No indication for a defect in toll-like receptor signaling in patients with hyper-IgE syndrome.
about
Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced T(H)17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome.Toll-like receptor signaling in primary immune deficiencies.Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndromeDefects in Jak-STAT-mediated cytokine signals cause hyper-IgE syndrome: lessons from a primary immunodeficiency.Hyper IgE syndrome: review and future directions.
P2860
No indication for a defect in toll-like receptor signaling in patients with hyper-IgE syndrome.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
No indication for a defect in ...... ients with hyper-IgE syndrome.
@en
No indication for a defect in ...... ients with hyper-IgE syndrome.
@nl
type
label
No indication for a defect in ...... ients with hyper-IgE syndrome.
@en
No indication for a defect in ...... ients with hyper-IgE syndrome.
@nl
prefLabel
No indication for a defect in ...... ients with hyper-IgE syndrome.
@en
No indication for a defect in ...... ients with hyper-IgE syndrome.
@nl
P2093
P1476
No indication for a defect in ...... ients with hyper-IgE syndrome.
@en
P2093
B H Belohradsky
E D Renner
F Hoffmann
S Rothenfusser
P2860
P2888
P304
P356
10.1007/S10875-005-4183-2
P577
2005-07-01T00:00:00Z