Effects of oleic acid on distinct populations of neurons in the hypothalamic arcuate nucleus are dependent on extracellular glucose levels. - Wikidata - awgldk - TriplyDB

Effects of oleic acid on distinct populations of neurons in the hypothalamic arcuate nucleus are dependent on extracellular glucose levels.

Effects of oleic acid on distinct populations of neurons in the hypothalamic arcuate nucleus are dependent on extracellular glucose levels.

http://www.wikidata.org/entity/Q46818472

about

Metabolic imprinting: critical impact of the perinatal environment on the regulation of energy homeostasis Insights into the role of neuronal glucokinase Acyl coenzyme A thioesterase 7 regulates neuronal fatty acid metabolism to prevent neurotoxicity.Lipid-induced peroxidation in the intestine is involved in glucose homeostasis imbalance in mice Wired on sugar: the role of the CNS in the regulation of glucose homeostasis.The response of glucose-excited neurones in the ventromedial hypothalamus to decreased glucose is enhanced in a murine model of type 2 diabetes mellitus.Fatty acid transporter CD36 mediates hypothalamic effect of fatty acids on food intake in rats.Hypothalamic survival circuits: blueprints for purposive behaviors Gut-brain mechanisms controlling glucose homeostasis.Metabolic sensing and the brain: who, what, where, and how?Orexins: neuropeptides for all seasons and functions.Antecedent hindbrain glucoprivation does not impair the counterregulatory response to hypoglycemia Fatty acid-induced astrocyte ketone production and the control of food intake.Fasting enhances the response of arcuate neuropeptide Y-glucose-inhibited neurons to decreased extracellular glucose Oleic acid directly regulates POMC neuron excitability in the hypothalamus Central but not systemic lipid infusion augments the counterregulatory response to hypoglycemia.Characteristics and mechanisms of hypothalamic neuronal fatty acid sensing.Glucose sensing and the pathogenesis of obesity and type 2 diabetes.Enhanced hypothalamic glucose sensing in obesity: alteration of redox signaling.Nervous glucose sensing regulates postnatal β cell proliferation and glucose homeostasis.Hippocampal lipoprotein lipase regulates energy balance in rodents.Sensing the fuels: glucose and lipid signaling in the CNS controlling energy homeostasis Hypothalamic control of energy and glucose metabolism.Brain insulin signaling: a key component of cognitive processes and a potential basis for cognitive impairment in type 2 diabetes.The regulation of food intake in mammalian hibernators: a review.The receptive function of hypothalamic and brainstem centres to hormonal and nutrient signals affecting energy balance.Targeting lipid sensing in the central nervous system: new therapy against the development of obesity and type 2 diabetes.CNS control of glucose metabolism: response to environmental challenges.Glucagon and lipid signaling in the hypothalamus.Physiological and pathophysiological implications of lipid sensing in the brain.Hypothalamic glucose sensing: making ends meet.Does nutrient sensing determine how we "see" food?Glucose regulates hypothalamic long-chain fatty acid metabolism via AMP-activated kinase (AMPK) in neurons and astrocytes Tanycytes and a differential fatty acid metabolism in the hypothalamus.Pharmacological stimulation of brain carnitine palmitoyl-transferase-1 decreases food intake and body weight.Hypothalamic eIF2α signaling regulates food intake.Oleate induces KATP channel-dependent hyperpolarization in mouse hypothalamic glucose-excited neurons without altering cellular energy charge.Diet-derived nutrients mediate the inhibition of hypothalamic NPY neurons in the arcuate nucleus of mice during refeeding.Prior hypoglycemia enhances glucose responsiveness in some ventromedial hypothalamic glucosensing neurons.Oleic Acid in the Ventral Tegmental Area Inhibits Feeding, Food Reward, and Dopamine Tone.

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P2860

Effects of oleic acid on distinct populations of neurons in the hypothalamic arcuate nucleus are dependent on extracellular glucose levels.

http://www.wikidata.org/entity/Q46818472

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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Effects of oleic acid on disti ...... extracellular glucose levels.

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Effects of oleic acid on disti ...... extracellular glucose levels.

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type

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Effects of oleic acid on disti ...... extracellular glucose levels.

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Effects of oleic acid on disti ...... extracellular glucose levels.

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Journal of Neurophysiology

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Effects of oleic acid on disti ...... extracellular glucose levels.

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P2093

Cotero VE

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Cruciani-Guglielmacci C

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Magnan C

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Migrenne S

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Routh VH

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Wang R

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P2860

Inhibition of cystic fibrosis transmembrane conductance regulator by novel interaction with the metabolic sensor AMP-activated protein kinase

C75, a fatty acid synthase inhibitor, modulates AMP-activated protein kinase to alter neuronal energy metabolism

C75, a fatty acid synthase inhibitor, reduces food intake via hypothalamic AMP-activated protein kinase

Inhibition of hypothalamic carnitine palmitoyltransferase-1 decreases food intake and glucose production

C75 increases peripheral energy utilization and fatty acid oxidation in diet-induced obesity

Hypothalamic K(ATP) channels control hepatic glucose production.

Effects of unsaturated fatty acids on calcium-activated potassium current in gastric myocytes of guinea pigs.

Oxidative stress and potassium channel function.

Cerulenin mimics effects of leptin on metabolic rate, food intake, and body weight independent of the melanocortin system, but unlike leptin, cerulenin fails to block neuroendocrine effects of fasting.

The fatty acid synthase inhibitor cerulenin and feeding, like leptin, activate hypothalamic pro-opiomelanocortin (POMC) neurons.

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1491-1498

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10.1152/JN.00697.2005

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