PKG in thick ascending limbs Iodinated contrast media cause direct tubular cell damage, leading to oxidative stress, low nitric oxide, and impairment of tubuloglomerular feedback.Renal Inhibition of Heme Oxygenase-1 Increases Blood Pressure in Angiotensin II-Dependent Hypertension Bilirubin, renal hemodynamics, and blood pressure.Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.Angiotensin II stimulates epithelial sodium channels in the cortical collecting duct of the rat kidney Expression of heme oxygenase-1 in thick ascending loop of henle attenuates angiotensin II-dependent hypertension Role of carbon monoxide in kidney function: is a little carbon monoxide good for the kidney?Lack of significant renal tubular injury despite acute kidney injury in acute decompensated heart failure.NADPH oxidase inhibition reduces tubular sodium transport and improves kidney oxygenation in diabetes.Mechanisms of angiotensin II-enhanced connecting tubule glomerular feedback.Angiotensin II type 2 receptor-mediated inhibition of NaCl absorption is blunted in thick ascending limbs from Dahl salt-sensitive rats.Angiotensin II stimulates superoxide production in the thick ascending limb by activating NOX4.NADPH oxidase 4 mediates flow-induced superoxide production in thick ascending limbs.Relation of systemic and urinary neutrophil gelatinase-associated lipocalin levels to different aspects of impaired renal function in patients with acute decompensated heart failure Angiotensin II stimulates superoxide production by nitric oxide synthase in thick ascending limbs.Renal intramedullary infusion of tempol normalizes the blood pressure response to intrarenal blockade of heme oxygenase-1 in angiotensin II-dependent hypertension Angiotensin II stimulates basolateral 10-pS Cl channels in the thick ascending limb.Heme oxygenase attenuates angiotensin II-mediated superoxide production in cultured mouse thick ascending loop of Henle cells Na-K-Cl Cotransporter-1 in the mechanism of ammonia-induced astrocyte swelling.Paracellular epithelial sodium transport maximizes energy efficiency in the kidney.Modulation of pressure-natriuresis by renal medullary reactive oxygen species and nitric oxide.Renal medullary oxidative stress, pressure-natriuresis, and hypertension.Abundance of the Na-K-2Cl cotransporter NKCC2 is increased by high-fat feeding in Fischer 344 X Brown Norway (F1) rats.Nitric oxide reduces flow-induced superoxide production via cGMP-dependent protein kinase in thick ascending limbs.Renal function in diabetic disease models: the tubular system in the pathophysiology of the diabetic kidney.Cellular stretch increases superoxide production in the thick ascending limb.

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P2860

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

http://www.wikidata.org/entity/Q46430631

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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name

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@en

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@nl

type

label

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@en

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@nl

prefLabel

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@en

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@nl

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AJPRENAL.00348.2004

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American Journal of Physiology - Renal Physiology

P1476

Superoxide enhances Na-K-2Cl cotransporter activity in the thick ascending limb.

@en

P2093

Jeffrey L Garvin

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Ramiro Juncos

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P2860

Cl(-)-channel blockers in the thick ascending limb of the loop of Henle. Structure activity relationship

Production and actions of superoxide in the renal medulla.

Nitric oxide blockade enhances renal responses to superoxide dismutase inhibition in dogs.

Ammonia transport by early and late proximal convoluted tubule of the rat

The role of reactive oxygen species in the regulation of tubular function.

Na+:K+:2Cl- cotransport and the thick ascending limb.

Effects of antidiuretic hormone on cellular conductive pathways in mouse medullary thick ascending limbs of Henle: II. determinants of the ADH-mediated increases in transepithelial voltage and in net Cl-absorption.

Ion transport ATPases as targets for free radical damage. Protection by an aminosteroid of the Ca2+ pump ATPase and Na+/K+ pump ATPase of human red blood cell membranes.

NO decreases thick ascending limb chloride absorption by reducing Na(+)-K(+)-2Cl(-) cotransporter activity.

Characteristics and superoxide-induced activation of reconstituted myocardial mitochondrial ATP-sensitive potassium channels.

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10.1152/AJPRENAL.00348.2004

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5

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288

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