Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
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PodocytesDrug Targets for Oxidative Podocyte Injury in Diabetic NephropathyRibosomal biogenesis induction by high glucose requires activation of upstream binding factor in kidney glomerular epithelial cellsEffects of the antioxidant drug tempol on renal oxygenation in mice with reduced renal mass.Astragaloside IV, a novel antioxidant, prevents glucose-induced podocyte apoptosis in vitro and in vivo.2,3',4,5'-Tetramethoxystilbene prevents deoxycorticosterone-salt-induced hypertension: contribution of cytochrome P-450 1B1Canonical transient receptor potential channels in diabetes.CXCL16 and oxLDL are induced in the onset of diabetic nephropathy.Protective effects of leukemia inhibitory factor against oxidative stress during high glucose-induced apoptosis in podocytes.Cytochrome P450 1B1 gene disruption minimizes deoxycorticosterone acetate-salt-induced hypertension and associated cardiac dysfunction and renal damage in mice.Mechanism of oxidative DNA damage in diabetes: tuberin inactivation and downregulation of DNA repair enzyme 8-oxo-7,8-dihydro-2'-deoxyguanosine-DNA glycosylaseMechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.Nox4-derived reactive oxygen species mediate cardiomyocyte injury in early type 1 diabetesNephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.Oxidative stress, Nox isoforms and complications of diabetes--potential targets for novel therapies.Renin-angiotensin system within the diabetic podocyte.Role of Protein Kinase G and Reactive Oxygen Species in the Regulation of Podocyte Function in Health and Disease.Research Progress on Mechanism of Podocyte Depletion in Diabetic Nephropathy.Interplay between the Notch and PI3K/Akt pathways in high glucose-induced podocyte apoptosis.Heat shock response protects human peritoneal mesothelial cells from dialysate-induced oxidative stress and mitochondrial injury.Potential role of branched-chain amino acids in glucose metabolism through the accelerated induction of the glucose-sensing apparatus in the liver.
P2860
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P2860
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@en
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@nl
type
label
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@en
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@nl
prefLabel
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@en
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@nl
P2093
P2860
P1476
Redox dependence of glomerular epithelial cell hypertrophy in response to glucose.
@en
P2093
Basant Bhandari
Goutam Ghosh Choudhury
Hanna E Abboud
Hernan Rincon-Choles
Nam-Ho Kim
P2860
P304
P356
10.1152/AJPRENAL.00313.2005
P50
P577
2005-10-18T00:00:00Z